Gain of chromosome 3q defines the transition from severe dysplasia to invasive carcinoma of the uterine cervix - Wikidata - awgldk - TriplyDB

Gain of chromosome 3q defines the transition from severe dysplasia to invasive carcinoma of the uterine cervix

Gain of chromosome 3q defines the transition from severe dysplasia to invasive carcinoma of the uterine cervix

http://www.wikidata.org/entity/Q33632013

about

Ultra-high throughput sequencing-based small RNA discovery and discrete statistical biomarker analysis in a collection of cervical tumours and matched controls Identification of novel candidate target genes, including EPHB3, MASP1 and SST at 3q26.2-q29 in squamous cell carcinoma of the lung Duplication of ATR inhibits MyoD, induces aneuploidy and eliminates radiation-induced G1 arrest SCCRO promotes glioma formation and malignant progression in mice Genomic amplification of the human telomerase gene (TERC) in pap smears predicts the development of cervical cancer Chromosomal imbalances in four new uterine cervix carcinoma derived cell lines Chromosomal amplifications, 3q gain and deletions of 2q33-q37 are the frequent genetic changes in cervical carcinoma The molecular basis of lung cancer: molecular abnormalities and therapeutic implications Chromosomal aberrations in patients with head and neck squamous cell carcinoma do not vary based on severity of tobacco/alcohol exposure.Microarray comparative genomic hybridization detection of chromosomal imbalances in uterine cervix carcinoma.Patterns of Chromosomal Aberrations in Solid Tumors The quiescent cellular state is Arf/p53-dependent and associated with H2AX downregulation and genome stability The consequences of chromosomal aneuploidy on the transcriptome of cancer cells Inhibition of Chk1 kills tetraploid tumor cells through a p53-dependent pathway Gene network reconstruction reveals cell cycle and antiviral genes as major drivers of cervical cancer Multipolar mitosis of tetraploid cells: inhibition by p53 and dependency on Mos.Protein expression patterns in primary carcinoma of the vagina.Gene dosage, expression, and ontology analysis identifies driver genes in the carcinogenesis and chemoradioresistance of cervical cancer.Distinct cytogenetic alterations in squamous intraepithelial lesions of the cervix revealed by laser-assisted microdissection and comparative genomic hybridization.Differential tissue-specific protein markers of vaginal carcinoma.Human Papilloma Virus (HPV) in head and neck region: review of literature.Analysis of genetic copy number changes in cervical disease progression.Genomic changes defining the genesis, progression, and malignancy potential in solid human tumors: a phenotype/genotype correlation.Impact of gene dosage on gene expression, biological processes and survival in cervical cancer: a genome-wide follow-up study.The molecular genetics of cervical carcinoma.Viral E6-E7 transcription in the basal layer of organotypic cultures without apparent p21cip1 protein precedes immortalization of human papillomavirus type 16- and 18-transfected human keratinocytes p63 and p73 in tumor suppression and promotion.Expression of the HPV18/E6 oncoprotein induces DNA damage.The guanine nucleotide exchange factor (GEF) Ect2 is an oncogene in human cancer.Contemporary theories of cervical carcinogenesis: the virus, the host, and the stem cell.Differential deletions of chromosome 3p are associated with the development of uterine cervical carcinoma in Indian patients Progressive 3q amplification consistently targets SOX2 in preinvasive squamous lung cancer.Onset of quiescence following p53 mediated down-regulation of H2AX in normal cells.A Comprehensive Review of Dysregulated miRNAs Involved in Cervical Cancer.Multiple centrosomes arise from tetraploidy checkpoint failure and mitotic centrosome clusters in p53 and RB pocket protein-compromised cells.Increase in viral load, viral integration, and gain of telomerase genes during uterine cervical carcinogenesis can be simultaneously assessed by the HPV 16/18 MLPA-assay.Amplified genes may be overexpressed, unchanged, or downregulated in cervical cancer cell lines.TP53 and PIK3CA gene mutations in adenocarcinoma, squamous cell carcinoma and high-grade intraepithelial neoplasia of the cervix.Surrogate endpoint biomarkers and their modulation in cervical chemoprevention trials.Inactivation of SLIT2-ROBO1/2 pathway in premalignant lesions of uterine cervix: clinical and prognostic significances

