Stimulation of angiotensin AT2 receptors by the non-peptide agonist, Compound 21, evokes vasodepressor effects in conscious spontaneously hypertensive rats. - Wikidata - awgldk - TriplyDB

Stimulation of angiotensin AT2 receptors by the non-peptide agonist, Compound 21, evokes vasodepressor effects in conscious spontaneously hypertensive rats.

Stimulation of angiotensin AT2 receptors by the non-peptide agonist, Compound 21, evokes vasodepressor effects in conscious spontaneously hypertensive rats.

http://www.wikidata.org/entity/Q33685969

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AT2 receptor activities and pathophysiological implications Role of neurons and glia in the CNS actions of the renin-angiotensin system in cardiovascular control Metabolic actions of angiotensin II and insulin: a microvascular endothelial balancing act Direct angiotensin AT2 receptor stimulation using a novel AT2 receptor agonist, compound 21, evokes neuroprotection in conscious hypertensive rats AT2 Receptors: Potential Therapeutic Targets for Hypertension.AT2 Receptor Activation Prevents Sodium Retention and Reduces Blood Pressure in Angiotensin II-Dependent Hypertension.The intrarenal renin-angiotensin and dopaminergic systems: control of renal sodium excretion and blood pressure AT2 receptors: beneficial counter-regulatory role in cardiovascular and renal function Direct stimulation of angiotensin II type 2 receptor enhances spatial memory Nonclassical renin-angiotensin system and renal function.Activation of central angiotensin type 2 receptors by compound 21 improves arterial baroreflex sensitivity in rats with heart failure.The Impact of Overnutrition on Insulin Metabolic Signaling in the Heart and the Kidney Angiotensin (1-7) receptor antagonism equalizes angiotensin II-induced hypertension in male and female spontaneously hypertensive rats.Electronic sculpting of ligand-GPCR subtype selectivity: the case of angiotensin II.Angiotensin type 2 receptors: blood pressure regulation and end organ damage Cardiac insulin resistance and microRNA modulators.Hypotensive and sympathoinhibitory responses to selective central AT2 receptor stimulation in spontaneously hypertensive rats.Differential mechanisms of ang (1-7)-mediated vasodepressor effect in adult and aged candesartan-treated rats Reporter mouse strain provides a novel look at angiotensin type-2 receptor distribution in the central nervous system Activation of central angiotensin type 2 receptors suppresses norepinephrine excretion and blood pressure in conscious rats Adaptive mechanisms to compensate for overnutrition-induced cardiovascular abnormalities.Influence of Angiotensin II Subtype 2 Receptor (AT(2)R) Antagonist, PD123319, on Cardiovascular Remodelling of Aged Spontaneously Hypertensive Rats during Chronic Angiotensin II Subtype 1 Receptor (AT(1)R) Blockade Angiotensin II, a Neuropeptide at the Frontier between Endocrinology and Neuroscience: Is There a Link between the Angiotensin II Type 2 Receptor and Alzheimer's Disease?International Union of Basic and Clinical Pharmacology. XCIX. Angiotensin Receptors: Interpreters of Pathophysiological Angiotensinergic Stimuli [corrected].Small-molecule AT2 receptor agonists.The Angiotensin II Type 2 Receptor in Brain Functions: An Update.Gender Difference in Renal Blood Flow Response to Angiotensin II Administration after Ischemia/Reperfusion in Rats: The Role of AT2 Receptor.Neuroimmune communication in hypertension and obesity: a new therapeutic angle?Angiotensin AT2 receptor stimulation is anti-inflammatory in lipopolysaccharide-activated THP-1 macrophages via increased interleukin-10 production.Angiotensin II type-2 receptor-specific effects on the cardiovascular system.Impact of AT2-receptor stimulation on vascular biology, kidney function, and blood pressure.AT2 receptor non-peptide agonist C21 promotes natriuresis in obese Zucker rats.Activation of angiotensin type 2 receptors partially ameliorates streptozotocin-induced diabetes in male rats by islet protection.New insights into angiotensin receptor actions: from blood pressure to aging.AT2 receptor signaling and sympathetic regulation.Role of angiotensin II receptor subtype activation in cognitive function and ischaemic brain damage.Blood pressure and renal hemodynamic effects of angiotensin fragments.Angiotensin II and the vascular phenotype in hypertension.Angiotensin II type 2 receptor agonists: where should they be applied?The regenerative potential of angiotensin AT2 receptor in cardiac repair.

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Stimulation of angiotensin AT2 receptors by the non-peptide agonist, Compound 21, evokes vasodepressor effects in conscious spontaneously hypertensive rats.

http://www.wikidata.org/entity/Q33685969

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2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Stimulation of angiotensin AT2 ...... ontaneously hypertensive rats.

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Stimulation of angiotensin AT2 ...... ontaneously hypertensive rats.

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Stimulation of angiotensin AT2 ...... ontaneously hypertensive rats.

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J.1476-5381.2009.00575.X

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British Journal of Pharmacology

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Stimulation of angiotensin AT2 ...... pontaneously hypertensive rats

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A Hallberg

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E S Jones

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P2860

Behavioural and cardiovascular effects of disrupting the angiotensin II type-2 receptor in mice

Angiotensin type 2 receptor in resistance arteries of type 2 diabetic hypertensive patients

International union of pharmacology. XXIII. The angiotensin II receptors

Flow (shear stress)-induced endothelium-dependent dilation is altered in mice lacking the gene encoding for dystrophin

Angiotensin type 2 receptor-mediated hypotension in angiotensin type-1 receptor-blocked rats.

Update on the role of the AT2 receptor.

First reported nonpeptide AT1 receptor agonist (L-162,313) acts as an AT2 receptor agonist in vivo.

Design, synthesis, and biological evaluation of the first selective nonpeptide AT2 receptor agonist.

Angiotensin II relaxes microvessels via the AT(2) receptor and Ca(2+)-activated K(+) (BK(Ca)) channels.

Physiological and pathophysiological functions of the AT(2) subtype receptor of angiotensin II: from large arteries to the microcirculation.

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