Bordetella evades the host immune system by inducing IL-10 through a type III effector, BopN - Wikidata - awgldk - TriplyDB

Bordetella evades the host immune system by inducing IL-10 through a type III effector, BopN

Bordetella evades the host immune system by inducing IL-10 through a type III effector, BopN

http://www.wikidata.org/entity/Q33590435

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Rediscovering Pertussis IFN-γ Priming Effects on the Maintenance of Effector Memory CD4(+) T Cells and on Phagocyte Function: Evidences from Infectious Diseases BteA Secreted from the Bordetella bronchiseptica Type III Secetion System Induces Necrosis through an Actin Cytoskeleton Signaling Pathway and Inhibits Phagocytosis by Macrophages Modulation of NF-κB signalling by microbial pathogens Pertussis: Microbiology, Disease, Treatment, and Prevention.Tobacco upregulates P. gingivalis fimbrial proteins which induce TLR2 hyposensitivity.Hijacking of the pleiotropic cytokine interferon-γ by the type III secretion system of Yersinia pestis Differential expression of type III effector BteA protein due to IS481 insertion in Bordetella pertussis.IL-10 regulates viral lung immunopathology during acute respiratory syncytial virus infection in mice The type III secreted protein BspR regulates the virulence genes in Bordetella bronchiseptica.The pore-forming toxin β hemolysin/cytolysin triggers p38 MAPK-dependent IL-10 production in macrophages and inhibits innate immunity.Bordetella pertussis pathogenesis: current and future challenges.Polymorphisms influencing expression of dermonecrotic toxin in Bordetella bronchiseptica.IL-27 promotes IL-10 production by effector Th1 CD4+ T cells: a critical mechanism for protection from severe immunopathology during malaria infection The Bordetella Secreted Regulator BspR Is Translocated into the Nucleus of Host Cells via Its N-Terminal Moiety: Evaluation of Bacterial Effector Translocation by the Escherichia coli Type III Secretion System.Inhibition of Nuclear Transport of NF-ĸB p65 by the Salmonella Type III Secretion System Effector SpvD.Interleukin-10 induction is an important virulence function of the Yersinia pseudotuberculosis type III effector YopM.Piscirickettsia salmonis Imbalances the Innate Immune Response to Succeed in a Productive Infection in a Salmonid Cell Line Model.Immunomodulation and Disease Tolerance to Staphylococcus aureus Regulatory B cell (B10 Cell) expansion during Listeria infection governs innate and cellular immune responses in mice.Exploitation of Interleukin-10 (IL-10) Signaling Pathways: Alternate Roles of Viral and Cellular IL-10 in Rhesus Cytomegalovirus Infection.The Aeromonas salmonicida subsp. salmonicida exoproteome: determination of the complete repertoire of Type-Three Secretion System effectors and identification of other virulence factors.Differentially expressed genes in Bordetella pertussis strains belonging to a lineage which recently spread globally The Bordetella bronchiseptica type III secretion system is required for persistence and disease severity but not transmission in swine.Pertussis: a matter of immune modulation.Functional domains and motifs of bacterial type III effector proteins and their roles in infection.Subversion of innate immune responses by bacterial hindrance of NF-κB pathway.Immunity to the respiratory pathogen Bordetella pertussis.Bacterial interference with canonical NFκB signalling.Aeromonas salmonicida subsp. salmonicida in the light of its type-three secretion system Immune Homeostatic Macrophages Programmed by the Bacterial Surface Protein NhhA Potentiate Nasopharyngeal Carriage of Neisseria meningitidis.Bordetella effector BopN is translocated into host cells via its N-terminal residues.Invasion of Dendritic Cells, Macrophages and Neutrophils by the Bordetella Adenylate Cyclase Toxin: A Subversive Move to Fool Host Immunity.Blood or Serum Exposure Induce Global Transcriptional Changes, Altered Antigenic Profile, and Increased Cytotoxicity by Classical Bordetellae Respiratory Carriage is Associated with Broad Phenotypic Alterations of Peripheral CD4⁺CD25⁺ T Cells and Differentially Affects Immune Responses to Secondary Non-Infectious and Infectious Stimuli in Mice

P2860

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P2860

Bordetella evades the host immune system by inducing IL-10 through a type III effector, BopN

http://www.wikidata.org/entity/Q33590435

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

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2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

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name

Bordetella evades the host imm ...... ough a type III effector, BopN

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Bordetella evades the host imm ...... ough a type III effector, BopN

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Bordetella evades the host imm ...... ough a type III effector, BopN

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Bordetella evades the host imm ...... ough a type III effector, BopN

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Bordetella evades the host imm ...... ough a type III effector, BopN

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Bordetella evades the host imm ...... ough a type III effector, BopN

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P1433

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P2860

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P1433

Journal of Experimental Medicine

P1476

Bordetella evades the host imm ...... ough a type III effector, BopN

@en

P2093

Akio Abe

http://www.w3.org/2001/XMLSchema#string

Asaomi Kuwae

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Hitomi Mimuro

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Kanna Nagamatsu

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Masahiro Eguchi

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Mineo Watanabe

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Sei Yoshida

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Shigenori Nagai

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Tadashi Konaka

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P2860

The NF-kappa B and I kappa B proteins: new discoveries and insights

Molecular pathogenesis, epidemiology, and clinical manifestations of respiratory infections due to Bordetella pertussis and other Bordetella subspecies.

The nucleosomal response associated with immediate-early gene induction is mediated via alternative MAP kinase cascades: MSK1 as a potential histone H3/HMG-14 kinase

Phosphorylation meets ubiquitination: the control of NF-[kappa]B activity

NF-kappa B and Rel proteins: evolutionarily conserved mediators of immune responses

Protein delivery into eukaryotic cells by type III secretion machines

Two enteropathogenic Escherichia coli type III secreted proteins, EspA and EspB, are virulence factors.

BopC is a novel type III effector secreted by Bordetella bronchiseptica and has a critical role in type III-dependent necrotic cell death.

Supermolecular structure of the enteropathogenic Escherichia coli type III secretion system and its direct interaction with the EspA-sheath-like structure

The Shigella flexneri effector OspG interferes with innate immune responses by targeting ubiquitin-conjugating enzymes

P304

3073-3088

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P356

10.1084/JEM.20090494

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P433

13

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206

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2009-12-14T00:00:00Z

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40686261

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20008527

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2806459

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