Two nucleotides immediately upstream of the essential A6G3 slippery sequence modulate the pattern of G insertions during Sendai virus mRNA editing.
about
"Rule of six": how does the Sendai virus RNA polymerase keep count?Picornavirus RNA polyadenylation by 3D(pol), the viral RNA-dependent RNA polymeraseVesicular Stomatitis Virus mRNA Capping Machinery Requires Specific cis-Acting Signals in the RNAInfluenza A virus RNA polymerase has the ability to stutter at the polyadenylation site of a viral RNA template during RNA replication.Sendai virus C proteins counteract the interferon-mediated induction of an antiviral stateSendai virus gene start signals are not equivalent in reinitiation capacity: moderation at the fusion protein gene.Polymerase slippage at vesicular stomatitis virus gene junctions to generate poly(A) is regulated by the upstream 3'-AUAC-5' tetranucleotide: implications for the mechanism of transcription termination.Chemical modification of nucleotide bases and mRNA editing depend on hexamer or nucleoprotein phase in Sendai virus nucleocapsids.Ambisense sendai viruses are inherently unstable but are useful to study viral RNA synthesis.Distinct and overlapping roles of Nipah virus P gene products in modulating the human endothelial cell antiviral response.Antagonism of innate immunity by paramyxovirus accessory proteins.Nonlinearity in genetic decoding: homologous DNA replicase genes use alternatives of transcriptional slippage or translational frameshiftingSendai virus as a backbone for vaccines against RSV and other human paramyxovirusesNipah virus edits its P gene at high frequency to express the V and W proteins.A novel sweet potato potyvirus open reading frame (ORF) is expressed via polymerase slippage and suppresses RNA silencing.La Piedad Michoacán Mexico Virus V protein antagonizes type I interferon response by binding STAT2 protein and preventing STATs nuclear translocationPolymerases of paramyxoviruses and pneumoviruses.The versatility of paramyxovirus RNA polymerase stuttering.Identification of a mutation in editing of defective Newcastle disease virus recombinants that modulates P-gene mRNA editing and restores virus replication and pathogenicity in chicken embryos.Mutational analysis of the Potyviridae transcriptional slippage site utilized for expression of the P3N-PIPO and P1N-PISPO proteins.
P2860
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P2860
Two nucleotides immediately upstream of the essential A6G3 slippery sequence modulate the pattern of G insertions during Sendai virus mRNA editing.
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Two nucleotides immediately up ...... ing Sendai virus mRNA editing.
@ast
Two nucleotides immediately up ...... ing Sendai virus mRNA editing.
@en
type
label
Two nucleotides immediately up ...... ing Sendai virus mRNA editing.
@ast
Two nucleotides immediately up ...... ing Sendai virus mRNA editing.
@en
prefLabel
Two nucleotides immediately up ...... ing Sendai virus mRNA editing.
@ast
Two nucleotides immediately up ...... ing Sendai virus mRNA editing.
@en
P2093
P2860
P1433
P1476
Two nucleotides immediately up ...... ring Sendai virus mRNA editing
@en
P2093
D Kolakofsky
S Hausmann
P2860
P304
P577
1999-01-01T00:00:00Z