Gram-positive sepsis. Mechanisms and differences from gram-negative sepsis.
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Recognition of Staphylococcus aureus by the innate immune system.Gene expression-based classifiers identify Staphylococcus aureus infection in mice and humansSepsis pathophysiology and anesthetic considerationThe involvement of CD14 in the activation of human monocytes by peptidoglycan monomersPeptidoglycan and lipoteichoic acid modify monocyte phenotype in human whole bloodGene expression profiling of porcine mammary epithelial cells after challenge with Escherichia coli and Staphylococcus aureus in vitroIncreased β-haemolytic group A streptococcal M6 serotype and streptodornase B-specific cellular immune responses in Swedish narcolepsy cases.Posterior reversible encephalopathy syndrome, part 2: controversies surrounding pathophysiology of vasogenic edema.Effect of deficiency of tumor necrosis factor alpha or both of its receptors on Streptococcus pneumoniae central nervous system infection and peritonitis.Evaluation of the in vitro pyrogen test system based on proinflammatory cytokine release from human monocytes: comparison with a human whole blood culture test system and with the rabbit pyrogen test.Commercial preparations of lipoteichoic acid contain endotoxin that contributes to activation of mouse macrophages in vitroDifferential tumor necrosis factor alpha expression and release from peritoneal mouse macrophages in vitro in response to proliferating gram-positive versus gram-negative bacteria.Lipoteichoic acid from Staphylococcus aureus exacerbates respiratory disease in porcine respiratory coronavirus-infected pigsAnomalous role of tumor necrosis factor alpha in experimental enterococcal infection.The many faces of Staphylococcus aureus infection. Recognizing and managing its life-threatening manifestations.Mechanisms of tissue injury in sepsis: contrasts between gram positive and gram negative infection.Involvement of NO in the failure of neutrophil migration in sepsis induced by Staphylococcus aureus.Endogenous versus exogenous glucocorticoid responses to experimental bacterial sepsis.Sensing of Gram-positive bacteria in Drosophila: GNBP1 is needed to process and present peptidoglycan to PGRP-SATeichoic acids are not required for Streptococcus pneumoniae and Staphylococcus aureus cell walls to trigger the release of tumor necrosis factor by peripheral blood monocytesDifference in Resistance to Streptococcus pneumoniae Infection in MiceReceptors, mediators, and mechanisms involved in bacterial sepsis and septic shock.A continuum of anionic charge: structures and functions of D-alanyl-teichoic acids in gram-positive bacteriaCenter Variation in Intestinal Microbiota Prior to Late-Onset Sepsis in Preterm Infants.CYP3A-dependent drug metabolism is reduced in bacterial inflammation in miceIntravenous polyclonal IgM-enriched immunoglobulin therapy in sepsis: a review of clinical efficacy in relation to microbiological aetiology and severity of sepsis.Bench-to-bedside review: sepsis, severe sepsis and septic shock - does the nature of the infecting organism matter?Cellular mechanisms in sepsis.Early gene expression changes induced by the bacterial superantigen staphylococcal enterotoxin B and its modulation by a proteasome inhibitor.Lipoprotein lipase and hydrofluoric acid deactivate both bacterial lipoproteins and lipoteichoic acids, but platelet-activating factor-acetylhydrolase degrades only lipoteichoic acidsSepsis, complement and the dysregulated inflammatory response.Chronic Inflammation: Synergistic Interactions of Recruiting Macrophages (TAMs) and Eosinophils (Eos) with Host Mast Cells (MCs) and Tumorigenesis in CALTs. M-CSF, Suitable Biomarker for Cancer Diagnosis!Tailoring gut immune responses with lipoteichoic acid-deficient Lactobacillus acidophilus.Characterization of a monoclonal antibody that binds to an epitope on soluble bacterial peptidoglycan fragments.Staphylococcal enterotoxin B-induced acute inflammation is inhibited by dexamethasone: important role of CXC chemokines KC and macrophage inflammatory protein 2.Requirements of peptidoglycan structure that allow detection by the Drosophila Toll pathway.Mixed bacterial infection model of sepsis in rabbits and its application to evaluate superantigen-adsorbing device.Alveolar macrophage phagocytic activity is enhanced with LPS priming, and combined stimulation of LPS and lipoteichoic acid synergistically induce pro-inflammatory cytokines in pigs.Differential activation of the immune system by virulent Streptococcus pneumoniae strains determines recovery or death of the host.Effects of bacterial toxins on air-exposed cultured human respiratory sinus epithelium.
P2860
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P2860
Gram-positive sepsis. Mechanisms and differences from gram-negative sepsis.
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Gram-positive sepsis. Mechanisms and differences from gram-negative sepsis.
@ast
Gram-positive sepsis. Mechanisms and differences from gram-negative sepsis.
@en
type
label
Gram-positive sepsis. Mechanisms and differences from gram-negative sepsis.
@ast
Gram-positive sepsis. Mechanisms and differences from gram-negative sepsis.
@en
prefLabel
Gram-positive sepsis. Mechanisms and differences from gram-negative sepsis.
@ast
Gram-positive sepsis. Mechanisms and differences from gram-negative sepsis.
@en
P921
P1476
Gram-positive sepsis. Mechanisms and differences from gram-negative sepsis
@en
P2093
P304
P356
10.1016/S0891-5520(05)70082-9
P577
1999-06-01T00:00:00Z