Microarray analysis of hippocampal CA1 neurons implicates early endosomal dysfunction during Alzheimer's disease progression.
about
Mild cognitive impairment: pathology and mechanisms.G Protein-Coupled Receptors (GPCRs) in Alzheimer's Disease: A Focus on BACE1 Related GPCRsThe endosomal-lysosomal system: from acidification and cargo sorting to neurodegenerationThe synaptic maintenance problem: membrane recycling, Ca2+ homeostasis and late onset degenerationCommon transcriptional signatures in brain tissue from patients with HIV-associated neurocognitive disorders, Alzheimer's disease, and Multiple SclerosisAmyloid precursor protein-mediated endocytic pathway disruption induces axonal dysfunction and neurodegenerationRab GTPases, membrane trafficking and diseasesTrisomy for synaptojanin1 in Down syndrome is functionally linked to the enlargement of early endosomesProtective effects of positive lysosomal modulation in Alzheimer's disease transgenic mouse modelsMonoacylglycerol lipase inhibitor JZL184 improves behavior and neural properties in Ts65Dn mice, a model of down syndromePlasma BDNF levels vary in relation to body weight in femalesZebrafish Rab5 proteins and a role for Rab5ab in nodal signalling.Locus coeruleus cellular and molecular pathology during the progression of Alzheimer's disease.Microarray analyses of laser-captured hippocampus reveal distinct gray and white matter signatures associated with incipient Alzheimer's disease.Lysosomal dysfunction in the brain of a mouse model with intraneuronal accumulation of carboxyl terminal fragments of the amyloid precursor protein.Brain gene expression patterns differentiate mild cognitive impairment from normal aged and Alzheimer's disease.Deep sleep and parietal cortex gene expression changes are related to cognitive deficits with age.Gene expression levels assessed by CA1 pyramidal neuron and regional hippocampal dissections in Alzheimer's diseaseMicroarray analysis of CA1 pyramidal neurons in a mouse model of tauopathy reveals progressive synaptic dysfunction.Epigenetic memory: the Lamarckian brainExpression profile analysis of vulnerable CA1 pyramidal neurons in young-Middle-Aged Ts65Dn mice.Regional selectivity of rab5 and rab7 protein upregulation in mild cognitive impairment and Alzheimer's diseaseA Review of Recent Advances in Neuroprotective Potential of 3-N-Butylphthalide and Its DerivativesThe polyphenol oleuropein aglycone protects TgCRND8 mice against Aß plaque pathology.Expression profile analysis of hippocampal CA1 pyramidal neurons in aged Ts65Dn mice, a model of Down syndrome (DS) and Alzheimer's disease (AD).Autophagy failure in Alzheimer's disease--locating the primary defect.Identification and quantification of amyloid beta-related peptides in human plasma using matrix-assisted laser desorption/ionization time-of-flight mass spectrometry.Reduction of β-amyloid and γ-secretase by calorie restriction in female Tg2576 miceGender differences in neurotrophin and glutamate receptor expression in cholinergic nucleus basalis neurons during the progression of Alzheimer's disease.A systems biology investigation of neurodegenerative dementia reveals a pivotal role of autophagy.Upregulation of select rab GTPases in cholinergic basal forebrain neurons in mild cognitive impairment and Alzheimer's disease.Hippocampal endosomal, lysosomal, and autophagic dysregulation in mild cognitive impairment: correlation with aβ and tau pathologyHook proteins: association with Alzheimer pathology and regulatory role of hook3 in amyloid beta generation7,8-dihydroxyflavone, a small-molecule TrkB agonist, reverses memory deficits and BACE1 elevation in a mouse model of Alzheimer's disease.Expression profiling in neuropsychiatric disorders: emphasis on glutamate receptors in bipolar disorderComparative lipidomic analysis of mouse and human brain with Alzheimer disease.TrkB reduction exacerbates Alzheimer's disease-like signaling aberrations and memory deficits without affecting β-amyloidosis in 5XFAD mice.Age-related neurodegeneration and memory loss in down syndrome.Significance of transcytosis in Alzheimer's disease: BACE1 takes the scenic route to axons.Strain-Specific Altered Regulatory Response of Rab7a and Tau in Creutzfeldt-Jakob Disease and Alzheimer's Disease.
