Controlling the receptor for advanced glycation end-products to conquer diabetic vascular complications - Wikidata - awgldk - TriplyDB

Controlling the receptor for advanced glycation end-products to conquer diabetic vascular complications

Controlling the receptor for advanced glycation end-products to conquer diabetic vascular complications

http://www.wikidata.org/entity/Q33609776

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Environmental Contaminants and Pancreatic Beta-Cells Induction of receptor for advanced glycation end products by insufficient leptin action triggers pancreatic β-cell failure in type 2 diabetes.Intestinal transepithelial permeability of oxytocin into the blood is dependent on the receptor for advanced glycation end products in mice.RAGE-Mediated Inflammation, Type 2 Diabetes, and Diabetic Vascular Complication.Preventive Effect of Salicylate and Pyridoxamine on Diabetic Nephropathy.Pattern-recognition receptors: Signaling pathways and dysregulation in canine chronic enteropathies-brief review.Investigation of Gal-3 Expression Pattern in Serum and Cerebrospinal Fluid of Patients Suffering From Neurodegenerative Disorders

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Controlling the receptor for advanced glycation end-products to conquer diabetic vascular complications

http://www.wikidata.org/entity/Q33609776

description

2012 nî lūn-bûn

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2012 թուականի Մարտին հրատարակուած գիտական յօդուած

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2012 թվականի մարտին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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J.2040-1124.2011.00191.X

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Journal of Diabetes Investigation

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Controlling the receptor for a ...... iabetic vascular complications

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Hiroshi Yamamoto

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Yasuhiko Yamamoto

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Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways

RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

Cloning and expression of a cell surface receptor for advanced glycosylation end products of proteins

RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease

Interaction of the RAGE cytoplasmic domain with diaphanous-1 is required for ligand-stimulated cellular migration through activation of Rac1 and Cdc42

The glyoxalase system: new developments towards functional characterization of a metabolic pathway fundamental to biological life

The nuclear protein HMGB1 is secreted by monocytes via a non-classical, vesicle-mediated secretory pathway

Loss of pain perception in diabetes is dependent on a receptor of the immunoglobulin superfamily

Advanced glycation endproduct (AGE) receptor 1 is a negative regulator of the inflammatory response to AGE in mesangial cells

Orally absorbed reactive glycation products (glycotoxins): an environmental risk factor in diabetic nephropathy

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107-114

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scholarly article

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10.1111/J.2040-1124.2011.00191.X

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4020727

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