Activation of AKT by O-linked N-acetylglucosamine induces vascular calcification in diabetes mellitus - Wikidata - awgldk - TriplyDB

Activation of AKT by O-linked N-acetylglucosamine induces vascular calcification in diabetes mellitus

Activation of AKT by O-linked N-acetylglucosamine induces vascular calcification in diabetes mellitus

http://www.wikidata.org/entity/Q33647822

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Transcription Factor Runx2 Promotes Aortic Fibrosis and Stiffness in Type 2 Diabetes Mellitus.Redox signaling in cardiovascular pathophysiology: A focus on hydrogen peroxide and vascular smooth muscle cells Store-operated calcium entry and diabetic complications Teaching the basics of autophagy and mitophagy to redox biologists--mechanisms and experimental approaches Increased activity of TNAP compensates for reduced adenosine production and promotes ectopic calcification in the genetic disease ACDC.Protein O-GlcNAcylation and cardiovascular (patho)physiology.Inhibition of FOXO1/3 promotes vascular calcification Vascular smooth muscle LRP6 limits arteriosclerotic calcification in diabetic LDLR-/- mice by restraining noncanonical Wnt signals MicroRNA-32 promotes calcification in vascular smooth muscle cells: Implications as a novel marker for coronary artery calcification.O-GlcNAcomic Profiling Identifies Widespread O-Linked β-N-Acetylglucosamine Modification (O-GlcNAcylation) in Oxidative Phosphorylation System Regulating Cardiac Mitochondrial Function Bidirectional Translation in Cardiovascular Calcification.Disruption of O-linked N-Acetylglucosamine Signaling Induces ER Stress and β Cell Failure.Nutrition in Cardioskeletal Health.Modifying vascular calcification in diabetes mellitus: contribution of O-GlcNAcylation.Are reactive oxygen species still the basis for diabetic complications?Akt/PKB: one kinase, many modifications.Pathophysiology of Vascular Calcification.The long non-coding RNA HOTAIR enhances pancreatic cancer resistance to TNF-related apoptosis-inducing ligand.Calmodulin antagonists promote TRA-8 therapy of resistant pancreatic cancer Where do we stand on vascular calcification?Pathology of Human Coronary and Carotid Artery Atherosclerosis and Vascular Calcification in Diabetes Mellitus.Arterial Calcification in Diabetes Mellitus: Preclinical Models and Translational Implications.O-GlcNAcylation and cardiovascular disease.Roles of platelet-derived growth factor in vascular calcification.Sphingosine 1-phosphate activation of ERM contributes to vascular calcification.Dietary potassium regulates vascular calcification and arterial stiffness.Increased O-GlcNAcylation of Endothelial Nitric Oxide Synthase Compromises the Anti-contractile Properties of Perivascular Adipose Tissue in Metabolic Syndrome.A GTPase-activating protein binding protein (G3BP1) / antiviral protein relay conveys arteriosclerotic Wnt signals in aortic smooth muscle cells.Regulation of Vascular Calcification by Growth Hormone-Releasing Hormone and its Agonists.AKT-independent activation of p38 MAP kinase promotes vascular calcification.FTI-277 inhibits smooth muscle cell calcification by up-regulating PI3K/Akt signaling and inhibiting apoptosis.Placental Vascular Calcification and Cardiovascular Health: It Is Time to Determine How Much of Maternal and Offspring Health Is Written in Stone Cross-Dysregulation of -GlcNAcylation and PI3K/AKT/mTOR Axis in Human Chronic Diseases O-GlcNAcylation mediates metastasis of cholangiocarcinoma through FOXO3 and MAN1A1

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Activation of AKT by O-linked N-acetylglucosamine induces vascular calcification in diabetes mellitus

http://www.wikidata.org/entity/Q33647822

description

2014 nî lūn-bûn

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2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի փետրվարին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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CIRCRESAHA.114.302968

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Circulation Research

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Activation of AKT by O-linked ...... ification in diabetes mellitus

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Hui Wu

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Jack M Heath

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John C Chatham

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Kaiyu Yuan

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Louis J Dell'Italia

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Silvio Litovsky

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Yabing Chen

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Yong Sun

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Active-site inhibitors of mTOR target rapamycin-resistant outputs of mTORC1 and mTORC2

Insulin-dependent activation of endothelial nitric oxide synthase is impaired by O-linked glycosylation modification of signaling proteins in human coronary endothelial cells

Crystal structure of an activated Akt/protein kinase B ternary complex with GSK3-peptide and AMP-PNP

Hyperglycemia-induced mitochondrial superoxide overproduction activates the hexosamine pathway and induces plasminogen activator inhibitor-1 expression by increasing Sp1 glycosylation

Vascular ossification-calcification in metabolic syndrome, type 2 diabetes mellitus, chronic kidney disease, and calciphylaxis-calcific uremic arteriolopathy: the emerging role of sodium thiosulfate

Molecular mechanism for the regulation of protein kinase B/Akt by hydrophobic motif phosphorylation

A potent mechanism-inspired O-GlcNAcase inhibitor that blocks phosphorylation of tau in vivo

Studies of streptozotocin-induced insulitis and diabetes

Hyperglycemia inhibits endothelial nitric oxide synthase activity by posttranslational modification at the Akt site

O-linked β-N-acetylglucosamine supports p38 MAPK activation by high glucose in glomerular mesangial cells

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114

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