P2860

Q21245318-391FBF1B-EF9D-4A23-9AAC-79B5482D0F8D Q21260385-16B813FD-42A3-4BED-9767-AF1397AB8537 Q24317123-17EC6318-E37A-4A95-90D0-CAEF3BD29DFA Q24633442-82617E37-7ED1-45E9-9B37-44DBD32612CD Q24675639-61C6E884-427E-4636-B993-F8DF7838894D Q24792376-03DD7834-C140-43F7-8669-223DB428EBC0 Q24799110-D737C5A1-C474-4EB6-A0EB-0A319FD48E22 Q24802445-72A308A9-C98D-449F-B643-9D796B71ECC2 Q24803764-C06E2E32-3E1B-4AEF-A1D4-E41DB22FEAEB Q24813298-80D57CA8-3CCB-440E-8B4E-941C4DB607B5 Q26786127-EDBD7712-3719-4A48-AEB9-42BD637363B2 Q26864097-2EE6762C-0733-4A6C-A184-42D01B7AB573 Q26995532-6745027D-70ED-4284-87D4-CE249DF39576 Q27302281-7E6E3D92-2ADF-4FCF-8676-BFB9AA168D69 Q28119171-DD2A1DDB-21A0-44DF-AEED-12055A9D03C4 Q30494172-8009A3BA-DB92-4478-BBE3-F716C4C0E49D Q30763429-7AA59A1D-7EFB-461A-9E22-8F0515AC7BF0 Q30884181-ECAC7914-B4C9-4257-95B9-00EB1EB5BF52 Q31920793-519E59AB-90E9-4061-8748-B24DAB37A933 Q33430245-9CC0A63A-A605-4D04-A97B-A9B1B531F7CE Q33626377-C308F763-813C-405B-838A-DEEB89E874B8 Q33659642-82489406-FA07-4575-A802-6A0CD74EAD4C Q33668711-C72D3D97-0AC0-4EB9-BAD8-68354DEDEDFD Q33690029-63B57B5D-664C-4B6D-AF68-A6E48D29FD64 Q33724928-BEE55C1B-1AB0-4C77-B0EB-9CD617E8AAD7 Q33782160-9CAA1208-6ACD-4208-96D1-D6E9832D258A Q33791424-7ABD34CA-66AA-4365-A323-03327C22CC9D Q33811217-CF6D4EBF-4C33-4620-B7F9-EABC81F2B4F6 Q33827549-8F0E9216-8DC9-412F-B4D1-024E111528F4 Q33886737-BDBE4B9F-A9EE-4019-8A6F-1A8F7254CCFD Q33919221-48D27ECD-4A7F-4F3C-8D2D-1D67D3150F72 Q33988144-8FACC964-B27A-40D6-ACAD-6422CE653ADF Q33999624-9B72AB3A-99FB-4743-B48E-AC1253DDF668 Q34049716-F1ECB6F2-845B-4D43-97D5-3DC204A867B9 Q34075825-39C24B5E-61AD-474D-B136-CD32A48CB9E9 Q34162496-13B0C050-68CB-4381-B952-4636D3CDFF64 Q34193299-8F616CAF-44EE-435E-ABFC-CD48C899B563 Q34239911-487B5D6C-4374-47C1-BC9F-E7C217771D8E Q34240418-AC056DDF-BBCC-4ED6-9A30-12A9015F9F3A Q34311871-FDBA9B0D-5642-412F-BDC7-2B1AE3847DD8

P2860

Gain of chromosome 3q defines the transition from severe dysplasia to invasive carcinoma of the uterine cervix

