Substance P signaling contributes to granuloma formation in Taenia crassiceps infection, a murine model of cysticercosis - Wikidata - awgldk - TriplyDB

Substance P signaling contributes to granuloma formation in Taenia crassiceps infection, a murine model of cysticercosis

Substance P signaling contributes to granuloma formation in Taenia crassiceps infection, a murine model of cysticercosis

http://www.wikidata.org/entity/Q33635087

about

Somatostatin negatively regulates parasite burden and granulomatous responses in cysticercosis Neurokinin-1 receptor: functional significance in the immune system in reference to selected infections and inflammation.Solitary cysticercus granuloma.Role of substance P in the regulation of glucose metabolism via insulin signaling-associated pathways.The Therapeutic Potential of Targeting Substance P/NK-1R Interactions in Inflammatory CNS Disorders Aprepitant limits in vivo neuroinflammatory responses in a rhesus model of Lyme neuroborreliosis.Development of the S3Pvac vaccine against murine Taenia crassiceps cysticercosis: a historical review.Substance P and the regulation of inflammation in infections and inflammatory bowel disease.Role of Substance P Neuropeptide in Inflammation, Wound Healing, and Tissue Homeostasis.Human microglia and astrocytes constitutively express the neurokinin-1 receptor and functionally respond to substance P.

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P2860

Substance P signaling contributes to granuloma formation in Taenia crassiceps infection, a murine model of cysticercosis

http://www.wikidata.org/entity/Q33635087

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Substance P signaling contribu ...... murine model of cysticercosis

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Substance P signaling contribu ...... murine model of cysticercosis

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Substance P signaling contribu ...... murine model of cysticercosis

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Substance P signaling contribu ...... murine model of cysticercosis

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Substance P signaling contribu ...... murine model of cysticercosis

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Substance P signaling contribu ...... murine model of cysticercosis

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Journal of Biomedicine and Biotechnology

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Substance P signaling contribu ...... murine model of cysticercosis

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Armandina Garza

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Balaji Viswanathan

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David J Tweardy

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Joel Weinstock

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P2860

Current consensus guidelines for treatment of neurocysticercosis

Human monocytes and macrophages express substance P and neurokinin-1 receptor

TNF-alpha increases expression of IL-6 and ICAM-1 genes through activation of NF-kappaB in osteoblast-like ROS17/2.8 cells

The role of the granuloma in expansion and dissemination of early tuberculous infection.

Sequential expression of the neuropeptides substance P and somatostatin in granulomas associated with murine cysticercosis.

Taenia solium taeniosis/cysticercosis in Asia: epidemiology, impact and issues.

Epilepsy in developing countries.

The neuropeptide substance P activates p38 mitogen-activated protein kinase resulting in IL-6 expression independently from NF-kappa B.

Substance P induces TNF-alpha and IL-6 production through NF kappa B in peritoneal mast cells.

Absence of the SP/SP receptor circuitry in the substance P-precursor knockout mice or SP receptor, neurokinin (NK)1 knockout mice leads to an inhibited cytokine response in granulomas associated with murine Taenia crassiceps infection.

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2010

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