Deletion of CXCR4 in cardiomyocytes exacerbates cardiac dysfunction following isoproterenol administration
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Chemokines and Heart Disease: A Network Connecting Cardiovascular Biology to Immune and Autonomic Nervous SystemsExtracellular Ubiquitin: Role in Myocyte Apoptosis and Myocardial Remodeling.SDF-1α (Stromal Cell-Derived Factor 1α) Induces Cardiac Fibroblasts, Renal Microvascular Smooth Muscle Cells, and Glomerular Mesangial Cells to Proliferate, Cause Hypertrophy, and Produce Collagen.CXCR4 and CXCR7 play distinct roles in cardiac lineage specification and pharmacologic β-adrenergic response.Endoplasmic Reticulum Protein TXNDC5 Augments Myocardial Fibrosis by Facilitating Extracellular Matrix Protein Folding and Redox-Sensitive Cardiac Fibroblast Activation.
P2860
Deletion of CXCR4 in cardiomyocytes exacerbates cardiac dysfunction following isoproterenol administration
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name
Deletion of CXCR4 in cardiomyo ...... g isoproterenol administration
@ast
Deletion of CXCR4 in cardiomyo ...... g isoproterenol administration
@en
type
label
Deletion of CXCR4 in cardiomyo ...... g isoproterenol administration
@ast
Deletion of CXCR4 in cardiomyo ...... g isoproterenol administration
@en
prefLabel
Deletion of CXCR4 in cardiomyo ...... g isoproterenol administration
@ast
Deletion of CXCR4 in cardiomyo ...... g isoproterenol administration
@en
P2093
P2860
P356
P1433
P1476
Deletion of CXCR4 in cardiomyo ...... g isoproterenol administration
@en
P2093
A A Jarrah
M Schwarzkopf
S T Tarzami
P2860
P2888
P304
P356
10.1038/GT.2014.23
P577
2014-03-20T00:00:00Z