Hyperglycemia, a neglected factor during cancer progression - Wikidata - awgldk - TriplyDB

Hyperglycemia, a neglected factor during cancer progression

Hyperglycemia, a neglected factor during cancer progression

http://www.wikidata.org/entity/Q33594115

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Hyperglycemia as a risk factor for cancer progression High glucose dephosphorylates serine 46 and inhibits p53 apoptotic activity Metabolic Mediators of the Association Between Adult Weight Gain and Colorectal Cancer: Data From the European Prospective Investigation into Cancer and Nutrition (EPIC) Cohort.High glucose enhances progression of cholangiocarcinoma cells via STAT3 activation.Poor Preoperative Glycemic Control Is Associated with Dismal Prognosis after Radical Nephroureterectomy for Upper Tract Urothelial Carcinoma: A Korean Multicenter Study.ZnCl2 sustains the adriamycin-induced cell death inhibited by high glucose.High glucose levels boost the aggressiveness of highly metastatic cholangiocarcinoma cells via O-GlcNAcylation Alteration in Taste Perception in Cancer: Causes and Strategies of Treatment Hyperglycemia triggers HIPK2 protein degradation.Activation of PI3K in response to high glucose leads to regulation of SOCS-3 and STAT1/3 signals and induction of glomerular mesangial extracellular matrix formation.Resveratrol in the treatment of pancreatic cancer.Adrenocortical tumors and insulin resistance: What is the first step?Enemies or weapons in hands: investigational anti-diabetic drug glibenclamide and cancer risk.Metformin, Diabetes, and Survival among U.S. Veterans with Colorectal Cancer Diabetes and its link with cancer: providing the fuel and spark to launch an aggressive growth regime.PTEN deficiency and mutant p53 confer glucose-addiction to thyroid cancer cells: impact of glucose depletion on cell proliferation, cell survival, autophagy and cell migration.Hyperglycemia exacerbates colon cancer malignancy through hexosamine biosynthetic pathway Glucose impairs tamoxifen responsiveness modulating connective tissue growth factor in breast cancer cells.Energy metabolism in glioblastoma stem cells: PPARα a metabolic adaptor to intratumoral microenvironment.Hyperglycemia and aberrant O-GlcNAcylation: contributions to tumor progression.Risk of hyperglycemia attributable to everolimus in cancer patients: A meta-analysis.Hyperglycemia Promotes Chemoresistance Through the Reduction of the Mitochondrial DNA Damage, the Bax/Bcl-2 and Bax/Bcl-XL Ratio, and the Cells in Sub-G1 Phase Due to Antitumoral Drugs Induced-Cytotoxicity in Human Colon Adenocarcinoma Cells Inhibitory Effect of Extract on Hyperglycemia-Related Expression and Cancer Proliferation

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Hyperglycemia, a neglected factor during cancer progression

http://www.wikidata.org/entity/Q33594115

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2014 nî lūn-bûn

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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի ապրիլին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Hyperglycemia, a neglected factor during cancer progression

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Hyperglycemia, a neglected factor during cancer progression

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Hyperglycemia, a neglected factor during cancer progression

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Hyperglycemia, a neglected factor during cancer progression

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Hyperglycemia, a neglected factor during cancer progression

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BioMed Research International

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Hyperglycemia, a neglected factor during cancer progression

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Jianjun Lei

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Qingyong Ma

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Qinhong Xu

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Xin Shen

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Yongtian Yu

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Creative Commons Attribution 3.0 Unported

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Hallmarks of Cancer: The Next Generation

Glucose deprivation contributes to the development of KRAS pathway mutations in tumor cells

Biochemistry and molecular cell biology of diabetic complications

The role of the p38 MAPK signaling pathway in high glucose-induced epithelial-mesenchymal transition of cultured human renal tubular epithelial cells

National, regional, and global trends in fasting plasma glucose and diabetes prevalence since 1980: systematic analysis of health examination surveys and epidemiological studies with 370 country-years and 2·7 million participants

The distribution of secondary growths in cancer of the breast. 1889

THE METABOLISM OF TUMORS IN THE BODY

Mitochondrial metabolism and ROS generation are essential for Kras-mediated tumorigenicity

Glucose transporter GLUT1 in colorectal adenocarcinoma cell lines is inversely correlated with tumour cell proliferation.

Paracrine regulation of pancreatic cancer cell invasion by peripheral nerves.

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2014

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