MYCN-regulated microRNAs repress estrogen receptor-alpha (ESR1) expression and neuronal differentiation in human neuroblastoma - Wikidata - awgldk - TriplyDB

MYCN-regulated microRNAs repress estrogen receptor-alpha (ESR1) expression and neuronal differentiation in human neuroblastoma

MYCN-regulated microRNAs repress estrogen receptor-alpha (ESR1) expression and neuronal differentiation in human neuroblastoma

http://www.wikidata.org/entity/Q33667823

about

Germline deletion of the miR-17∼92 cluster causes skeletal and growth defects in humans Chemoresistance, cancer stem cells, and miRNA influences: the case for neuroblastoma miRNA biogenesis: biological impact in the development of cancer Significance and therapeutic value of miRNAs in embryonal neural tumors The miR-17/92 cluster: a comprehensive update on its genomics, genetics, functions and increasingly important and numerous roles in health and disease Focal DNA copy number changes in neuroblastoma target MYCN regulated genes MicroRNAs in Neurotoxicity MYC on the path to cancer Deep sequencing reveals differential expression of microRNAs in favorable versus unfavorable neuroblastoma.Comprehensive DNA methylation study identifies novel progression-related and prognostic markers for cutaneous melanoma A sketch of known and novel MYCN-associated miRNA networks in neuroblastoma.Differential expression of microRNAs in dorsal root ganglia after sciatic nerve injury.Tumour-suppressor microRNAs let-7 and mir-101 target the proto-oncogene MYCN and inhibit cell proliferation in MYCN-amplified neuroblastoma.The many faces of estrogen signaling.MicroRNAs 10a and 10b are potent inducers of neuroblastoma cell differentiation through targeting of nuclear receptor corepressor 2.A neurogenetics approach to defining differential susceptibility to institutional care MicroRNA expression is differentially altered by xenobiotic drugs in different human cell lines.Targeting of the MYCN protein with small molecule c-MYC inhibitors MicroRNA and DNA methylation alterations mediating retinoic acid induced neuroblastoma cell differentiation.Estrogen receptor α enhances the transcriptional activity of ETS-1 and promotes the proliferation, migration and invasion of neuroblastoma cell in a ligand dependent manner.MYCN-targeting miRNAs are predominantly downregulated during MYCN‑driven neuroblastoma tumor formation.Uterine microRNA signature and consequence of their dysregulation in uterine disorders.Functional interactions among members of the miR-17-92 cluster in lymphocyte development, differentiation and malignant transformation.Functional MYCN signature predicts outcome of neuroblastoma irrespective of MYCN amplification.A subset of Drosophila Myc sites remain associated with mitotic chromosomes colocalized with insulator proteins.The c-Myc-regulated microRNA-17~92 (miR-17~92) and miR-106a~363 clusters target hCYP19A1 and hGCM1 to inhibit human trophoblast differentiation.MicroRNA-17-92 cluster mediates the proliferation and survival of neural progenitor cells after stroke.Target delivery of MYCN siRNA by folate-nanoliposomes delivery system in a metastatic neuroblastoma model E2F1-miR-20a-5p/20b-5p auto-regulatory feedback loop involved in myoblast proliferation and differentiation Transcription factor activity of estrogen receptor α activation upon nonylphenol or bisphenol A treatment enhances the in vitro proliferation, invasion, and migration of neuroblastoma cells.Neuroblastoma and MYCN Primate-specific miR-515 family members inhibit key genes in human trophoblast differentiation and are upregulated in preeclampsia A microRNA negative feedback loop downregulates vesicle transport and inhibits fear memory.A mouse model of MYCN-driven retinoblastoma reveals MYCN-independent tumor reemergence.Therapeutic targeting of miRNAs in neuroblastoma.Myc: Maestro of MicroRNAs The role of genetic and epigenetic alterations in neuroblastoma disease pathogenesis.Polycomb group proteins and MYC: the cancer connection.MicroRNAs in neuroblastoma: small-sized players with a large impact.Targeting RNA polymerase I to treat MYC-driven cancer.

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MYCN-regulated microRNAs repress estrogen receptor-alpha (ESR1) expression and neuronal differentiation in human neuroblastoma

http://www.wikidata.org/entity/Q33667823

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2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

MYCN-regulated microRNAs repre ...... tiation in human neuroblastoma

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MYCN-regulated microRNAs repre ...... tiation in human neuroblastoma

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type

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MYCN-regulated microRNAs repre ...... tiation in human neuroblastoma

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MYCN-regulated microRNAs repre ...... tiation in human neuroblastoma

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MYCN-regulated microRNAs repre ...... tiation in human neuroblastoma

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MYCN-regulated microRNAs repre ...... tiation in human neuroblastoma

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Proceedings of the National Academy of Sciences of the United States of America

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MYCN-regulated microRNAs repre ...... tiation in human neuroblastoma

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Erik Fredlund

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Inga Müller

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Jakob Lovén

http://www.w3.org/2001/XMLSchema#string

Marie Henriksson

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Nikolay Zinin

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Sven Påhlman

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Therese Wahlström

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Ulf Ribacke

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P2860

Widespread microRNA repression by Myc contributes to tumorigenesis

The impact of microRNAs on protein output

Most mammalian mRNAs are conserved targets of microRNAs

Posttranscriptional regulation of the heterochronic gene lin-14 by lin-4 mediates temporal pattern formation in C. elegans

A microRNA polycistron as a potential human oncogene

The C. elegans heterochronic gene lin-4 encodes small RNAs with antisense complementarity to lin-14

c-Myc-regulated microRNAs modulate E2F1 expression

MicroRNAs: genomics, biogenesis, mechanism, and function

MicroRNA-34a functions as a potential tumor suppressor by inducing apoptosis in neuroblastoma cells

Estrogen carcinogenesis in breast cancer

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