Response of Npt2a knockout mice to dietary calcium and phosphorus. - Wikidata - awgldk - TriplyDB

Response of Npt2a knockout mice to dietary calcium and phosphorus.

Response of Npt2a knockout mice to dietary calcium and phosphorus.

http://www.wikidata.org/entity/Q33610690

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Intraperitoneal pyrophosphate treatment reduces renal calcifications in Npt2a null mice.

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Response of Npt2a knockout mice to dietary calcium and phosphorus.

http://www.wikidata.org/entity/Q33610690

description

2017 nî lūn-bûn

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2017 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2017 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

name

Response of Npt2a knockout mice to dietary calcium and phosphorus.

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Response of Npt2a knockout mice to dietary calcium and phosphorus.

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type

label

Response of Npt2a knockout mice to dietary calcium and phosphorus.

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Response of Npt2a knockout mice to dietary calcium and phosphorus.

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Response of Npt2a knockout mice to dietary calcium and phosphorus.

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Response of Npt2a knockout mice to dietary calcium and phosphorus.

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JOURNAL.PONE.0176232

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Response of Npt2a knockout mice to dietary calcium and phosphorus

@en

P2093

Alyssa Chen

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Chuanlong Zhu

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Daniel Caballero

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Harald Jüppner

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Julian Ponsetto

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Jun Guo

http://www.w3.org/2001/XMLSchema#string

Marie Demay

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Yuwen Li

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P2860

SLC34A3 mutations in patients with hereditary hypophosphatemic rickets with hypercalciuria predict a key role for the sodium-phosphate cotransporter NaPi-IIc in maintaining phosphate homeostasis

Hereditary hypophosphatemic rickets with hypercalciuria is caused by mutations in the sodium-phosphate cotransporter gene SLC34A3.

Idiopathic calcium nephrolithiasis: a review of pathogenic mechanisms in the light of genetic studies

ENPP1-Fc prevents mortality and vascular calcifications in rodent model of generalized arterial calcification of infancy

Autosomal-Recessive Mutations in SLC34A1 Encoding Sodium-Phosphate Cotransporter 2A Cause Idiopathic Infantile Hypercalcemia

Renal tubular acidosis: the clinical entity

Targeted inactivation of Npt2 in mice leads to severe renal phosphate wasting, hypercalciuria, and skeletal abnormalities

Genetic associations at 53 loci highlight cell types and biological pathways relevant for kidney function.

Genetic modulation of nephrocalcinosis in mouse models of ectopic mineralization: the Abcc6(tm1Jfk) and Enpp1(asj) mutant mice

Association of eGFR-Related Loci Identified by GWAS with Incident CKD and ESRD.

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e0176232

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scholarly article

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10.1371/JOURNAL.PONE.0176232

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4

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12

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Clemens Bergwitz

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2017-04-27T00:00:00Z

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28448530

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5407772

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