P2860
Q24631255-350B022E-9460-4FCE-9F88-A25BC0DF61FBQ26752403-32733E18-F716-47D8-8F98-3C9EBC1F6ACDQ26785986-0E582D36-1091-4B01-AC85-276EE51C0186Q27000356-5F21979A-5B2A-454B-A6C9-CECB30FD7BAFQ27007041-12F3C23F-0903-428E-87AE-363F9DB97B4BQ27331605-4AAAA973-4B94-4923-888C-B2E417A8090CQ27687621-295FDCE6-0BFE-42B4-88AC-D476BA0957DDQ28118988-418460AB-B911-41B1-ABD7-E20B5338898CQ28478524-764257F6-FD68-4C2C-942A-0D18B556E251Q28542515-39E6AA56-8898-471C-A0D1-00FADFE27E6DQ28727632-9435CEF4-D9F7-4072-9BD9-C4EA12DEDAC8Q30615569-A84D3F80-B6F9-41E8-AE77-3F74B1A7DE6AQ30835713-3C9C1CA3-F21C-4CF9-89B1-DB241CA648F4Q31023394-F3945A84-4111-4474-9EAC-B1A59F66F5B0Q33603071-DE03EE56-3AA7-46D4-939D-43100092DE46Q33792226-27678279-CC57-444A-B2C4-62E306C6E90FQ33869293-EC870DF6-2515-406F-BDEF-1ECD9A1549EDQ33984420-F7674171-03B7-4A95-A432-8D6D1A102C0DQ34073334-917D0017-2FF6-4C74-818E-C80CDDEE8C49Q34323841-1F878556-F9A5-44A1-8BC8-E6C6EDAAB277Q34514070-58A0C758-2EC7-4868-AE69-510AB7DD38DFQ34543633-675BBB38-317E-494A-9023-225EFCD3D739Q34548561-51BF3DFB-31EA-4F11-80C3-A5E7D737AA2FQ34950366-7ECC7906-559E-4CB0-8E31-1558C3BFFBE6Q34979826-4EA05E19-6B45-4166-9E5A-25020EEF6AB1Q34987827-D197DAA0-1AE6-4BA8-B58A-4DD6222AB532Q35118876-40146D7A-D3D3-4CCE-B012-084342ECD6DDQ35137488-02364507-A29E-491D-9876-0C885F6CC4ABQ35162941-A268E198-E826-4C05-9F32-3A29D3D885C2Q35183471-33A92476-992F-48D6-AB42-E27276C134ECQ35186435-C399C5AB-22AA-4B6D-B4C2-F0A4442207EDQ35196299-3D62C96E-93EB-46A8-BBF8-CB9C85928E7DQ35208328-688D987C-8447-49ED-B74D-765A7BF11341Q35622640-801AB3EC-BFCA-4514-9C35-8783265168A0Q35661134-32711BC6-D28E-40CC-906D-D0B8CE7D34CEQ35710041-6385D5E1-1935-492E-A080-50BE88D7543BQ35753185-3EDFB670-E3AB-4995-A6DC-8939913D6DE5Q35867093-65A98CAF-7913-4184-A75B-FEF7B6F1BE08Q35883077-FBD40509-5514-445F-97B7-6B52D51A11ABQ35894510-0A6BC83A-0F60-46F8-BA5C-BD1F0A55DC7F
P2860
Microarray analysis of hippocampal CA1 neurons implicates early endosomal dysfunction during Alzheimer's disease progression.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Microarray analysis of hippoca ...... zheimer's disease progression.
@ast
Microarray analysis of hippoca ...... zheimer's disease progression.
@en
type
label
Microarray analysis of hippoca ...... zheimer's disease progression.
@ast
Microarray analysis of hippoca ...... zheimer's disease progression.
@en
prefLabel
Microarray analysis of hippoca ...... zheimer's disease progression.
@ast
Microarray analysis of hippoca ...... zheimer's disease progression.
@en
P2093
P2860
P1476
Microarray analysis of hippoca ...... zheimer's disease progression.
@en
P2093
Anne M Cataldo
Elliott J Mufson
Joanne Wuu
Melissa J Alldred
Rachael L Neve
Ralph A Nixon
Scott E Counts
Shaoli Che
Ying Jiang
P2860
P304
P356
10.1016/J.BIOPSYCH.2010.05.030
P407
P577
2010-07-23T00:00:00Z