Traumatic brain injury results in disparate regions of chondroitin sulfate proteoglycan expression that are temporally limited
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Proteoglycans in the central nervous system: role in development, neural repair, and Alzheimer's diseaseEnriched housing enhances recovery of limb placement ability and reduces aggrecan-containing perineuronal nets in the rat somatosensory cortex after experimental strokeBrain stimulation: Neuromodulation as a potential treatment for motor recovery following traumatic brain injuryPericontusion axon sprouting is spatially and temporally consistent with a growth-permissive environment after traumatic brain injury.Chondroitinase ABC enhances pericontusion axonal sprouting but does not confer robust improvements in behavioral recoveryExpression of miRNAs and their cooperative regulation of the pathophysiology in traumatic brain injuryDiffuse traumatic axonal injury in the mouse induces atrophy, c-Jun activation, and axonal outgrowth in the axotomized neuronal populationAstrocyte roles in traumatic brain injury.Alterations in chondroitin sulfate proteoglycan expression occur both at and far from the site of spinal contusion injury.Cortical reorganization after experimental traumatic brain injury: a functional autoradiography studyChondroitinase enhances cortical map plasticity and increases functionally active sprouting axons after brain injuryAlterations in sulfated chondroitin glycosaminoglycans following controlled cortical impact injury in mice.Hitting a moving target: Basic mechanisms of recovery from acquired developmental brain injury.Bridging the Divide between Neuroprosthetic Design, Tissue Engineering and Neurobiology.Time dependent integration of matrix metalloproteinases and their targeted substrates directs axonal sprouting and synaptogenesis following central nervous system injury.Disconnection and hyper-connectivity underlie reorganization after TBI: A rodent functional connectomic analysis.Phosphacan and receptor protein tyrosine phosphatase β expression mediates deafferentation-induced synaptogenesis.HIF-1α involves in neuronal apoptosis after traumatic brain injury in adult rats.Use-dependent dendritic regrowth is limited after unilateral controlled cortical impact to the forelimb sensorimotor cortex.Trajectory of Parvalbumin Cell Impairment and Loss of Cortical Inhibition in Traumatic Brain Injury.Extracellular matrix and traumatic brain injury.Phosphorylated retinoblastoma protein (p-Rb) is involved in neuronal apoptosis after traumatic brain injury in adult rats.
P2860
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P2860
Traumatic brain injury results in disparate regions of chondroitin sulfate proteoglycan expression that are temporally limited
description
2009 nî lūn-bûn
@nan
2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Traumatic brain injury results ...... on that are temporally limited
@ast
Traumatic brain injury results ...... on that are temporally limited
@en
type
label
Traumatic brain injury results ...... on that are temporally limited
@ast
Traumatic brain injury results ...... on that are temporally limited
@en
prefLabel
Traumatic brain injury results ...... on that are temporally limited
@ast
Traumatic brain injury results ...... on that are temporally limited
@en
P2093
P2860
P356
P1476
Traumatic brain injury results ...... on that are temporally limited
@en
P2093
N G Harris
R L Sutton
S T Carmichael
P2860
P304
P356
10.1002/JNR.22115
P577
2009-10-01T00:00:00Z