High Sensitivity of SIRT3 Deficient Hearts to Ischemia-Reperfusion Is Associated with Mitochondrial Abnormalities.
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Potential mechanisms linking SIRT activity and hypoxic 2-hydroxyglutarate generation: no role for direct enzyme (de)acetylation.Regulation of Sirtuin-Mediated Protein Deacetylation by Cardioprotective Phytochemicals.SIRT3: A New Regulator of Cardiovascular Diseases.Acetylation of Mitochondrial Proteins in the Heart: The Role of SIRT3
P2860
High Sensitivity of SIRT3 Deficient Hearts to Ischemia-Reperfusion Is Associated with Mitochondrial Abnormalities.
description
2017 nî lūn-bûn
@nan
2017 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2017 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2017年の論文
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2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
name
High Sensitivity of SIRT3 Defi ...... h Mitochondrial Abnormalities.
@ast
High Sensitivity of SIRT3 Defi ...... h Mitochondrial Abnormalities.
@en
type
label
High Sensitivity of SIRT3 Defi ...... h Mitochondrial Abnormalities.
@ast
High Sensitivity of SIRT3 Defi ...... h Mitochondrial Abnormalities.
@en
prefLabel
High Sensitivity of SIRT3 Defi ...... h Mitochondrial Abnormalities.
@ast
High Sensitivity of SIRT3 Defi ...... h Mitochondrial Abnormalities.
@en
P2093
P2860
P356
P1476
High Sensitivity of SIRT3 Defi ...... h Mitochondrial Abnormalities.
@en
P2093
Pedro J Rullán
Rebecca M Parodi-Rullán
Sabzali Javadov
Sehwan Jang
Xavier Chapa-Dubocq
P2860
P356
10.3389/FPHAR.2017.00275
P577
2017-05-16T00:00:00Z