Sestrin2 inhibits uncoupling protein 1 expression through suppressing reactive oxygen species. - Wikidata - awgldk - TriplyDB

Sestrin2 inhibits uncoupling protein 1 expression through suppressing reactive oxygen species.

Sestrin2 inhibits uncoupling protein 1 expression through suppressing reactive oxygen species.

http://www.wikidata.org/entity/Q33694643

about

A New Role for Browning as a Redox and Stress Adaptive Mechanism?Sestrin2, a Regulator of Thermogenesis and Mitohormesis in Brown Adipose Tissue Janus-faced Sestrin2 controls ROS and mTOR signalling through two separate functional domains Increased Energy Expenditure, Ucp1 Expression, and Resistance to Diet-induced Obesity in Mice Lacking Nuclear Factor-Erythroid-2-related Transcription Factor-2 (Nrf2)Sestrin 3 protein enhances hepatic insulin sensitivity by direct activation of the mTORC2-Akt signaling Generation of reactive oxygen species by lethal attacks from competing microbes.Upregulation of Sestrin2 expression protects against macrophage apoptosis induced by oxidized low-density lipoprotein.Oxidative stress, redox regulation and diseases of cellular differentiation.Type VI Secretion System Transports Zn2+ to Combat Multiple Stresses and Host Immunity.Mitochondrial Hormesis links nutrient restriction to improved metabolism in fat cell.Tumor suppressive role of sestrin2 during colitis and colon carcinogenesis.Biochemical Basis of Sestrin Physiological Activities.Atgl deficiency induces podocyte apoptosis and leads to glomerular filtration barrier damage.Adipocyte-Specific Deletion of Manganese Superoxide Dismutase Protects From Diet-Induced Obesity Through Increased Mitochondrial Uncoupling and Biogenesis Sestrin 2 suppresses cells proliferation through AMPK/mTORC1 pathway activation in colorectal cancer Metabolic activity of brown, "beige," and white adipose tissues in response to chronic adrenergic stimulation in male mice.mTORC1 is Required for Brown Adipose Tissue Recruitment and Metabolic Adaptation to Cold.Mitochondrial reactive oxygen species and adipose tissue thermogenesis: Bridging physiology and mechanisms.Stress turns on the heat: Regulation of mitochondrial biogenesis and UCP1 by ROS in adipocytes.Downregulation of B-myb promotes senescence via the ROS-mediated p53/p21 pathway, in vascular endothelial cells.Unraveling the truth about antioxidants: mitohormesis explains ROS-induced health benefits.Influence of anaerobic and aerobic exercise on age-related pathways in skeletal muscle.

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P2860

Sestrin2 inhibits uncoupling protein 1 expression through suppressing reactive oxygen species.

http://www.wikidata.org/entity/Q33694643

description

2014 nî lūn-bûn

@nan

2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Sestrin2 inhibits uncoupling p ...... ssing reactive oxygen species.

@ast

Sestrin2 inhibits uncoupling p ...... ssing reactive oxygen species.

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type

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Sestrin2 inhibits uncoupling p ...... ssing reactive oxygen species.

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Sestrin2 inhibits uncoupling p ...... ssing reactive oxygen species.

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prefLabel

Sestrin2 inhibits uncoupling p ...... ssing reactive oxygen species.

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Sestrin2 inhibits uncoupling p ...... ssing reactive oxygen species.

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PNAS.1401787111

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Sestrin2 inhibits uncoupling p ...... ssing reactive oxygen species.

@en

P2093

Elena Feinstein

http://www.w3.org/2001/XMLSchema#string

Golda Damari

http://www.w3.org/2001/XMLSchema#string

Haeli Park

http://www.w3.org/2001/XMLSchema#string

Haewon Park

http://www.w3.org/2001/XMLSchema#string

Hwan-Woo Park

http://www.w3.org/2001/XMLSchema#string

Ian A Semple

http://www.w3.org/2001/XMLSchema#string

Insook Jang

http://www.w3.org/2001/XMLSchema#string

Jeong Sig Kim

http://www.w3.org/2001/XMLSchema#string

Jun Hee Lee

http://www.w3.org/2001/XMLSchema#string

Maya Golikov

http://www.w3.org/2001/XMLSchema#string

P2860

Antioxidant supplements for prevention of mortality in healthy participants and patients with various diseases

Antioxidants prevent health-promoting effects of physical exercise in humans

Reactive oxygen species in health and disease

Regeneration of peroxiredoxins by p53-regulated sestrins, homologs of bacterial AhpD

Sestrin 2 is not a reductase for cysteine sulfinic acid of peroxiredoxins

Sestrins activate Nrf2 by promoting p62-dependent autophagic degradation of Keap1 and prevent oxidative liver damage

p53 target genes sestrin1 and sestrin2 connect genotoxic stress and mTOR signaling

Mice lacking mitochondrial uncoupling protein are cold-sensitive but not obese

Maintenance of metabolic homeostasis by Sestrin2 and Sestrin3

Thermoregulatory and metabolic phenotypes of mice lacking noradrenaline and adrenaline

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scholarly article

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10.1073/PNAS.1401787111

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21

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P478

111

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2014-05-13T00:00:00Z

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24825887

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4040599

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