Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.
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Non-steroidal anti-inflammatory drugs and cognitive function: are prostaglandins at the heart of cognitive impairment in dementia and delirium?Therapeutic potential of monoacylglycerol lipase inhibitorsInflammatory process in Alzheimer's DiseaseA role for cyclooxygenase-1 in beta-amyloid-induced neuroinflammation.Unesterified docosahexaenoic acid is protective in neuroinflammation.Cyclooxygenase-1-selective inhibitors based on the (E)-2'-des-methyl-sulindac sulfide scaffold.Targeting cyclooxygenases-1 and -2 in neuroinflammation: Therapeutic implications.Cyclooxygenases and 5-lipoxygenase in Alzheimer's disease.Thalidomide Analogues Suppress Lipopolysaccharide-Induced Synthesis of TNF-α and Nitrite, an Intermediate of Nitric Oxide, in a Cellular Model of InflammationCyclooxygenase-1 is involved in the inhibition of hippocampal neurogenesis after lipopolysaccharide-induced neuroinflammationEndocannabinoid hydrolysis generates brain prostaglandins that promote neuroinflammation.Minocycline and aspirin in the treatment of bipolar depression: a protocol for a proof-of-concept, randomised, double-blind, placebo-controlled, 2x2 clinical trialCyclooxygenases-1 and -2 differentially modulate leukocyte recruitment into the inflamed brain.Inhibition of NADPH oxidase promotes alternative and anti-inflammatory microglial activation during neuroinflammationTumor necrosis factor-α synthesis inhibitor 3,6'-dithiothalidomide attenuates markers of inflammation, Alzheimer pathology and behavioral deficits in animal models of neuroinflammation and Alzheimer's disease3,6'-Dithiothalidomide, a new TNF-α synthesis inhibitor, attenuates the effect of Aβ1-42 intracerebroventricular injection on hippocampal neurogenesis and memory deficit.The Alzheimer's disease transcriptome mimics the neuroprotective signature of IGF-1 receptor-deficient neurons.Deletion of Monoglyceride Lipase in Astrocytes Attenuates Lipopolysaccharide-induced NeuroinflammationA dysregulated endocannabinoid-eicosanoid network supports pathogenesis in a mouse model of Alzheimer's disease.Cyclooxygenase-1 inhibition reduces amyloid pathology and improves memory deficits in a mouse model of Alzheimer's disease.Therapeutic implications of the prostaglandin pathway in Alzheimer's disease.Targeting cyclooxygenase-2 in depression is not a viable therapeutic approach and may even aggravate the pathophysiology underpinning depression.The role of inflammatory and oxidative stress mechanisms in the pathogenesis of Parkinson's disease: focus on astrocytes.Pharmacological approaches for Alzheimer's disease: neurotransmitter as drug targets.Phosphorylation of amyloid beta (Aβ) peptides - a trigger for formation of toxic aggregates in Alzheimer's disease.Lipopolysaccharide Preconditioning Induces an Anti-inflammatory Phenotype in BV2 Microglia.LPS induces mediators of neuroinflammation, cell proliferation, and GFAP expression in human astrocytoma cells U373MG: the anti-inflammatory and anti-proliferative effect of guggulipid.Monoglyceride lipase as a drug target: At the crossroads of arachidonic acid metabolism and endocannabinoid signaling.Targeting neuroinflammation in Alzheimer's disease: evidence for NSAIDs and novel therapeutics.Alleviation of Microglial Activation Induced by p38 MAPK/MK2/PGE2 Axis by Capsaicin: Potential Involvement of other than TRPV1 Mechanism/s.Highly Selective Cyclooxygenase-1 Inhibitors P6 and Mofezolac Counteract Inflammatory State both In Vitro and In Vivo Models of Neuroinflammation.β-Amyloid induced effects on cholinergic, serotonergic, and dopaminergic neurons is differentially counteracted by anti-inflammatory drugs.Melatonin attenuates impairments of structural hippocampal neuroplasticity in OXYS rats during active progression of Alzheimer's disease-like pathology.NSAID Exposure and Risk of Alzheimer's Disease: An Updated Meta-Analysis From Cohort Studies.Emerging PET Radiotracers and Targets for Imaging of Neuroinflammation in Neurodegenerative Diseases: Outlook Beyond TSPOA Selection of Important Genes and Their Correlated Behavior in Alzheimer's Disease
P2860
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P2860
Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.
description
2009 nî lūn-bûn
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2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.
@ast
Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.
@en
Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.
@nl
type
label
Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.
@ast
Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.
@en
Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.
@nl
prefLabel
Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.
@ast
Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.
@en
Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.
@nl
P2860
P356
P1433
P1476
Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.
@en
P2093
Francesca Bosetti
Sang-Ho Choi
P2860
P304
P356
10.18632/AGING.100021
P577
2009-02-11T00:00:00Z