Modulation of human aorta smooth muscle cell phenotype: a study of muscle-specific variants of vinculin, caldesmon, and actin expression.
about
Olfactomedin 2, a novel regulator for transforming growth factor-β-induced smooth muscle differentiation of human embryonic stem cell-derived mesenchymal cellsComplete sequence of human vinculin and assignment of the gene to chromosome 10Monocyte cells and cancer cells express novel paxillin isoforms with different binding properties to focal adhesion proteinsGenomic structure of the human caldesmon geneHuman smooth muscle VLA-1 integrin: purification, substrate specificity, localization in aorta, and expression during developmentThe molecular anatomy of caldesmonNuclear proteins bind a cis-acting element in the smooth muscle alpha-actin promoter.Expression of the smooth muscle myosin heavy chain gene is regulated by a negative-acting GC-rich element located between two positive-acting serum response factor-binding elements.Molecular control of vascular smooth muscle cell differentiation.h-Caldesmon as a specific marker of smooth muscle cell differentiation in some soft tissue tumors of the skin.Heterogeneous distribution of isoactins in cultured vascular smooth muscle cells does not reflect segregation of contractile and cytoskeletal domains.Targeted expression of SV40 large T-antigen to visceral smooth muscle induces proliferation of contractile smooth muscle cells and results in megacolonCaldesmon regulates the motility of vascular smooth muscle cells by modulating the actin cytoskeleton stability.Modulation of myocardin function by the ubiquitin E3 ligase UBR5.Caldesmon and the regulation of cytoskeletal functions.Caldesmon as a therapeutic target for proliferative vascular diseases.Hyperplastic Growth of Pulmonary Artery Smooth Muscle Cells from Subjects with Pulmonary Arterial Hypertension Is Activated through JNK and p38 MAPK.Identification of target antigens of anti-endothelial cell and anti-vascular smooth muscle cell antibodies in patients with giant cell arteritis: a proteomic approachGeneration of C-reactive protein and complement components in atherosclerotic plaques.Hypoxia selectively induces proliferation in a specific subpopulation of smooth muscle cells in the bovine neonatal pulmonary arterial mediaVarious cell types in human atherosclerotic lesions express ICAM-1. Further immunocytochemical and immunochemical studies employing monoclonal antibody 10F3.Cell division cycle 7 is a novel regulator of transforming growth factor-β-induced smooth muscle cell differentiation.Transforming growth factor-β and smooth muscle differentiationExpression of smooth muscle-specific proteins in myoepithelium and stromal myofibroblasts of normal and malignant human breast tissue.Intimal pericytes as the second line of immune defence in atherosclerosis.A 32-nucleotide exon-splicing enhancer regulates usage of competing 5' splice sites in a differential internal exon.Regulation and characteristics of vascular smooth muscle cell phenotypic diversity.Cell division cycle 7 mediates transforming growth factor-β-induced smooth muscle maturation through activation of myocardin gene transcription.The phenotypes of smooth muscle expressed in human atheroma.The complexity of cell composition of the intima of large arteries: focus on pericyte-like cells.Signature patterns of gene expression in mouse atherosclerosis and their correlation to human coronary disease.A serum response factor-dependent transcriptional regulatory program identifies distinct smooth muscle cell sublineages.Response gene to complement 32, a novel regulator for transforming growth factor-beta-induced smooth muscle differentiation of neural crest cells.Myosin light chain diphosphorylation is enhanced by growth promotion of cultured smooth muscle cells.Human foetal lung (IMR-90) cells: myofibroblasts with smooth muscle-like contractile properties.Transcriptional regulation of the chicken caldesmon gene. Activation of gizzard-type caldesmon promoter requires a CArG box-like motif.Expression of extra domain A fibronectin sequence in vascular smooth muscle cells is phenotype dependent.The transition of smooth muscle cells from a contractile to a migratory, phagocytic phenotype: direct demonstration of phenotypic modulation.Characterization of cloned aortic smooth muscle cells from young ratsSmooth muscle FGF/TGFβ cross talk regulates atherosclerosis progression
P2860
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P2860
Modulation of human aorta smooth muscle cell phenotype: a study of muscle-specific variants of vinculin, caldesmon, and actin expression.
description
1988 nî lūn-bûn
@nan
1988 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1988 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
name
Modulation of human aorta smoo ...... ldesmon, and actin expression.
@ast
Modulation of human aorta smoo ...... ldesmon, and actin expression.
@en
type
label
Modulation of human aorta smoo ...... ldesmon, and actin expression.
@ast
Modulation of human aorta smoo ...... ldesmon, and actin expression.
@en
prefLabel
Modulation of human aorta smoo ...... ldesmon, and actin expression.
@ast
Modulation of human aorta smoo ...... ldesmon, and actin expression.
@en
P2093
P2860
P356
P1476
Modulation of human aorta smoo ...... aldesmon, and actin expression
@en
P2093
A E Kabakov
A M Belkin
D N Mukhin
M A Glukhova
O I Ornatsky
V E Koteliansky
V N Smirnov
P2860
P304
P356
10.1073/PNAS.85.24.9542
P407
P577
1988-12-01T00:00:00Z