Generation and repair of AID-initiated DNA lesions in B lymphocytes. - Wikidata - awgldk - TriplyDB

Generation and repair of AID-initiated DNA lesions in B lymphocytes.

Generation and repair of AID-initiated DNA lesions in B lymphocytes.

http://www.wikidata.org/entity/Q33690522

about

Antibody Affinity Maturation in Fishes-Our Current Understanding AID-initiated DNA lesions are differentially processed in distinct B cell populations.Unexpected effects of different genetic backgrounds on identification of genomic rearrangements via whole-genome next generation sequencing.Imbalanced PTEN and PI3K Signaling Impairs Class Switch Recombination.Combined deletion of Xrcc4 and Trp53 in mouse germinal center B cells leads to novel B cell lymphomas with clonal heterogeneity Interplay between Target Sequences and Repair Pathways Determines Distinct Outcomes of AID-Initiated Lesions Non-bulky Lesions in Human DNA: the Ways of Formation, Repair, and Replication.Chemotherapy-induced differential cell cycle arrest in B cell lymphomas affects their sensitivity to Wee1 inhibition.PTEN-Regulated AID Transcription in Germinal Center B Cells Is Essential for the Class-Switch Recombination and IgG Antibody Responses.Transcriptome analysis of immune genes in peripheral blood mononuclear cells of young foals and adult horses

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Generation and repair of AID-initiated DNA lesions in B lymphocytes.

http://www.wikidata.org/entity/Q33690522

description

2014 nî lūn-bûn

@nan

2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

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2014年論文

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2014年论文

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name

Generation and repair of AID-initiated DNA lesions in B lymphocytes.

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Generation and repair of AID-initiated DNA lesions in B lymphocytes.

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type

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Generation and repair of AID-initiated DNA lesions in B lymphocytes.

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Generation and repair of AID-initiated DNA lesions in B lymphocytes.

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Generation and repair of AID-initiated DNA lesions in B lymphocytes.

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Generation and repair of AID-initiated DNA lesions in B lymphocytes.

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S11684-014-0324-4

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Frontiers of medicine

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Generation and repair of AID-initiated DNA lesions in B lymphocytes.

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Zhangguo Chen

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P2860

DNA ligase III promotes alternative nonhomologous end-joining during chromosomal translocation formation

The mechanism of double-strand DNA break repair by the nonhomologous DNA end-joining pathway

Oncogenic transformation in the absence of Xrcc4 targets peripheral B cells that have undergone editing and switching

Reprogramming towards pluripotency requires AID-dependent DNA demethylation

Robust chromosomal DNA repair via alternative end-joining in the absence of X-ray repair cross-complementing protein 1 (XRCC1)

Human activation-induced cytidine deaminase causes transcription-dependent, strand-biased C to U deaminations

Activation-induced cytidine deaminase deaminates deoxycytidine on single-stranded DNA but requires the action of RNase

A cell cycle-dependent regulatory circuit composed of 53BP1-RIF1 and BRCA1-CtIP controls DNA repair pathway choice.

53BP1 mediates productive and mutagenic DNA repair through distinct phosphoprotein interactions.

DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139

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