Candida albicans SUR7 contributes to secretion, biofilm formation, and macrophage killing.
about
Candida albicans Biofilms and Human DiseaseCandida Biofilms: Development, Architecture, and ResistanceAn automated high-throughput cell-based multiplexed flow cytometry assay to identify novel compounds to target Candida albicans virulence-related proteinsCandida albicans VMA3 is necessary for V-ATPase assembly and function and contributes to secretion and filamentation.The contribution of Candida albicans vacuolar ATPase subunit V₁B, encoded by VMA2, to stress response, autophagy, and virulence is independent of environmental pH.The Candida albicans Sur7 protein is needed for proper synthesis of the fibrillar component of the cell wall that confers strength.Sur7 promotes plasma membrane organization and is needed for resistance to stressful conditions and to the invasive growth and virulence of Candida albicans.Membrane Compartment Occupied by Can1 (MCC) and Eisosome Subdomains of the Fungal Plasma Membrane.Lipid Biosynthetic Genes Affect Candida albicans Extracellular Vesicle Morphology, Cargo, and Immunostimulatory PropertiesFunctional Analysis of the Exocyst Subunit Sec15 in Candida albicans.Deletion of vacuolar proton-translocating ATPase V(o)a isoforms clarifies the role of vacuolar pH as a determinant of virulence-associated traits in Candida albicansEisosomes promote the ability of Sur7 to regulate plasma membrane organization in Candida albicans.Candida species: new insights into biofilm formation.Eisosomes and plasma membrane organization.Methods for obtaining reliable and reproducible results in studies of Candida biofilms formed in vitro.Multispectral imaging: a review of its technical aspects and applications in anatomic pathology.Plasma membrane organization promotes virulence of the human fungal pathogen Candida albicans.Paradoxical antifungal activity and structural observations in biofilms formed by echinocandin-resistant Candida albicans clinical isolates.Contribution of the glycolytic flux and hypoxia adaptation to efficient biofilm formation by Candida albicans.MCC/Eisosomes Regulate Cell Wall Synthesis and Stress Responses in Fungi.The Gpr1-regulated Sur7 family protein Sfp2 is required for hyphal growth and cell wall stability in the mycoparasite Trichoderma atroviride
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P2860
Candida albicans SUR7 contributes to secretion, biofilm formation, and macrophage killing.
description
2010 nî lūn-bûn
@nan
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Candida albicans SUR7 contribu ...... ation, and macrophage killing.
@ast
Candida albicans SUR7 contribu ...... ation, and macrophage killing.
@en
Candida albicans SUR7 contribu ...... ation, and macrophage killing.
@nl
type
label
Candida albicans SUR7 contribu ...... ation, and macrophage killing.
@ast
Candida albicans SUR7 contribu ...... ation, and macrophage killing.
@en
Candida albicans SUR7 contribu ...... ation, and macrophage killing.
@nl
prefLabel
Candida albicans SUR7 contribu ...... ation, and macrophage killing.
@ast
Candida albicans SUR7 contribu ...... ation, and macrophage killing.
@en
Candida albicans SUR7 contribu ...... ation, and macrophage killing.
@nl
P2860
P356
P1433
P1476
Candida albicans SUR7 contribu ...... ation, and macrophage killing.
@en
P2093
Samuel A Lee
Stella M Bernardo
P2860
P2888
P356
10.1186/1471-2180-10-133
P577
2010-04-30T00:00:00Z
P5875
P6179
1020372368