Yersinia pestis two-component gene regulatory systems promote survival in human neutrophils - Wikidata - awgldk - TriplyDB

Yersinia pestis two-component gene regulatory systems promote survival in human neutrophils

Yersinia pestis two-component gene regulatory systems promote survival in human neutrophils

http://www.wikidata.org/entity/Q33613887

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Environmental Regulation of Yersinia Pathophysiology Antibiotic development challenges: the various mechanisms of action of antimicrobial peptides and of bacterial resistance A direct link between the global regulator PhoP and the Csr regulon in Y. pseudotuberculosis through the small regulatory RNA CsrC Omics strategies for revealing Yersinia pestis virulence Yersinia pestis survival and replication within human neutrophil phagosomes and uptake of infected neutrophils by macrophages.Neutrophils are resistant to Yersinia YopJ/P-induced apoptosis and are protected from ROS-mediated cell death by the type III secretion system Influence of PhoP and intra-species variations on virulence of Yersinia pseudotuberculosis during the natural oral infection route.Expression of signal transduction system encoding genes of Yersinia pseudotuberculosis IP32953 at 28°C and 3°C.Molecular characterization of transcriptional regulation of rovA by PhoP and RovA in Yersinia pestis Role of a new intimin/invasin-like protein in Yersinia pestis virulence.The KdpD/KdpE two-component system: integrating K⁺ homeostasis and virulence.Lipopolysaccharide Biosynthesis Genes of Yersinia pseudotuberculosis Promote Resistance to Antimicrobial Chemokines Role of the PhoP-PhoQ gene regulatory system in adaptation of Yersinia pestis to environmental stress in the flea digestive tract.Early sensing of Yersinia pestis airway infection by bone marrow cells.OmpR, a response regulator of the two-component signal transduction pathway, influences inv gene expression in Yersinia enterocolitica O9.Bacterial Evasion of Host Antimicrobial Peptide Defenses Induction of the Yersinia pestis PhoP-PhoQ regulatory system in the flea and its role in producing a transmissible infection.Intranasal prophylaxis with CpG oligodeoxynucleotide can protect against Yersinia pestis infection.The Pathogenicity of Pseudomonas syringae MB03 against Caenorhabditis elegans and the Transcriptional Response of Nematicidal Genes upon Different Nutritional Conditions.Yersinia pestis subverts the dermal neutrophil response in a mouse model of bubonic plague.The role of reactive-oxygen-species in microbial persistence and inflammation.Host Defense and the Airway Epithelium: Frontline Responses That Protect against Bacterial Invasion and Pneumonia.The Two-Component System CpxRA Negatively Regulates the Locus of Enterocyte Effacement of Enterohemorrhagic Escherichia coli Involving σ(32) and Lon protease The interacting Cra and KdpE regulators are involved in the expression of multiple virulence factors in enterohemorrhagic Escherichia coli.The Role of OmpR in the Expression of Genes of the KdgR Regulon Involved in the Uptake and Depolymerization of Oligogalacturonides in Yersinia enterocolitica Characterization of Yersinia pestis Interactions with Human Neutrophils In vitro.The CpxRA two-component system is essential for Citrobacter rodentium virulence.A Novel RAYM_RS09735/RAYM_RS09740 Two-Component Signaling System Regulates Gene Expression and Virulence in Riemerella anatipestifer.Adaptation to Potassium-Limitation Is Essential for Acinetobacter baumannii Pneumonia Pathogenesis.Host Antimicrobial Peptides in Bacterial Homeostasis and Pathogenesis of Disease.Extracellular DNA-induced antimicrobial peptide resistance mechanisms in Pseudomonas aeruginosa.Impact of Gentamicin Concentration and Exposure Time on Intracellular Yersinia pestis.IgG keeps virulent Salmonella from evading dendritic cell uptake.Insect Immunity to Entomopathogenic Nematodes and Their Mutualistic Bacteria.

P2860

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P2860

Yersinia pestis two-component gene regulatory systems promote survival in human neutrophils

http://www.wikidata.org/entity/Q33613887

description

2009 nî lūn-bûn

@nan

2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

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2009年論文

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2009年论文

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name

Yersinia pestis two-component ...... survival in human neutrophils

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Yersinia pestis two-component ...... survival in human neutrophils

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type

label

Yersinia pestis two-component ...... survival in human neutrophils

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Yersinia pestis two-component ...... survival in human neutrophils

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Yersinia pestis two-component ...... survival in human neutrophils

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Yersinia pestis two-component ...... survival in human neutrophils

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Infection and Immunity

P1476

Yersinia pestis two-component ...... survival in human neutrophils

@en

P2093

Jason L O'Loughlin

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Justin L Spinner

http://www.w3.org/2001/XMLSchema#string

Scott A Minnich

http://www.w3.org/2001/XMLSchema#string

Scott D Kobayashi

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P2860

Genome sequence of Yersinia pestis KIM

Killing activity of neutrophils is mediated through activation of proteases by K+ flux

The pleiotropic two-component regulatory system PhoP-PhoQ

KdpD and KdpE, proteins that control expression of the kdpABC operon, are members of the two-component sensor-effector class of regulators

One-step inactivation of chromosomal genes in Escherichia coli K-12 using PCR products

Neutrophil granules and secretory vesicles in inflammation

Variation in lipid A structure in the pathogenic yersiniae.

Pathogenesis of Yersinia pestis infection in BALB/c mice: effects on host macrophages and neutrophils.

Mechanism of the binding, insertion and destabilization of phospholipid bilayer membranes by alpha-helical antimicrobial and cell non-selective membrane-lytic peptides.

The response regulator PhoP is important for survival under conditions of macrophage-induced stress and virulence in Yersinia pestis.

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773-782

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scholarly article

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10.1128/IAI.00718-09

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P433

2

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78

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P577

2009-11-23T00:00:00Z

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19933831

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Yersinia pestis

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2812203

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