CD36/fatty acid translocase, an inflammatory mediator, is involved in hyperlipidemia-induced exacerbation in ischemic brain injury. - Wikidata - awgldk - TriplyDB

CD36/fatty acid translocase, an inflammatory mediator, is involved in hyperlipidemia-induced exacerbation in ischemic brain injury.

CD36/fatty acid translocase, an inflammatory mediator, is involved in hyperlipidemia-induced exacerbation in ischemic brain injury.

http://www.wikidata.org/entity/Q33697097

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Obesity and stroke: Can we translate from rodents to patients?Isorhamnetin attenuates atherosclerosis by inhibiting macrophage apoptosis via PI3K/AKT activation and HO-1 induction Critical role of matrix metalloprotease-9 in chronic high fat diet-induced cerebral vascular remodelling and increase of ischaemic brain injury in mice†.Deregulation of inflammatory response in the diabetic condition is associated with increased ischemic brain injury.RODENT STROKE MODEL GUIDELINES FOR PRECLINICAL STROKE TRIALS (1ST EDITION).Role of spleen-derived monocytes/macrophages in acute ischemic brain injury.Scavenger receptor class-A has a central role in cerebral ischemia-reperfusion injury.Hyperlipidemia disrupts cerebrovascular reflexes and worsens ischemic perfusion defect.Detrimental impact of hyperlipidemia on the peripheral nervous system: A novel target of medical epidemiological and fundamental research study Targeting CD36-mediated inflammation reduces acute brain injury in transient, but not permanent, ischemic stroke.Genetic variant of BDNF (Val66Met) polymorphism attenuates stroke-induced angiogenic responses by enhancing anti-angiogenic mediator CD36 expression Salvianolic acid B inhibits macrophage uptake of modified low density lipoprotein (mLDL) in a scavenger receptor CD36-dependent manner.Role of Neutrophils in Exacerbation of Brain Injury After Focal Cerebral Ischemia in Hyperlipidemic Mice CD36 as a therapeutic target for endothelial dysfunction in stroke.Transglutaminase inhibition protects against oxidative stress-induced neuronal death downstream of pathological ERK activation.Daidzein Augments Cholesterol Homeostasis via ApoE to Promote Functional Recovery in Chronic Stroke CD36 genotype associated with ischemic stroke in Chinese Han.Genetic deletion of CD36 enhances injury after acute neonatal stroke Method parameters' impact on mortality and variability in mouse stroke experiments: a meta-analysis.CD36: a multi-modal target for acute stroke therapy.An adaptive role for BDNF Val66Met polymorphism in motor recovery in chronic stroke.Toll-like receptors: new players in myocardial ischemia/reperfusion injury.The influence of stroke risk factors and comorbidities on assessment of stroke therapies in humans and animals.Microglia and Monocyte-Derived Macrophages in Stroke Fatty acid transporting proteins: Roles in brain development, aging, and stroke.CD36 is involved in astrocyte activation and astroglial scar formation Administration of mesenchymal stromal cells before renal ischemia/reperfusion attenuates kidney injury and may modulate renal lipid metabolism in rats A role for spleen monocytes in post-ischemic brain inflammation and injury.CD36 in the periphery and brain synergizes in stroke injury in hyperlipidemia Insulin-associated neuroinflammatory pathways as therapeutic targets for traumatic brain injury Targeted liposomal drug delivery to monocytes and macrophages Sarcolipin and ubiquitin carboxy-terminal hydrolase 1 mRNAs are over-expressed in skeletal muscles of alpha-tocopherol deficient mice.Cell Surface CD36 Protein in Monocyte/Macrophage Contributes to Phagocytosis during the Resolution Phase of Ischemic Stroke in Mice.Commentary on Myers et al.: growing role of the innate immunity receptor CD36 in central nervous system diseases.Brain 11: what's new in stroke research?What Do Experimental Models Teach Us About Comorbidities in Stroke?Critical Role of Flavin and Glutathione in Complex I-Mediated Bioenergetic Failure in Brain Ischemia/Reperfusion Injury.

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P2860

CD36/fatty acid translocase, an inflammatory mediator, is involved in hyperlipidemia-induced exacerbation in ischemic brain injury.

http://www.wikidata.org/entity/Q33697097

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2008年の論文

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JNEUROSCI.0982-08.2008

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CD36/fatty acid translocase, a ...... tion in ischemic brain injury.

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Aaron T Tolhurst

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Eunhee Kim

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Lu Ye Qin

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Maria Febbraio

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Sunghee Cho

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Xin-Yuan Chen

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P2860

Targeted disruption of the class B scavenger receptor CD36 protects against atherosclerotic lesion development in mice

Oxidized LDL-induced NF-kappa B activation and subsequent expression of proinflammatory genes are defective in monocyte-derived macrophages from CD36-deficient patients

Identification of a novel family of oxidized phospholipids that serve as ligands for the macrophage scavenger receptor CD36

Cloning of a rat adipocyte membrane protein implicated in binding or transport of long-chain fatty acids that is induced during preadipocyte differentiation. Homology with human CD36

The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions

Inflammation, atherosclerosis, and stroke.

Inflammatory mediators and stroke: new opportunities for novel therapeutics.

Inflammation and stroke: putative role for cytokines, adhesion molecules and iNOS in brain response to ischemia.

Macrophage scavenger receptor class A: A multifunctional receptor in atherosclerosis.

Loss of receptor-mediated lipid uptake via scavenger receptor A or CD36 pathways does not ameliorate atherosclerosis in hyperlipidemic mice

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4661-4670

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10.1523/JNEUROSCI.0982-08.2008

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