Piperine Attenuates Pathological Cardiac Fibrosis Via PPAR-γ/AKT Pathways. - Wikidata - awgldk - TriplyDB

Piperine Attenuates Pathological Cardiac Fibrosis Via PPAR-γ/AKT Pathways.

Piperine Attenuates Pathological Cardiac Fibrosis Via PPAR-γ/AKT Pathways.

http://www.wikidata.org/entity/Q33603596

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Interactions between TGF-β1, canonical WNT/β-catenin pathway and PPAR γ in radiation-induced fibrosis.Cucurbitacin B Protects Against Pressure Overload Induced Cardiac Hypertrophy.Rosmarinic acid attenuates cardiac fibrosis following long-term pressure overload via AMPKα/Smad3 signaling.Therapeutic Potential of Polyphenols in Cardiac Fibrosis.Cardiac fibrosis: new insights into the pathogenesis Geniposide Alleviates Isoproterenol-Induced Cardiac Fibrosis Partially via SIRT1 Activation and Saikosaponin A Protects From Pressure Overload-Induced Cardiac Fibrosis via Inhibiting Fibroblast Activation or Endothelial Cell EndMT

P2860

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P2860

Piperine Attenuates Pathological Cardiac Fibrosis Via PPAR-γ/AKT Pathways.

http://www.wikidata.org/entity/Q33603596

description

2017 nî lūn-bûn

@nan

2017 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2017 թվականի մարտին հրատարակված գիտական հոդված

@hy

2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

name

Piperine Attenuates Pathological Cardiac Fibrosis Via PPAR-γ/AKT Pathways.

@ast

Piperine Attenuates Pathological Cardiac Fibrosis Via PPAR-γ/AKT Pathways.

@en

type

label

Piperine Attenuates Pathological Cardiac Fibrosis Via PPAR-γ/AKT Pathways.

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Piperine Attenuates Pathological Cardiac Fibrosis Via PPAR-γ/AKT Pathways.

@en

prefLabel

Piperine Attenuates Pathological Cardiac Fibrosis Via PPAR-γ/AKT Pathways.

@ast

Piperine Attenuates Pathological Cardiac Fibrosis Via PPAR-γ/AKT Pathways.

@en

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P356

J.EBIOM.2017.03.021

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P1476

Piperine Attenuates Pathological Cardiac Fibrosis Via PPAR-γ/AKT Pathways.

@en

P2093

Sha-Sha Wang

http://www.w3.org/2001/XMLSchema#string

Si-Chi Xu

http://www.w3.org/2001/XMLSchema#string

Xin Zhang

http://www.w3.org/2001/XMLSchema#string

Yu-Pei Yuan

http://www.w3.org/2001/XMLSchema#string

Zhen-Guo Ma

http://www.w3.org/2001/XMLSchema#string

P2860

Cardiac nonmyocytes in the hub of cardiac hypertrophy

Smad-dependent and Smad-independent pathways in TGF-beta family signalling

Serum and glucocorticoid-responsive kinase-1 regulates cardiomyocyte survival and hypertrophic response

Molecular characterization of angiotensin II--induced hypertrophy of cardiac myocytes and hyperplasia of cardiac fibroblasts. Critical role of the AT1 receptor subtype

Regulation of cardiac hypertrophy by intracellular signalling pathways

Cardiac hypertrophy: the good, the bad, and the ugly

Piperidine alkaloids from Piper retrofractum Vahl. protect against high-fat diet-induced obesity by regulating lipid metabolism and activating AMP-activated protein kinase.

A synthetic PPAR-γ agonist triterpenoid ameliorates experimental fibrosis: PPAR-γ-independent suppression of fibrotic responses

PPAR-γ ligands repress TGFβ-induced myofibroblast differentiation by targeting the PI3K/Akt pathway: implications for therapy of fibrosis.

Characterization of angiotensin II receptors in cultured adult rat cardiac fibroblasts. Coupling to signaling systems and gene expression.

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179-187

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scholarly article

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10.1016/J.EBIOM.2017.03.021

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18

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