Airway uric acid is a sensor of inhaled protease allergens and initiates type 2 immune responses in respiratory mucosa
about
Group 2 Innate Lymphoid Cells Promote an Early Antibody Response to a Respiratory Antigen in Mice.Pulmonary ORMDL3 is critical for induction of Alternaria-induced allergic airways disease.Pulmonary receptor for advanced glycation end-products promotes asthma pathogenesis through IL-33 and accumulation of group 2 innate lymphoid cellsATP drives eosinophil effector responses through P2 purinergic receptors.Group 2 innate lymphoid cells utilize the IRF4-IL-9 module to coordinate epithelial cell maintenance of lung homeostasisCockroach protease allergen induces allergic airway inflammation via epithelial cell activation.What makes an allergen?ILC2s and fungal allergy.Profiling the Extended Cleavage Specificity of the House Dust Mite Protease Allergens Der p 1, Der p 3 and Der p 6 for the Prediction of New Cell Surface Protein Substrates.Interplay between barrier epithelial cells and dendritic cells in allergic sensitization through the lung and the skin.IL-33: biological properties, functions, and roles in airway disease.Physiological functions and pathogenic potential of uric acid: A review.Mucosal production of uric acid by airway epithelial cells contributes to particulate matter-induced allergic sensitization.Caspase-1 activation by NLRP3 inflammasome dampens IL-33-dependent house dust mite-induced allergic lung inflammation.Ozone-Induced Nasal Type 2 Immunity in Mice Is Dependent on Innate Lymphoid Cells.Interleukin-33 (IL-33): A nuclear cytokine from the IL-1 family.Allergens produce serine proteases-dependent distinct release of metabolite DAMPs in human bronchial epithelial cells.Orchestration of epithelial-derived cytokines and innate immune cells in allergic airway inflammation.Endogenous Protease Inhibitors in Airway Epithelial Cells Contribute to Eosinophilic Chronic Rhinosinusitis.Emerging Role of Proteases in the Pathogenesis of Chronic Rhinosinusitis with Nasal Polyps.MR-PheWAS: exploring the causal effect of SUA level on multiple disease outcomes by using genetic instruments in UK Biobank.Distinct sustained structural and functional effects of interleukin-33 and interleukin-25 on the airways in a murine asthma surrogate.Subcutaneous Allergic Sensitization to Protease Allergen Is Dependent on Mast Cells but Not IL-33: Distinct Mechanisms between Subcutaneous and Intranasal Routes.The Initiation of Th2 Immunity Towards Food Allergens.
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P2860
Airway uric acid is a sensor of inhaled protease allergens and initiates type 2 immune responses in respiratory mucosa
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2014 թուականի Մարտին հրատարակուած գիտական յօդուած
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2014年の論文
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Airway uric acid is a sensor o ...... esponses in respiratory mucosa
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Airway uric acid is a sensor o ...... esponses in respiratory mucosa
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Airway uric acid is a sensor o ...... sponses in respiratory mucosa.
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Airway uric acid is a sensor o ...... esponses in respiratory mucosa
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Airway uric acid is a sensor o ...... esponses in respiratory mucosa
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Airway uric acid is a sensor o ...... sponses in respiratory mucosa.
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Airway uric acid is a sensor o ...... esponses in respiratory mucosa
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Airway uric acid is a sensor o ...... esponses in respiratory mucosa
@en
Airway uric acid is a sensor o ...... sponses in respiratory mucosa.
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P2093
P2860
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Airway uric acid is a sensor o ...... esponses in respiratory mucosa
@en
P2093
Gail M Kephart
Hirohito Kita
Ichiro Tojima
Kenichiro Hara
Koji Iijima
Martha K Elias
Masahiko Kurabayashi
Satoshi Seno
Takao Kobayashi
P2860
P304
P356
10.4049/JIMMUNOL.1400110
P407
P577
2014-03-24T00:00:00Z