Mutations within the TNF-like core domain of RANKL impair osteoclast differentiation and activation
about
A Novel Therapeutic Peptide as a Partial Agonist of RANKL in Ischemic Stroke.Cytoskeleton-centric protein transportation by exosomes transforms tumor-favorable macrophages.Osteoblasts subjected to mechanical strain inhibit osteoclastic differentiation and bone resorption in a co-culture system.Bafilomycin A1 Attenuates Osteoclast Acidification and Formation, Accompanied by Increased Levels of SQSTM1/p62 Protein.A RANKL G278R mutation causing osteopetrosis identifies a functional amino acid essential for trimer assembly in RANKL and TNF
P2860
Mutations within the TNF-like core domain of RANKL impair osteoclast differentiation and activation
description
2008 nî lūn-bûn
@nan
2008 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Mutations within the TNF-like ...... differentiation and activation
@ast
Mutations within the TNF-like ...... differentiation and activation
@en
type
label
Mutations within the TNF-like ...... differentiation and activation
@ast
Mutations within the TNF-like ...... differentiation and activation
@en
prefLabel
Mutations within the TNF-like ...... differentiation and activation
@ast
Mutations within the TNF-like ...... differentiation and activation
@en
P2093
P2860
P50
P356
P1476
Mutations within the TNF-like ...... differentiation and activation
@en
P2093
Cathy Wang
Jamie We-Yin Tan
Jillian Cornish
Ming-Hao Zheng
P2860
P356
10.1210/ME.2007-0465
P577
2008-11-13T00:00:00Z