Helminth cysteine proteases inhibit TRIF-dependent activation of macrophages via degradation of TLR3.
about
Modulation of specific and allergy-related immune responses by helminthsChronic filarial infection provides protection against bacterial sepsis by functionally reprogramming macrophagesPeroxiredoxin-1 from the Human Hookworm Ancylostoma ceylanicum Forms a Stable Oxidized Decamer and Is Covalently Inhibited by Conoidin AA family of helminth molecules that modulate innate cell responses via molecular mimicry of host antimicrobial peptidesCathelicidin-like helminth defence molecules (HDMs): absence of cytotoxic, anti-microbial and anti-protozoan activities imply a specific adaptation to immune modulationInduction of protective immune responses against schistosomiasis using functionally active cysteine peptidasesCollagenolytic activities of the major secreted cathepsin L peptidases involved in the virulence of the helminth pathogen, Fasciola hepatica.The absence of MyD88 has no effect on the induction of alternatively activated macrophage during Fasciola hepatica infectionFasciola hepatica is associated with the failure to detect bovine tuberculosis in dairy cattle.Cofactors that may influence vaccine responses.Immune modulation by helminth parasites of ruminants: implications for vaccine development and host immune competence.Secreted proteins from the helminth Fasciola hepatica inhibit the initiation of autoreactive T cell responses and prevent diabetes in the NOD mouseHuman schistosomiasis is associated with endotoxemia and Toll-like receptor 2- and 4-bearing B cells.Fasciola hepatica Kunitz type molecule decreases dendritic cell activation and their ability to induce inflammatory responses.Transcriptional analysis identifies key genes involved in metabolism, fibrosis/tissue repair and the immune response against Fasciola hepatica in sheep liver.Fasciola hepatica fatty acid binding protein inhibits TLR4 activation and suppresses the inflammatory cytokines induced by lipopolysaccharide in vitro and in vivo.Toll-like receptor activation by helminths or helminth products to alleviate inflammatory bowel diseaseSecretory Products of Trichinella spiralis Muscle Larvae and Immunomodulation: Implication for Autoimmune Diseases, Allergies, and Malignancies.IL-10 Production in Macrophages Is Regulated by a TLR-Driven CREB-Mediated Mechanism That Is Linked to Genes Involved in Cell Metabolism.Interaction Between the Neglected Tropical Disease Human Schistosomiasis and HCV Infection in Egypt: a Puzzling Relationship.Does helminth activation of toll-like receptors modulate immune response in multiple sclerosis patients?House dust allergy and immunotherapy.A parasite-derived 68-mer peptide ameliorates autoimmune disease in murine models of Type 1 diabetes and multiple sclerosis.Th2 responses in schistosomiasis.Innate affairs of allergens.Induction of regulatory cells by helminth parasites: exploitation for the treatment of inflammatory diseases.Toll-like receptor signaling in parasitic infections.The Extracellular Vesicles of the Helminth Pathogen, Fasciola hepatica: Biogenesis Pathways and Cargo Molecules Involved in Parasite Pathogenesis.Interaction Between Helminths and Toll-Like Receptors: Possibilities and Potentials for Asthma Therapy.Anisakis - immunology of a foodborne parasitosis.Secreted products of Fasciola hepatica inhibit the induction of T cell responses that mediate allergy.Network Analysis of the Systemic Response to Fasciola hepatica Infection in Sheep Reveals Changes in Fibrosis, Apoptosis, Toll-Like Receptors 3/4, and B Cell Function.Host parasite communications-Messages from helminths for the immune system: Parasite communication and cell-cell interactions.Helminth defence molecules-immunomodulators designed by parasites!FH8--a small EF-hand protein from Fasciola hepatica.Characterization of the secreted cathepsin B cysteine proteases family of the carcinogenic liver fluke Clonorchis sinensis.Rhbdd3 controls autoimmunity by suppressing the production of IL-6 by dendritic cells via K27-linked ubiquitination of the regulator NEMO.Novel Therapeutics for Multiple Sclerosis Designed by Parasitic Worms.RNA interference in Fasciola gigantica: Establishing and optimization of experimental RNAi in the newly excysted juveniles of the fluke.Infection by the helminth parasite Fasciola hepatica requires rapid regulation of metabolic, virulence, and invasive factors to adjust to its mammalian host.
P2860
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P2860
Helminth cysteine proteases inhibit TRIF-dependent activation of macrophages via degradation of TLR3.
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Helminth cysteine proteases in ...... hages via degradation of TLR3.
@ast
Helminth cysteine proteases in ...... hages via degradation of TLR3.
@en
type
label
Helminth cysteine proteases in ...... hages via degradation of TLR3.
@ast
Helminth cysteine proteases in ...... hages via degradation of TLR3.
@en
prefLabel
Helminth cysteine proteases in ...... hages via degradation of TLR3.
@ast
Helminth cysteine proteases in ...... hages via degradation of TLR3.
@en
P2093
P2860
P50
P356
P1476
Helminth cysteine proteases in ...... hages via degradation of TLR3.
@en
P2093
Colin M Stack
John P Dalton
Sandra M O'Neill
P2860
P304
P356
10.1074/JBC.M109.060368
P407
P577
2009-11-18T00:00:00Z