Ubiquitin carboxyl-terminal hydrolase L1 is required for maintaining the structure and function of the neuromuscular junction - Wikidata - awgldk - TriplyDB

Ubiquitin carboxyl-terminal hydrolase L1 is required for maintaining the structure and function of the neuromuscular junction

Ubiquitin carboxyl-terminal hydrolase L1 is required for maintaining the structure and function of the neuromuscular junction

http://www.wikidata.org/entity/Q33667738

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To build a synapse: signaling pathways in neuromuscular junction assembly Ubiquitin-Synaptobrevin Fusion Protein Causes Degeneration of Presynaptic Motor Terminals in Mice Ubiquitin C-terminal hydrolase L1 (UCH-L1): structure, distribution and roles in brain function and dysfunction It Is All about (U)biquitin: Role of Altered Ubiquitin-Proteasome System and UCHL1 in Alzheimer Disease The Ubiquitin-Proteasome System: Potential Therapeutic Targets for Alzheimer's Disease and Spinal Cord Injury The role of deubiquitinating enzymes in synaptic function and nervous system diseases Corticospinal Motor Neurons Are Susceptible to Increased ER Stress and Display Profound Degeneration in the Absence of UCHL1 Function Dysregulation of Ubiquitin-Proteasome System in Neurodegenerative Diseases Bassoon and Piccolo maintain synapse integrity by regulating protein ubiquitination and degradation Excess α-synuclein worsens disease in mice lacking ubiquitin carboxy-terminal hydrolase L1 Identification of a gene set to evaluate the potential effects of loud sounds from seismic surveys on the ears of fishes: a study with Salmo salar.In vivo transduction of neurons with TAT-UCH-L1 protects brain against controlled cortical impact injury.An optimal ubiquitin-proteasome pathway in the nervous system: the role of deubiquitinating enzymes.Recessive loss of function of the neuronal ubiquitin hydrolase UCHL1 leads to early-onset progressive neurodegeneration UCHL1 regulates ubiquitination and recycling of the neural cell adhesion molecule NCAM.Parkin-mediated K63-polyubiquitination targets ubiquitin C-terminal hydrolase L1 for degradation by the autophagy-lysosome system.Human papillomavirus (HPV) upregulates the cellular deubiquitinase UCHL1 to suppress the keratinocyte's innate immune response The deubiquitinating enzyme USP-46 negatively regulates the degradation of glutamate receptors to control their abundance in the ventral nerve cord of Caenorhabditis elegans.Genetic background alters the severity and onset of neuromuscular disease caused by the loss of ubiquitin-specific protease 14 (usp14)Significance of ubiquitin carboxy-terminal hydrolase L1 elevations in athletes after sub-concussive head hits Ubiquitin carboxy-terminal hydrolase-l1 as a serum neurotrauma biomarker for exposure to occupational low-level blast Ubiquitination in postsynaptic function and plasticity Balancing act: deubiquitinating enzymes in the nervous system.The point mutation UCH-L1 C152A protects primary neurons against cyclopentenone prostaglandin-induced cytotoxicity: implications for post-ischemic neuronal injury.Current status of fluid biomarkers in mild traumatic brain injury Spinal Muscular Atrophy Patient iPSC-Derived Motor Neurons Have Reduced Expression of Proteins Important in Neuronal Development.Emerging evidence of coding mutations in the ubiquitin-proteasome system associated with cerebellar ataxias Structural and Functional Abnormalities of the Neuromuscular Junction in the Trembler-J Homozygote Mouse Model of Congenital Hypomyelinating Neuropathy Absence of UCHL 1 function leads to selective motor neuropathy.APP interacts with LRP4 and agrin to coordinate the development of the neuromuscular junction in mice.A Uchl1-Histone2BmCherry:GFP-gpi BAC transgene for imaging neuronal progenitors.Therapeutic strategies in Parkinson's disease: what we have learned from animal models.Ubiquitin C-Terminal Hydrolase L1 regulates myoblast proliferation and differentiation.Advances in understanding the role of disease-associated proteins in spinal muscular atrophy.Life and death in the trash heap: The ubiquitin proteasome pathway and UCHL1 in brain aging, neurodegenerative disease and cerebral Ischemia.Ubiquitin C-terminal hydrolase L1 interacts with choline transporter in cholinergic cells.Coordination of synaptic vesicle trafficking and turnover by the Rab35 signaling network.The ubiquitin C-terminal hydrolase UCH-L1 promotes bacterial invasion by altering the dynamics of the actin cytoskeleton.NH2-truncated human tau induces deregulated mitophagy in neurons by aberrant recruitment of Parkin and UCHL-1: implications in Alzheimer's disease.Electrophysiological characterization of neuromuscular synaptic dysfunction in mice.

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P2860

Ubiquitin carboxyl-terminal hydrolase L1 is required for maintaining the structure and function of the neuromuscular junction

http://www.wikidata.org/entity/Q33667738

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2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Ubiquitin carboxyl-terminal hy ...... of the neuromuscular junction

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Ubiquitin carboxyl-terminal hy ...... of the neuromuscular junction

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Ubiquitin carboxyl-terminal hy ...... of the neuromuscular junction

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Ubiquitin carboxyl-terminal hy ...... of the neuromuscular junction

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PNAS.0911516107

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Proceedings of the National Academy of Sciences of the United States of America

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Ubiquitin carboxyl-terminal hy ...... of the neuromuscular junction

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P2093

Fujun Chen

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Kalisa Galina Myers

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Weichun Lin

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Yoshie Sugiura

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Yun Liu

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P2860

The role of calcium in neuromuscular facilitation

Short-term synaptic plasticity

Synaptotagmin-2 is essential for survival and contributes to Ca2+ triggering of neurotransmitter release in central and neuromuscular synapses

Proteasome-independent functions of ubiquitin in endocytosis and signaling

Transmitter release modulation by intracellular Ca2+ buffers in facilitating and depressing nerve terminals of pyramidal cells in layer 2/3 of the rat neocortex indicates a target cell-specific difference in presynaptic calcium dynamics

Progressive degeneration of motor nerve terminals in GAD mutant mouse with hereditary sensory axonopathy

Gracile axonal dystrophy (GAD), a new neurological mutant in the mouse

Ubiquitin carboxy-terminal hydrolase L1 binds to and stabilizes monoubiquitin in neuron

Neuropathology of gracile axonal dystrophy (GAD) mouse. An animal model of central distal axonopathy in primary sensory neurons

The neuron-specific protein PGP 9.5 is a ubiquitin carboxyl-terminal hydrolase

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107

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20080621

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2824399

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