Roles for cathepsins S, L, and B in insulitis and diabetes in the NOD mouse. - Wikidata - awgldk - TriplyDB

Roles for cathepsins S, L, and B in insulitis and diabetes in the NOD mouse.

Roles for cathepsins S, L, and B in insulitis and diabetes in the NOD mouse.

http://www.wikidata.org/entity/Q33653550

about

Intracellular nucleic acid sensors and autoimmunity Regulation of cathepsin G reduces the activation of proinsulin-reactive T cells from type 1 diabetes patients Cathepsin L inhibition prevents murine autoimmune diabetes via suppression of CD8(+) T cell activity.CTSH regulates β-cell function and disease progression in newly diagnosed type 1 diabetes patients.The peri-islet basement membrane, a barrier to infiltrating leukocytes in type 1 diabetes in mouse and human.Cathepsin B regulates the appearance and severity of mercury-induced inflammation and autoimmunity.Enhanced anti-serpin antibody activity inhibits autoimmune inflammation in type 1 diabetes Anti-serpin antibody-mediated regulation of proteases in autoimmune diabetes Strategies to reverse endothelial progenitor cell dysfunction in diabetes Lysosomal metal, redox and proton cycles influencing the CysHis cathepsin reaction.The Repertoires of Peptides Presented by MHC-II in the Thymus and in Peripheral Tissue: A Clue for Autoimmunity?The Roles of Lysosomes in Inflammation and Autoimmune Diseases.Treating the Underlying Pathophysiology of Primary Sjögren Syndrome: Recent Advances and Future Prospects.Correlation between serum cathepsin S and insulin resistance in type 2 diabetes.Downregulation of cathepsin G reduces the activation of CD4+ T cells in murine autoimmune diabetes.CA-074Me compound inhibits osteoclastogenesis via suppression of the NFATc1 and c-FOS signaling pathways.

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P2860

Roles for cathepsins S, L, and B in insulitis and diabetes in the NOD mouse.

http://www.wikidata.org/entity/Q33653550

description

2009 nî lūn-bûn

@nan

2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Roles for cathepsins S, L, and B in insulitis and diabetes in the NOD mouse.

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Roles for cathepsins S, L, and B in insulitis and diabetes in the NOD mouse.

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type

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Roles for cathepsins S, L, and B in insulitis and diabetes in the NOD mouse.

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Roles for cathepsins S, L, and B in insulitis and diabetes in the NOD mouse.

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Roles for cathepsins S, L, and B in insulitis and diabetes in the NOD mouse.

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Roles for cathepsins S, L, and B in insulitis and diabetes in the NOD mouse.

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J.JAUT.2009.07.003

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Journal of Autoimmunity

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Roles for cathepsins S, L, and B in insulitis and diabetes in the NOD mouse.

@en

P2093

Alexander Y Rudensky

http://www.w3.org/2001/XMLSchema#string

Barbara Houston

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Elizabeth A Kirk

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Lianne C Hsing

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Mark Caldwell

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Renee C LeBoeuf

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Shuo-Hung Hsiao

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Timothy S McMillen

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P2860

B7/CD28 costimulation is essential for the homeostasis of the CD4+CD25+ immunoregulatory T cells that control autoimmune diabetes

Where CD4+CD25+ T reg cells impinge on autoimmune diabetes

Cathepsin L deficiency as molecular defect of furless: hyperproliferation of keratinocytes and pertubation of hair follicle cycling

Cathepsin S required for normal MHC class II peptide loading and germinal center development

Identification of an MHC class I-restricted autoantigen in type 1 diabetes by screening an organ-specific cDNA library.

Tissue inhibitor of metalloproteinase-2 inhibits T-cell infiltration and preserves pancreatic beta-cell function in an in vitro type 1 diabetes mellitus model.

Cathepsin L is essential for onset of autoimmune diabetes in NOD mice.

Genetic and pathogenic basis of autoimmune diabetes in NOD mice.

Cathepsin B contributes to TNF-alpha-mediated hepatocyte apoptosis by promoting mitochondrial release of cytochrome c

Deficiency of cathepsin S reduces atherosclerosis in LDL receptor-deficient mice

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96-104

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scholarly article

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10.1016/J.JAUT.2009.07.003

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2

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34

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2009-08-06T00:00:00Z

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26726788

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19664906

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2822044

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