Loss of c-Met disrupts gene expression program required for G2/M progression during liver regeneration in mice - Wikidata - awgldk - TriplyDB

Loss of c-Met disrupts gene expression program required for G2/M progression during liver regeneration in mice

Loss of c-Met disrupts gene expression program required for G2/M progression during liver regeneration in mice

http://www.wikidata.org/entity/Q33700482

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The NF-κB subunit RelA/p65 is dispensable for successful liver regeneration after partial hepatectomy in mice Eight paths of ERK1/2 signalling pathway regulating hepatocyte proliferation in rat liver regeneration Novel functions of platelets in the liver.Platelets in liver disease, cancer and regeneration Exosomes from mesenchymal stem cells induce the conversion of hepatocytes into progenitor oval cells.Mir-34a is upregulated during liver regeneration in rats and is associated with the suppression of hepatocyte proliferation.P2Y2 purinergic receptor activation is essential for efficient hepatocyte proliferation in response to partial hepatectomy.New concepts in liver regeneration.Stathmin regulates keratinocyte proliferation and migration during cutaneous regeneration.Polμ deficiency increases resistance to oxidative damage and delays liver aging.Loss of hepatocyte EGFR has no effect alone but exacerbates carbon tetrachloride-induced liver injury and impairs regeneration in hepatocyte Met-deficient mice.The CHR promoter element controls cell cycle-dependent gene transcription and binds the DREAM and MMB complexes.Tissue-Specific Gain of RTK Signalling Uncovers Selective Cell Vulnerability during Embryogenesis.Hepatocyte growth factor/c-met signaling is required for stem-cell-mediated liver regeneration in mice.Loss of c-Met accelerates development of liver fibrosis in response to CCl(4) exposure through deregulation of multiple molecular pathways.Inhibition of hepcidin transcription by growth factors.Aurora B is regulated by the mitogen-activated protein kinase/extracellular signal-regulated kinase (MAPK/ERK) signaling pathway and is a valuable potential target in melanoma cells.Glutathione transferases P1/P2 regulate the timing of signaling pathway activations and cell cycle progression during mouse liver regeneration The MAPK MEK1/2-ERK1/2 Pathway and Its Implication in Hepatocyte Cell Cycle Control.FOXM1 participates in PLK1-regulated cell cycle progression in renal cell cancer cells.The retinoic acid derivative, ABPN, inhibits pancreatic cancer through induction of Nrdp1 A disintegrin and metalloproteinase 17 regulates TNF and TNFR1 levels in inflammation and liver regeneration in mice Specific fate decisions in adult hepatic progenitor cells driven by MET and EGFR signaling.Systematic screen with kinases inhibitors reveals kinases play distinct roles in growth of osteoprogenitor cells Tyrosine phosphorylation and liver regeneration: A glance at intracellular transducers.Novel therapy for liver regeneration by increasing the number of platelets.To divide or not to divide: revisiting liver regeneration.A combination of tyrosine kinase inhibitors, crizotinib and dasatinib for the treatment of glioblastoma multiforme.Hepatocyte growth factor and Met in drug discovery.Dissecting the role of epidermal growth factor receptor catalytic activity during liver regeneration and hepatocarcinogenesis.Serotonin promotes acinar dedifferentiation following pancreatitis-induced regeneration in the adult pancreas.EGFR: A Master Piece in G1/S Phase Transition of Liver Regeneration.Hepatic regenerative medicine: exploiting the liver's will to live.Tumour growth stimulation following partial hepatectomy in mice is associated with increased upregulation of c-Met.MicroRNA-221 overexpression accelerates hepatocyte proliferation during liver regeneration.Biological roles of hepatocyte growth factor-Met signaling from genetically modified animals.HGF-Met Pathway in Regeneration and Drug Discovery.[EGFR, liver regeneration and carcinogenesis: not the only culprit].

P2860

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P2860

Loss of c-Met disrupts gene expression program required for G2/M progression during liver regeneration in mice

http://www.wikidata.org/entity/Q33700482

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

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2010年論文

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2010年论文

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name

Loss of c-Met disrupts gene ex ...... ing liver regeneration in mice

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Loss of c-Met disrupts gene ex ...... ing liver regeneration in mice

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type

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Loss of c-Met disrupts gene ex ...... ing liver regeneration in mice

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Loss of c-Met disrupts gene ex ...... ing liver regeneration in mice

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Loss of c-Met disrupts gene ex ...... ing liver regeneration in mice

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Loss of c-Met disrupts gene ex ...... ing liver regeneration in mice

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Loss of c-Met disrupts gene ex ...... ing liver regeneration in mice

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Daekwan Seo

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Elizabeth A Conner

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Pal Kaposi-Novak

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Snorri S Thorgeirsson

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Tsuyoshi Ishikawa

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Valentina M Factor

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P2860

Identification of the hepatocyte growth factor receptor as the c-met proto-oncogene product

The decision to enter mitosis: feedback and redundancy in the mitotic entry network

Mitotic HeLa cells contain a CENP-E-associated minus end-directed microtubule motor

Plk1-dependent phosphorylation of FoxM1 regulates a transcriptional programme required for mitotic progression

The checkpoint protein MAD2 and the mitotic regulator CDC20 form a ternary complex with the anaphase-promoting complex to control anaphase initiation

A comparison of normalization methods for high density oligonucleotide array data based on variance and bias

Aurora-B phosphorylates Histone H3 at serine28 with regard to the mitotic chromosome condensation

Met, metastasis, motility and more

Induction of epithelial tubules by growth factor HGF depends on the STAT pathway

The ERK1/2 mitogen-activated protein kinase pathway as a master regulator of the G1- to S-phase transition

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Jesper B. Andersen

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2940888

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