Combining neuropeptide Y and mesenchymal stem cells reverses remodeling after myocardial infarction. - Wikidata - awgldk - TriplyDB

Combining neuropeptide Y and mesenchymal stem cells reverses remodeling after myocardial infarction.

Combining neuropeptide Y and mesenchymal stem cells reverses remodeling after myocardial infarction.

http://www.wikidata.org/entity/Q33589174

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Chemokines and Heart Disease: A Network Connecting Cardiovascular Biology to Immune and Autonomic Nervous Systems Mesenchymal stem cells in cardiac regeneration: a detailed progress report of the last 6 years (2010-2015)Suicide gene reveals the myocardial neovascularization role of mesenchymal stem cells overexpressing CXCR4 (MSC(CXCR4))Biodistribution, migration and homing of systemically applied mesenchymal stem/stromal cells Neuropeptide y and neuropeptide y y5 receptor interaction restores impaired growth potential of aging bone marrow stromal cells Cardiomyocyte-like cells differentiation from non β-catenin expression mesenchymal stem cells MicroRNA-377 regulates mesenchymal stem cell-induced angiogenesis in ischemic hearts by targeting VEGF Effects of neuropeptide Y on collateral development in a swine model of chronic myocardial ischemia Genetically manipulated progenitor cell sheet with diprotin A improves myocardial function and repair of infarcted hearts.Recent concepts for the roles of progenitor/stem cell niche in heart repair.Neuropeptide Y3-36 incorporated into PVAX nanoparticle improves functional blood flow in a murine model of hind limb ischemia.The immunomodulatory properties of mesenchymal stem cells: implications for surgical disease.Optimization of the cardiovascular therapeutic properties of mesenchymal stromal/stem cells-taking the next step.Reproductive stem cell differentiation: extracellular matrix, tissue microenvironment, and growth factors direct the mesenchymal stem cell lineage commitment.Rebuilding the Damaged Heart: Mesenchymal Stem Cells, Cell-Based Therapy, and Engineered Heart Tissue.Can the outcomes of mesenchymal stem cell-based therapy for myocardial infarction be improved? Providing weapons and armour to cells.Effects of Neuropeptide Y on Stem Cells and Their Potential Applications in Disease Therapy.Electrical Stimulation Enhances Cardiac Differentiation of Human Induced Pluripotent Stem Cells for Myocardial Infarction Therapy.Neuropeptide Y Induces Hematopoietic Stem/Progenitor Cell Mobilization by Regulating Matrix Metalloproteinase-9 Activity Through Y1 Receptor in Osteoblasts.

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Combining neuropeptide Y and mesenchymal stem cells reverses remodeling after myocardial infarction.

http://www.wikidata.org/entity/Q33589174

description

2009 nî lūn-bûn

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2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2009 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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Combining neuropeptide Y and m ...... g after myocardial infarction.

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Combining neuropeptide Y and m ...... g after myocardial infarction.

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Combining neuropeptide Y and m ...... g after myocardial infarction.

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Combining neuropeptide Y and m ...... g after myocardial infarction.

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Combining neuropeptide Y and m ...... g after myocardial infarction.

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Combining neuropeptide Y and m ...... g after myocardial infarction.

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Combining neuropeptide Y and m ...... g after myocardial infarction.

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Combining neuropeptide Y and m ...... g after myocardial infarction.

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AJPHEART.00765.2009

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American Journal of Physiology - Heart and Circulatory Physiology

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Combining neuropeptide Y and m ...... g after myocardial infarction.

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Ambikaipakan Balasubramaniam

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Atif Ashraf

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Dongsheng Zhang

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Muhammad Ashraf

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Tiemin Zhao

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Wei Huang

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Yigang Wang

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P2860

FGF1/p38 MAP kinase inhibitor therapy induces cardiomyocyte mitosis, reduces scarring, and rescues function after myocardial infarction

Plasmapheresis reverses all side-effects of a cisplatin overdose--a case report and treatment recommendation

Neuropeptide Y induces migration, proliferation, and tube formation of endothelial cells bimodally via Y1, Y2, and Y5 receptors

Expression of cyclin D1 and CDK4 causes hypertrophic growth of cardiomyocytes in culture: a possible implication for cardiac hypertrophy

Growth factor activation in myocardial vascularization: therapeutic implications

Cx43 mediates TGF-beta signaling through competitive Smads binding to microtubules.

Combining pharmacological mobilization with intramyocardial delivery of bone marrow cells over-expressing VEGF is more effective for cardiac repair.

Eukaryotic initiation factor 4E (eIF4E) and angiogenesis: prognostic markers for breast cancer

Forced expression of the cell cycle inhibitor p57Kip2 in cardiomyocytes attenuates ischemia-reperfusion injury in the mouse heart.

Translational control of growth factor and proto-oncogene expression.

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