Angiotensin-converting enzyme 2 overexpression protects against doxorubicin-induced cardiomyopathy by multiple mechanisms in rats. - Wikidata - awgldk - TriplyDB

Angiotensin-converting enzyme 2 overexpression protects against doxorubicin-induced cardiomyopathy by multiple mechanisms in rats.

Angiotensin-converting enzyme 2 overexpression protects against doxorubicin-induced cardiomyopathy by multiple mechanisms in rats.

http://www.wikidata.org/entity/Q33648557

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The sirtuin 6 prevents angiotensin II-mediated myocardial fibrosis and injury by targeting AMPK-ACE2 signaling.AMPK Phosphorylation of ACE2 in Endothelium Mitigates Pulmonary Hypertension.

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Angiotensin-converting enzyme 2 overexpression protects against doxorubicin-induced cardiomyopathy by multiple mechanisms in rats.

http://www.wikidata.org/entity/Q33648557

description

2017 nî lūn-bûn

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2017 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2017 թվականի ապրիլին հրատարակված գիտական հոդված

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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name

Angiotensin-converting enzyme ...... y multiple mechanisms in rats.

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Angiotensin-converting enzyme ...... y multiple mechanisms in rats.

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Angiotensin-converting enzyme ...... y multiple mechanisms in rats.

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Angiotensin-converting enzyme ...... y multiple mechanisms in rats.

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Bo Dong

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Hong-Yan Dai

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Hui Ma

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Jing Kong

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Jing-Jing Yang

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Jun-Long Li

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Nai-Hao Hei

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Wen-Jiang Yan

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Wen-Jing Liang

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Xin-Ran Cao

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HBP1 repression of the p47phox gene: cell cycle regulation via the NADPH oxidase

Hydrolysis of biological peptides by human angiotensin-converting enzyme-related carboxypeptidase

Angiotensin-converting enzyme 2 is an essential regulator of heart function

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Notch-1 mediated cardiac protection following embryonic and induced pluripotent stem cell transplantation in doxorubicin-induced heart failure

Schisandrin B prevents doxorubicin induced cardiac dysfunction by modulation of DNA damage, oxidative stress and inflammation through inhibition of MAPK/p53 signaling

Oral administration of an angiotensin-converting enzyme 2 activator ameliorates diabetes-induced cardiac dysfunction

Decreased Smad 7 expression contributes to cardiac fibrosis in the infarcted rat heart

Anthracyclines: molecular advances and pharmacologic developments in antitumor activity and cardiotoxicity

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