Clinical, biochemical, and radiographic effects of aminohydroxypropylidene bisphosphonate treatment in rheumatoid arthritis
about
Anti-TNF-alpha therapy as a clinical intervention for periprosthetic osteolysis.RANKL induces heterogeneous DC-STAMP(lo) and DC-STAMP(hi) osteoclast precursors of which the DC-STAMP(lo) precursors are the master fusogens.The final pathogenetic steps in focal bone erosions in rheumatoid arthritisDisease modifying drugs for rheumatoid arthritis: yesterday's treatment today or today's treatment tomorrow?Conventional treatments for ankylosing spondylitis.The Effect of Alendronate Loaded Biphasic Calcium Phosphate Scaffolds on Bone Regeneration in a Rat Tibial Defect Model.Clinical development of anti-RANKL therapyRegulation of bone destruction in rheumatoid arthritis through RANKL-RANK pathwaysIntravenous pamidronate for refractory rheumatoid arthritis.Differential effects of biologic versus bisphosphonate inhibition of wear debris-induced osteolysis assessed by longitudinal micro-CT.RANKL inhibition for the management of patients with benign metabolic bone disorders.Osteoporosis and inflammation.Pharmacodynamics of bisphosphonates in arthritis.Bisphosphonates: effects on osteoblast.Proteomics Analysis of Cellular Proteins Co-Immunoprecipitated with Nucleoprotein of Influenza A Virus (H7N9)Contrast between effects of aminobisphosphonates and non-aminobisphosphonates on collagen-induced arthritis in mice.Effects of low-dose, noncytotoxic, intraarticular liposomal clodronate on development of erosions and proteoglycan loss in established antigen-induced arthritis in rabbits.A six-month randomized, controlled, double-blind, dose-response comparison of intravenous pamidronate (60 mg versus 10 mg) in the treatment of nonsteroidal antiinflammatory drug-refractory ankylosing spondylitis.Single infusion of neridronate (6-amino-1-hydroxyhexylidene-1,1-bisphosphonate) in patients with active rheumatoid arthritis: effects on disease activity and bone resorption markers.Cherubism Mice Also Deficient in c-Fos Exhibit Inflammatory Bone Destruction Executed by Macrophages That Express MMP14 Despite the Absence of TRAP+ Osteoclasts.RANKL: A therapeutic target for bone destruction in rheumatoid arthritis.Downregulation of the inflammatory response by CORM-3 results in protective effects in a model of postmenopausal arthritis.
P2860
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P2860
Clinical, biochemical, and radiographic effects of aminohydroxypropylidene bisphosphonate treatment in rheumatoid arthritis
description
1989 nî lūn-bûn
@nan
1989 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Clinical, biochemical, and rad ...... atment in rheumatoid arthritis
@ast
Clinical, biochemical, and rad ...... atment in rheumatoid arthritis
@en
Clinical, biochemical, and rad ...... tment in rheumatoid arthritis.
@nl
type
label
Clinical, biochemical, and rad ...... atment in rheumatoid arthritis
@ast
Clinical, biochemical, and rad ...... atment in rheumatoid arthritis
@en
Clinical, biochemical, and rad ...... tment in rheumatoid arthritis.
@nl
prefLabel
Clinical, biochemical, and rad ...... atment in rheumatoid arthritis
@ast
Clinical, biochemical, and rad ...... atment in rheumatoid arthritis
@en
Clinical, biochemical, and rad ...... tment in rheumatoid arthritis.
@nl
P2093
P2860
P356
P1476
Clinical, biochemical, and rad ...... atment in rheumatoid arthritis
@en
P2093
Pitkeathly DA
Ralston SH
Willocks L
P2860
P304
P356
10.1136/ARD.48.5.396
P407
P577
1989-05-01T00:00:00Z