Endogenous nerve growth factor regulates collagen expression and bladder hypertrophy through Akt and MAPK pathways during cystitis.
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Suppression of the PI3K pathway in vivo reduces cystitis-induced bladder hypertrophy and restores bladder capacity examined by magnetic resonance imagingInflammasomes are important mediators of cyclophosphamide-induced bladder inflammation.Role of c-Jun N-terminal kinase (JNK) activation in micturition reflexes in cyclophosphamide (CYP)-induced cystitis in female rats.Neurotrophin signaling and visceral hypersensitivityThe urinary bladder of spontaneously hypertensive rat demonstrates bladder hypertrophy, inflammation, and fibrosis but not hyperplasiaIn vivo regulation of brain-derived neurotrophic factor in dorsal root ganglia is mediated by nerve growth factor-triggered Akt activation during cystitisRole for pAKT in rat urinary bladder with cyclophosphamide (CYP)-induced cystitis.Diallyl Disulfide Prevents Cyclophosphamide-Induced Hemorrhagic Cystitis in Rats through the Inhibition of Oxidative Damage, MAPKs, and NF-κB Pathways.Regulation of IGF-1 but not TGF-β1 by NGF in the smooth muscle of the inflamed urinary bladder.Activation of extracellular signal-regulated protein kinase 5 is essential for cystitis- and nerve growth factor-induced calcitonin gene-related peptide expression in sensory neuronsConnective tissue and its growth factor CTGF distinguish the morphometric and molecular remodeling of the bladder in a model of neurogenic bladder.Inflammation and activity augment brain-derived neurotrophic factor peripheral release.Activation of cannabinoid receptor 2 inhibits experimental cystitisInhibition of NMDAR reduces bladder hypertrophy and improves bladder function in cyclophosphamide induced cystitisThe mouse cyclophosphamide model of bladder pain syndrome: tissue characterization, immune profiling, and relationship to metabotropic glutamate receptors.Nerve growth factor in bladder dysfunction: contributing factor, biomarker, and therapeutic target.Neurotrophins in bladder function: what do we know and where do we go from here?Neuroprotection and CD131/GDNF/AKT Pathway of Carbamylated Erythropoietin in Hypoxic Neurons.The role of ERK and JNK signaling in connective tissue growth factor induced extracellular matrix protein production and scar formation.The passive and active contractile properties of the neurogenic, underactive bladder.Protective effects of lipoic acid and mesna on cyclophosphamide-induced haemorrhagic cystitis in mice.Urinary bladder organ hypertrophy is partially regulated by Akt1-mediated protein synthesis pathway.
P2860
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P2860
Endogenous nerve growth factor regulates collagen expression and bladder hypertrophy through Akt and MAPK pathways during cystitis.
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Endogenous nerve growth factor ...... MAPK pathways during cystitis.
@ast
Endogenous nerve growth factor ...... MAPK pathways during cystitis.
@en
type
label
Endogenous nerve growth factor ...... MAPK pathways during cystitis.
@ast
Endogenous nerve growth factor ...... MAPK pathways during cystitis.
@en
prefLabel
Endogenous nerve growth factor ...... MAPK pathways during cystitis.
@ast
Endogenous nerve growth factor ...... MAPK pathways during cystitis.
@en
P2093
P2860
P356
P1476
Endogenous nerve growth factor ...... MAPK pathways during cystitis.
@en
P2093
Chul-Won Chung
Li-Ya Qiao
Qing L Zhang
P2860
P304
P356
10.1074/JBC.M109.040444
P407
P577
2009-12-07T00:00:00Z