http://www.wikidata.org/entity/Q33632013

description

1996 nî lūn-bûn

@nan

1996 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

1996 թվականի հունվարին հրատարակված գիտական հոդված

@hy

1996年の論文

@ja

1996年論文

@yue

1996年論文

@zh-hant

1996年論文

@zh-hk

1996年論文

@zh-mo

1996年論文

@zh-tw

1996年论文

@wuu

name

Gain of chromosome 3q defines ...... arcinoma of the uterine cervix

@ast

Gain of chromosome 3q defines ...... arcinoma of the uterine cervix

@en

type

label

Gain of chromosome 3q defines ...... arcinoma of the uterine cervix

@ast

Gain of chromosome 3q defines ...... arcinoma of the uterine cervix

@en

prefLabel

Gain of chromosome 3q defines ...... arcinoma of the uterine cervix

@ast

Gain of chromosome 3q defines ...... arcinoma of the uterine cervix

@en

P1433

Q33632013-A72BE686-26D7-4266-A761-E748A421F115

P1476

Q33632013-207A54D3-6EE2-4570-9A9C-4242CD462482

P2093

Q33632013-3C52BD9D-7BE1-4D2B-880E-2BDB5F532DB2

Q33632013-46550393-F1B5-4139-BEC1-86BE36F50028

Q33632013-5C24E3DF-A522-40E3-9CBD-983FC5EBA745

Q33632013-647BC7CB-60A1-4F07-9ED4-3302B594830C

Q33632013-819DEF35-5672-4C67-9C2B-0E52EAA34925

Q33632013-8F0051BD-73E7-493F-BA4F-AEB5E6A700DF

Q33632013-CA100425-E80D-44C0-B266-5F4995F389F7

Q33632013-E6ABC126-F543-406E-88D0-953388782272

P2860

Q33632013-0EB14106-A7DD-450D-A47A-4AC1AF714E7D

Q33632013-131BB8F9-D23B-4F91-9203-CAE2648619AC

Q33632013-1C596BE5-8497-417A-9E36-22E8728E845A

Q33632013-1E100E7A-2DF6-4259-AB38-F37BE71E40FC

Q33632013-2D5BB184-0410-4A10-B797-8F54ADDA9910

Q33632013-3271CBF2-B953-42E0-8E84-F797E25E529C

Q33632013-488DF551-BF66-40E1-86EF-FE3CEABDE623

Q33632013-495BD70E-E103-43D5-BA1E-0B4ED808CFD7

Q33632013-69152DDB-6D17-4AFE-A513-4C2881CF26FA

Q33632013-70DC486F-5E26-4623-B583-468CA36EFF9B

P304

Q33632013-61DA254A-AA2E-43E1-B31D-B629F9DFFBB2

P31

Q33632013-1330386E-8271-4C71-918E-3ECED35B5ECA

P356

Q33632013-A9576EE6-A629-45AD-B0A8-3840EE6AB744

P407

Q33632013-AE62E831-F7A8-4CE8-9870-BE4B75F83801

P433

Q33632013-82B30649-8E8C-4D11-9B50-B9A5160F692F

P478

Q33632013-F0D8FF4D-DC8D-48D9-BEC6-A67216AEAA75

P577

Q33632013-FC3526BE-5F47-4036-81CF-F2AABF84CF1F

P5875

Q33632013-B5C3F6FC-28D0-47B4-9EF9-6D47EDF9F302

P698

Q33632013-75934CE1-5585-485F-907B-1C7EBA3BA5F3

P932

Q33632013-73C587CC-34C5-430B-AE59-26B0A0727B8C

P356

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Gain of chromosome 3q defines ...... arcinoma of the uterine cervix

@en

P2093

Auer G

http://www.w3.org/2001/XMLSchema#string

Blegen H

http://www.w3.org/2001/XMLSchema#string

Heselmeyer K

http://www.w3.org/2001/XMLSchema#string

Ried T

http://www.w3.org/2001/XMLSchema#string

Schröck E

http://www.w3.org/2001/XMLSchema#string

Shah K

http://www.w3.org/2001/XMLSchema#string

Steinbeck R

http://www.w3.org/2001/XMLSchema#string

du Manoir S

http://www.w3.org/2001/XMLSchema#string

P2860

A genetic model for colorectal tumorigenesis

The clonal evolution of tumor cell populations

The E6 oncoprotein encoded by human papillomavirus types 16 and 18 promotes the degradation of p53

The human papilloma virus-16 E7 oncoprotein is able to bind to the retinoblastoma gene product

Comparative genomic hybridization for molecular cytogenetic analysis of solid tumors

Prevalence of human papillomavirus in cervical cancer: a worldwide perspective. International biological study on cervical cancer (IBSCC) Study Group.

Loss of heterozygosity for alleles on chromosome II in cervical carcinoma.

Loss of heterozygosity in cervical carcinoma: subchromosomal localization of a putative tumor-suppressor gene to chromosome 11q22-q24

Deletion and translocation of chromosome 11q13 sequences in cervical carcinoma cell lines.

Comparative genomic hybridization of human malignant gliomas reveals multiple amplification sites and nonrandom chromosomal gains and losses

P304

479-484

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1073/PNAS.93.1.479

http://www.w3.org/2001/XMLSchema#string

P407

P433

1

http://www.w3.org/2001/XMLSchema#string

P478

93

http://www.w3.org/2001/XMLSchema#string

P577

1996-01-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

14647191

http://www.w3.org/2001/XMLSchema#string

P698

8552665

http://www.w3.org/2001/XMLSchema#string

P932

40262

http://www.w3.org/2001/XMLSchema#string