Long-term potentiation differentially affects two components of synaptic responses in hippocampus - Wikidata - awgldk - TriplyDB

Long-term potentiation differentially affects two components of synaptic responses in hippocampus

Long-term potentiation differentially affects two components of synaptic responses in hippocampus

http://www.wikidata.org/entity/Q33683038

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Slowly developing depression of N-methyl-D-aspartate receptor mediated responses in young rat hippocampi Opposing Effects of Neuronal Activity on Structural Plasticity The expression of long-term potentiation: reconciling the preists and the postivists Two sides to long-term potentiation: a view towards reconciliation Surface dynamics of GluN2B-NMDA receptors controls plasticity of maturing glutamate synapses Presynaptic BDNF promotes postsynaptic long-term potentiation in the dorsal striatum Protein kinase C activity is not responsible for the expression of long-term potentiation in hippocampus Silent synapses and the emergence of a postsynaptic mechanism for LTP.Dopamine transporter blockade increases LTP in the CA1 region of the rat hippocampus via activation of the D3 dopamine receptor Antidepressant mechanisms: functional and molecular correlates of excitatory amino acid neurotransmission.Glutamate receptor trafficking in synaptic plasticity.Silent synapses: what are they telling us about long-term potentiation?Long-term potentiation in the dentate gyrus of the anaesthetized rat is accompanied by an increase in extracellular glutamate: real-time measurements using a novel dialysis electrode Non-Hebbian properties of long-term potentiation enable high-capacity encoding of temporal sequences.Novel expression mechanism for synaptic potentiation: alignment of presynaptic release site and postsynaptic receptor.Protease-activated receptor-1 modulates hippocampal memory formation and synaptic plasticity Expression of NMDA receptor-dependent LTP in the hippocampus: bridging the divide.Long-term potentiation selectively expressed by NMDA receptors at hippocampal mossy fiber synapses Changes in presynaptic function during long-term potentiation.Ca2+/calmodulin-dependent protein kinase II phosphorylation of the presynaptic protein synapsin I is persistently increased during long-term potentiation.Increasing binding affinity of agonists to glutamate receptors increases synaptic responses at glutamatergic synapses A Primary Cortical Input to Hippocampus Expresses a Pathway-Specific and Endocannabinoid-Dependent Form of Long-Term Potentiation.State-dependent diffusion of actin-depolymerizing factor/cofilin underlies the enlargement and shrinkage of dendritic spines Presynaptic ultrastructural plasticity along CA3→CA1 axons during long-term potentiation in mature hippocampus.NMDA receptor-dependent long-term potentiation comprises a family of temporally overlapping forms of synaptic plasticity that are induced by different patterns of stimulation.Activity-dependent synaptic plasticity of NMDA receptors.Synaptic plasticity of NMDA receptors: mechanisms and functional implications.Links between long-term potentiation and neuropathology. An hypothesis involving calcium-activated proteases.Alterations in excitatory amino acid-mediated regulation of midbrain dopaminergic neurones induced by chronic psychostimulant administration and stress: relevance to behavioural sensitization and drug addiction.Review on the role of AMPA receptor nano-organization and dynamic in the properties of synaptic transmission.Plasticity of synaptic GluN receptors is required for the Src-dependent induction of long-term potentiation at CA3-CA1 synapses.Enhanced ventral hippocampal synaptic transmission and impaired synaptic plasticity in a rodent model of alcohol addiction vulnerability Long-term potentiation alters the modulator pharmacology of AMPA-type glutamate receptors.Postsynaptic conversion of silent synapses during LTP affects synaptic gain and transmission dynamics.Inositol hexakisphosphate-mediated regulation of glutamate receptors in rat brain sections.Contribution of NMDA receptor channels to the expression of LTP in the hippocampal dentate gyrus.Integrins modulate fast excitatory transmission at hippocampal synapses.Unconventional NMDA Receptor Signaling.Mechanisms underlying dedepression of synaptic NMDA receptors in the hippocampus.Focal photolysis of caged glutamate produces long-term depression of hippocampal glutamate receptors.

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P2860

Long-term potentiation differentially affects two components of synaptic responses in hippocampus

http://www.wikidata.org/entity/Q33683038

description

1988 nî lūn-bûn

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1988 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

1988 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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1988年の論文

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1988年学术文章

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1988年学术文章

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1988年学术文章

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P2860

Long-lasting potentiation of synaptic transmission in the dentate area of the anaesthetized rabbit following stimulation of the perforant path

The role of calcium in neuromuscular facilitation

Selective impairment of learning and blockade of long-term potentiation by an N-methyl-D-aspartate receptor antagonist, AP5

Magnesium gates glutamate-activated channels in mouse central neurones

Voltage-dependent block by Mg2+ of NMDA responses in spinal cord neurones.

A synaptic potential following single volleys in the hippocampal CA1 region possibly involved in the induction of long-lasting potentiation.

Role of N-methyl-D-aspartate receptors in the induction of synaptic potentiation by burst stimulation patterned after the hippocampal theta-rhythm.

Induction of synaptic potentiation in hippocampus by patterned stimulation involves two events.

Transient and enduring morphological correlates of synaptic activity and efficacy change in the rat hippocampal slice.

Excitatory amino acids in synaptic transmission in the Schaffer collateral-commissural pathway of the rat hippocampus.

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