Nitric oxide-dependent mitochondrial biogenesis generates Ca2+ signaling profile of lupus T cells.
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Stomatin-like protein 2 binds cardiolipin and regulates mitochondrial biogenesis and functionComputational properties of mitochondria in T cell activation and fateOxidative Stress and Treg and Th17 Dysfunction in Systemic Lupus ErythematosusThe role of Fatty Acid oxidation in the metabolic reprograming of activated t-cellsThe pathology of T cells in systemic lupus erythematosusSystems biology of lupus: mapping the impact of genomic and environmental factors on gene expression signatures, cellular signaling, metabolic pathways, hormonal and cytokine imbalance, and selecting targets for treatment.Differential calcium signaling and Kv1.3 trafficking to the immunological synapse in systemic lupus erythematosusRapamycin reduces disease activity and normalizes T cell activation-induced calcium fluxing in patients with systemic lupus erythematosus.Pathogenic mechanisms in systemic lupus erythematosus.Oxidative stress in the pathology and treatment of systemic lupus erythematosusHRES-1/Rab4-mediated depletion of Drp1 impairs mitochondrial homeostasis and represents a target for treatment in SLE.T-cell and B-cell signaling biomarkers and treatment targets in lupus.Increased mitochondrial electron transport chain activity at complex I is regulated by N-acetylcysteine in lymphocytes of patients with systemic lupus erythematosusThe biology of reactive intermediates in systemic lupus erythematosus.Estradiol differentially regulates calreticulin: a potential link with abnormal T cell function in systemic lupus erythematosus?Central role of nitric oxide in the pathogenesis of rheumatoid arthritis and systemic lupus erythematosus.mTOR signaling: a central pathway to pathogenesis in systemic lupus erythematosus?Mitochondrial DNA damage is associated with damage accrual and disease duration in patients with systemic lupus erythematosusEndothelial nitric oxide synthase reduces crescentic and necrotic glomerular lesions, reactive oxygen production, and MCP1 production in murine lupus nephritisActivation of mammalian target of rapamycin controls the loss of TCRzeta in lupus T cells through HRES-1/Rab4-regulated lysosomal degradationCalcium signaling in systemic lupus erythematosus T cells: a treatment targetUnderexpression of mitochondrial-DNA encoded ATP synthesis-related genes and DNA repair genes in systemic lupus erythematosus.The role of nitric oxide in abnormal T cell signal transduction in systemic lupus erythematosusIncreased mitochondrial mass characterizes the survival defect of HIV-specific CD8(+) T cells.Systemic lupus erythematosus serum deposits C4d on red blood cells, decreases red blood cell membrane deformability, and promotes nitric oxide production.Nitric oxide, mitochondrial hyperpolarization, and T cell activation.N-acetylcysteine reduces disease activity by blocking mammalian target of rapamycin in T cells from systemic lupus erythematosus patients: a randomized, double-blind, placebo-controlled trialDistinct metabolic programs in activated T cells: opportunities for selective immunomodulation.Metabolic regulation of organelle homeostasis in lupus T cellsNew insights into the pathogenesis of systemic lupus erythematosus.Mitochondria as a target for early detection and diagnosis of cancer.Generation of NO by bystander human CD8 T cells augments allogeneic responses by inhibiting cytokine deprivation-induced cell deathKinetic analysis of DAF-FM activation by NO: toward calibration of a NO-sensitive fluorescent dye.Association of common mitochondrial DNA variants with multiple sclerosis and systemic lupus erythematosus.Program-like aging and mitochondria: instead of random damage by free radicals.Metabolic control of T cell activation and death in SLE.Association of serum nitrate and nitrite levels with longitudinal assessments of disease activity and damage in systemic lupus erythematosus and lupus nephritis.Association of reactive oxygen and nitrogen intermediate and complement levels with apoptosis of peripheral blood mononuclear cells in lupus patientsFus1/Tusc2 is a novel regulator of mitochondrial calcium handling, Ca2+-coupled mitochondrial processes, and Ca2+-dependent NFAT and NF-κB pathways in CD4+ T cells.Immunobiology of Nitric Oxide and Regulation of Inducible Nitric Oxide Synthase.
P2860
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P2860
Nitric oxide-dependent mitochondrial biogenesis generates Ca2+ signaling profile of lupus T cells.
description
2004 nî lūn-bûn
@nan
2004 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Nitric oxide-dependent mitocho ...... ling profile of lupus T cells.
@ast
Nitric oxide-dependent mitocho ...... ling profile of lupus T cells.
@en
type
label
Nitric oxide-dependent mitocho ...... ling profile of lupus T cells.
@ast
Nitric oxide-dependent mitocho ...... ling profile of lupus T cells.
@en
prefLabel
Nitric oxide-dependent mitocho ...... ling profile of lupus T cells.
@ast
Nitric oxide-dependent mitocho ...... ling profile of lupus T cells.
@en
P2093
P2860
P1476
Nitric oxide-dependent mitocho ...... ling profile of lupus T cells.
@en
P2093
Gyorgy Nagy
Maureen Barcza
Nick Gonchoroff
Paul E Phillips
P2860
P304
P356
10.4049/JIMMUNOL.173.6.3676
P407
P50
P577
2004-09-01T00:00:00Z