The pollutant diethylhexyl phthalate regulates hepatic energy metabolism via species-specific PPARalpha-dependent mechanisms - Wikidata - awgldk - TriplyDB

The pollutant diethylhexyl phthalate regulates hepatic energy metabolism via species-specific PPARalpha-dependent mechanisms

The pollutant diethylhexyl phthalate regulates hepatic energy metabolism via species-specific PPARalpha-dependent mechanisms

http://www.wikidata.org/entity/Q33702683

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Do interactions between gut ecology and environmental chemicals contribute to obesity and diabetes?Phthalate exposure and childhood obesity Role of environmental chemicals in diabetes and obesity: a National Toxicology Program workshop review The paradox of progress: environmental disruption of metabolism and the diabetes epidemic Obesity, diabetes, and associated costs of exposure to endocrine-disrupting chemicals in the European Union The adverse cardiac effects of Di(2-ethylhexyl)phthalate and Bisphenol A The influence of phthalates and bisphenol A on the obesity development and glucose metabolism disorders.Influence of phthalates on glucose homeostasis and atherosclerosis in hyperlipidemic mice Effects of di(2-ethylhexyl) phthalate (DEHP) on female fertility and adipogenesis in C3H/N mice.Rodent thyroid, liver, and fetal testis toxicity of the monoester metabolite of bis-(2-ethylhexyl) tetrabromophthalate (tbph), a novel brominated flame retardant present in indoor dust Association of urinary concentrations of bisphenol A and phthalate metabolites with risk of type 2 diabetes: a prospective investigation in the Nurses' Health Study (NHS) and NHSII cohorts What Are We Putting in Our Food That Is Making Us Fat? Food Additives, Contaminants, and Other Putative Contributors to Obesity.Age and sex-specific relationships between phthalate exposures and obesity in Chinese children at puberty.Combined transcriptomic-(1)H NMR metabonomic study reveals that monoethylhexyl phthalate stimulates adipogenesis and glyceroneogenesis in human adipocytes.Ligand binding and activation of PPARγ by Firemaster® 550: effects on adipogenesis and osteogenesis in vitro Cellular and molecular effect of MEHP Involving LXRα in human fetal testis and ovary.The effects of Di-(2-ethylhexyl)-phthalate exposure on fertilization and embryonic development in vitro and testicular genomic mutation in vivo Evaluation of the INS-1 832/13 cell line as a beta-cell based screening system to assess pollutant effects on beta-cell function.Childhood obesity and environmental chemicals.Di-(2-ethylhexyl) phthalate metabolites in urine show age-related changes and associations with adiposity and parameters of insulin sensitivity in childhood.A Comparative Study of Mouse Hepatic and Intestinal Gene Expression Profiles under PPARα Knockout by Gene Set Enrichment Analysis Exposure to phthalates affects calcium handling and intercellular connectivity of human stem cell-derived cardiomyocytes Prenatal maternal blood triglyceride and fatty acid levels in relation to exposure to di(2-ethylhexyl)phthalate: a cross-sectional study Circulating levels of phthalate metabolites are associated with prevalent diabetes in the elderly Prenatal Phthalate Exposure and Childhood Growth and Blood Pressure: Evidence from the Spanish INMA-Sabadell Birth Cohort Study Urinary phthalate metabolite concentrations and diabetes among women in the National Health and Nutrition Examination Survey (NHANES) 2001-2008.In utero growth restriction and catch-up adipogenesis after developmental di (2-ethylhexyl) phthalate exposure cause glucose intolerance in adult male rats following a high-fat dietary challenge.The endocrine disruptor mono-(2-ethylhexyl) phthalate promotes adipocyte differentiation and induces obesity in mice.The Inflammation Response to DEHP through PPARγ in Endometrial Cells.Prenatal Phthalate Exposures and Body Mass Index Among 4- to 7-Year-old Children: A Pooled Analysis.Prenatal Phthalate Exposures and Childhood Fat Mass in a New York City Cohort Validation of a genomics-based hypothetical adverse outcome pathway: 2,4-dinitrotoluene perturbs PPAR signaling thus impairing energy metabolism and exercise endurance.A systematic approach for identifying and presenting mechanistic evidence in human health assessments.Adipocytes under assault: environmental disruption of adipose physiology.In utero exposure to di-(2-ethylhexyl) phthalate induces metabolic disorder and increases fat accumulation in visceral depots of C57BL/6J mice offspring.Phthalate exposure associated with self-reported diabetes among Mexican women.The hijacking of cellular signaling and the diabetes epidemic: mechanisms of environmental disruption of insulin action and glucose homeostasis.Mode of action framework analysis for receptor-mediated toxicity: The peroxisome proliferator-activated receptor alpha (PPARα) as a case study.Impact of di-ethylhexylphthalate exposure on metabolic programming in P19 ECC-derived cardiomyocytes.Endocrine-disrupting chemicals and the regulation of energy balance.

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The pollutant diethylhexyl phthalate regulates hepatic energy metabolism via species-specific PPARalpha-dependent mechanisms

http://www.wikidata.org/entity/Q33702683

description

2010 nî lūn-bûn

@nan

2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

The pollutant diethylhexyl pht ...... PPARalpha-dependent mechanisms

@ast

The pollutant diethylhexyl pht ...... PPARalpha-dependent mechanisms

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type

label

The pollutant diethylhexyl pht ...... PPARalpha-dependent mechanisms

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The pollutant diethylhexyl pht ...... PPARalpha-dependent mechanisms

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The pollutant diethylhexyl pht ...... PPARalpha-dependent mechanisms

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The pollutant diethylhexyl pht ...... PPARalpha-dependent mechanisms

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Environmental Health Perspectives

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The pollutant diethylhexyl pht ...... PPARalpha-dependent mechanisms

@en

P2093

Alan Gerber

http://www.w3.org/2001/XMLSchema#string

Carine Winkler

http://www.w3.org/2001/XMLSchema#string

Cristina Casals-Casas

http://www.w3.org/2001/XMLSchema#string

Elodie Bedu

http://www.w3.org/2001/XMLSchema#string

Frank J Gonzalez

http://www.w3.org/2001/XMLSchema#string

Jérôme N Feige

http://www.w3.org/2001/XMLSchema#string

Laurent Gelman

http://www.w3.org/2001/XMLSchema#string

Manuel Bueno

http://www.w3.org/2001/XMLSchema#string

Qian Yang

http://www.w3.org/2001/XMLSchema#string

P2860

FGF-21 as a novel metabolic regulator.

Concentrations of urinary phthalate metabolites are associated with increased waist circumference and insulin resistance in adult U.S. males

Developmental exposure to endocrine disruptors and the obesity epidemic

Activation of PPARalpha and PPARgamma by environmental phthalate monoesters

Perturbed nuclear receptor signaling by environmental obesogens as emerging factors in the obesity crisis

Resveratrol improves mitochondrial function and protects against metabolic disease by activating SIRT1 and PGC-1alpha

Organotin compounds promote adipocyte differentiation as agonists of the peroxisome proliferator-activated receptor gamma/retinoid X receptor pathway

Endocrine-disrupting organotin compounds are potent inducers of adipogenesis in vertebrates

trans-Activation of PPARalpha and PPARgamma by structurally diverse environmental chemicals

Corepressors selectively control the transcriptional activity of PPARgamma in adipocytes

P304

234-241

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scholarly article

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10.1289/EHP.0901217

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2

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P478

118

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Béatrice Desvergne

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2010-02-01T00:00:00Z

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41398750

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20123618

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2831923

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