Myeloid differentiation factor 88 (MyD88)-deficiency increases risk of diabetes in mice
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Animal Models of Nonalcoholic Steatohepatitis: Eat, Delete, and InflameHow gut microbes talk to organs: The role of endocrine and nervous routesCellular and molecular players in adipose tissue inflammation in the development of obesity-induced insulin resistanceMetagenome and metabolism: the tissue microbiota hypothesis.MyD88-dependent interplay between myeloid and endothelial cells in the initiation and progression of obesity-associated inflammatory diseases.Mechanisms of inflammatory responses in obese adipose tissue.Adipose tissue macrophages in the development of obesity-induced inflammation, insulin resistance and type 2 diabetes.Both bone marrow-derived and non-bone marrow-derived cells contribute to AIM2 and NLRP3 inflammasome activation in a MyD88-dependent manner in dietary steatohepatitis.Crosstalk between Gut Microbiota and Dietary Lipids Aggravates WAT Inflammation through TLR Signaling.Intestinal epithelial MyD88 is a sensor switching host metabolism towards obesity according to nutritional status.Inflammatory links between obesity and metabolic disease.NOD1 activators link innate immunity to insulin resistanceTalking microbes: When gut bacteria interact with diet and host organsSuppression of Adaptive Immune Cell Activation Does Not Alter Innate Immune Adipose Inflammation or Insulin Resistance in ObesityGastrointestinal and hepatic mechanisms limiting entry and dissemination of lipopolysaccharide into the systemic circulation.Hepatic recruitment of macrophages promotes nonalcoholic steatohepatitis through CCR2Macrophage-mediated inflammation in metabolic diseaseCommensal Microbe-specific Activation of B2 Cell Subsets Contributes to Atherosclerosis Development Independently of Lipid MetabolismRecent advances in obesity-induced inflammation and insulin resistance.Toll-like receptor 4 deficiency decreases atherosclerosis but does not protect against inflammation in obese low-density lipoprotein receptor-deficient mice.Myeloid cell-specific ABCA1 deletion does not worsen insulin resistance in HF diet-induced or genetically obese mouse modelsSingle-cell imaging of normal and malignant cell engraftment into optically clear prkdc-null SCID zebrafish.Innate immune activation in obesity.Flurbiprofen ameliorated obesity by attenuating leptin resistance induced by endoplasmic reticulum stress.The Addition of Liquid Fructose to a Western-Type Diet in LDL-R-/- Mice Induces Liver Inflammation and Fibrogenesis Markers without Disrupting Insulin Receptor Signalling after an Insulin ChallengeState of the union between metabolism and the immune system in type 2 diabetes.Toll-like receptor signaling: common pathways that drive cardiovascular disease and rheumatoid arthritis.Toll-like receptors, inflammation, metabolism and obesity.Regulation of metabolism: a cross talk between gut microbiota and its human host.DNA methylation of tumor necrosis factor-α, monocyte chemoattractant protein-1, and adiponectin genes in visceral adipose tissue is related to type 2 diabetes in the Xinjiang Uygur population.Innate immunity in diabetes and diabetic nephropathy.Resident T Cells Are Unable To Control Herpes Simplex Virus-1 Activity in the Brain Ependymal Region during Latency.Transgenic mice with ectopic expression of constitutively active TLR4 in adipose tissues do not show impaired insulin sensitivity.Naive Donor NK Cell Repertoires Associated with Less Leukemia Relapse after Allogeneic Hematopoietic Stem Cell Transplantation.Habitual physical activity protects against lipopolysaccharide-induced inflammation in mouse adipose tissue.Ly49C Impairs NK Cell Memory in Mouse Cytomegalovirus Infection.Hepatocyte MyD88 affects bile acids, gut microbiota and metabolome contributing to regulate glucose and lipid metabolism.Identification of pigment epithelium-derived factor as an adipocyte-derived inflammatory factor.Case-control study and mRNA expression analysis reveal the MyD88 gene is associated with digestive disorders in rabbit.Endocrine Disruptors Leading to Obesity and Related Diseases.
P2860
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P2860
Myeloid differentiation factor 88 (MyD88)-deficiency increases risk of diabetes in mice
description
2010 nî lūn-bûn
@nan
2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Myeloid differentiation factor 88 (MyD88)-deficiency increases risk of diabetes in mice
@ast
Myeloid differentiation factor 88 (MyD88)-deficiency increases risk of diabetes in mice
@en
type
label
Myeloid differentiation factor 88 (MyD88)-deficiency increases risk of diabetes in mice
@ast
Myeloid differentiation factor 88 (MyD88)-deficiency increases risk of diabetes in mice
@en
prefLabel
Myeloid differentiation factor 88 (MyD88)-deficiency increases risk of diabetes in mice
@ast
Myeloid differentiation factor 88 (MyD88)-deficiency increases risk of diabetes in mice
@en
P2093
P2860
P1433
P1476
Myeloid differentiation factor 88 (MyD88)-deficiency increases risk of diabetes in mice
@en
P2093
Koichiro Ozawa
Shizuo Akira
Shota Yokoyama
Suguru Matsuo
Toru Hosoi
P2860
P304
P356
10.1371/JOURNAL.PONE.0012537
P407
P577
2010-09-02T00:00:00Z