ADAMTS9 is a cell-autonomously acting, anti-angiogenic metalloprotease expressed by microvascular endothelial cells. - Wikidata - awgldk - TriplyDB

ADAMTS9 is a cell-autonomously acting, anti-angiogenic metalloprotease expressed by microvascular endothelial cells.

ADAMTS9 is a cell-autonomously acting, anti-angiogenic metalloprotease expressed by microvascular endothelial cells.

http://www.wikidata.org/entity/Q33703820

about

The ADAMTS (A Disintegrin and Metalloproteinase with Thrombospondin motifs) family Biosynthesis and expression of a disintegrin-like and metalloproteinase domain with thrombospondin-1 repeats-15: a novel versican-cleaving proteoglycanase Gene expression programs of mouse endothelial cells in kidney development and disease.A new Adamts9 conditional mouse allele identifies its non-redundant role in interdigital web regression.Extracellular protease ADAMTS9 suppresses esophageal and nasopharyngeal carcinoma tumor formation by inhibiting angiogenesis Pericellular versican regulates the fibroblast-myofibroblast transition: a role for ADAMTS5 protease-mediated proteolysis ADAMTS9-Mediated Extracellular Matrix Dynamics Regulates Umbilical Cord Vascular Smooth Muscle Differentiation and Rotation Analysis of uterine gene expression in interleukin-15 knockout mice reveals uterine natural killer cells do not play a major role in decidualization and associated angiogenesis ADAMTS-18 in the host tissues exerts little effect on breast tumor progress in a murine 4T1 breast cancer model Anti-angiogenic pathway associations of the 3p21.3 mapped BLU gene in nasopharyngeal carcinoma.Diabetes genes identified by genome-wide association studies are regulated in mice by nutritional factors in metabolically relevant tissues and by glucose concentrations in islets.Emerging Roles of ADAMTSs in Angiogenesis and Cancer.Impaired ADAMTS9 secretion: A potential mechanism for eye defects in Peters Plus Syndrome.Altered gene expression in dry age-related macular degeneration suggests early loss of choroidal endothelial cells Matricellular protein SPARCL1 regulates tumor microenvironment-dependent endothelial cell heterogeneity in colorectal carcinoma.ADAMTS6 suppresses tumor progression via the ERK signaling pathway and serves as a prognostic marker in human breast cancer.The murine allantois: a model system for the study of blood vessel formation.Vascular disrupting activity and the mechanism of action of EHT 6706, a novel anticancer tubulin polymerization inhibitor.The roles of ADAMTS in angiogenesis and cancer.Genetic and biochemical evidence that gastrulation defects in Pofut2 mutants result from defects in ADAMTS9 secretion.ADAMTS4 and its proteolytic fragments differentially affect melanoma growth and angiogenesis in mice.ADAMTS9 is a functional tumor suppressor through inhibiting AKT/mTOR pathway and associated with poor survival in gastric cancer.Cooperation of two ADAMTS metalloproteases in closure of the mouse palate identifies a requirement for versican proteolysis in regulating palatal mesenchyme proliferation.Differentially regulated ADAMTS1, 8, 9, and 18 in pancreas adenocarcinoma.Molecular Fingerprint for Terminal Abdominal Aortic Aneurysm Disease.Molecular Regulation of Sprouting Angiogenesis.ADAMTS13 and its variants promote angiogenesis via upregulation of VEGF and VEGFR2.ADAMTS9-Regulated Pericellular Matrix Dynamics Governs Focal Adhesion-Dependent Smooth Muscle Differentiation.Parallel Genome-wide Profiling of Coding and Non-coding RNAs to Identify Novel Regulatory Elements in Embryonic and Maturated Heart.Metalloproteinase-dependent and -independent processes contribute to inhibition of breast cancer cell migration, angiogenesis and liver metastasis by a disintegrin and metalloproteinase with thrombospondin motifs-15 ADAMTS9locus associates with increased risk of wet AMD

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ADAMTS9 is a cell-autonomously acting, anti-angiogenic metalloprotease expressed by microvascular endothelial cells.

http://www.wikidata.org/entity/Q33703820

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

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2010年論文

@zh-tw

2010年论文

@wuu

name

ADAMTS9 is a cell-autonomously ...... crovascular endothelial cells.

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ADAMTS9 is a cell-autonomously ...... crovascular endothelial cells.

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type

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ADAMTS9 is a cell-autonomously ...... crovascular endothelial cells.

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ADAMTS9 is a cell-autonomously ...... crovascular endothelial cells.

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prefLabel

ADAMTS9 is a cell-autonomously ...... crovascular endothelial cells.

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ADAMTS9 is a cell-autonomously ...... crovascular endothelial cells.

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AJPATH.2010.090655

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The American Journal of Pathology

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ADAMTS9 is a cell-autonomously ...... crovascular endothelial cells.

@en

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Bon-Hun Koo

http://www.w3.org/2001/XMLSchema#string

Courtney M Nelson

http://www.w3.org/2001/XMLSchema#string

Daniel J Lindner

http://www.w3.org/2001/XMLSchema#string

David M Coe

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Laura J Dixon

http://www.w3.org/2001/XMLSchema#string

Lauren W Wang

http://www.w3.org/2001/XMLSchema#string

Mary Elizabeth Young

http://www.w3.org/2001/XMLSchema#string

Robert P T Somerville

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Suneel S Apte

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P2860

Human Ehlers-Danlos syndrome type VII C and bovine dermatosparaxis are caused by mutations in the procollagen I N-proteinase gene

METH-1, a human ortholog of ADAMTS-1, and METH-2 are members of a new family of proteins with angio-inhibitory activity

Cell-surface processing of pro-ADAMTS9 by furin

Molecular cloning of a gene encoding a new type of metalloproteinase-disintegrin family protein with thrombospondin motifs as an inflammation associated gene

Characterization of ADAMTS-9 and ADAMTS-20 as a distinct ADAMTS subfamily related to Caenorhabditis elegans GON-1

ADAMTS10 mutations in autosomal recessive Weill-Marchesani syndrome

Physiological levels of tumstatin, a fragment of collagen IV alpha3 chain, are generated by MMP-9 proteolysis and suppress angiogenesis via alphaV beta3 integrin

ADAMTS1/METH1 inhibits endothelial cell proliferation by direct binding and sequestration of VEGF165

Identification of a tumor suppressive critical region mapping to 3p14.2 in esophageal squamous cell carcinoma and studies of a candidate tumor suppressor gene, ADAMTS9

A disintegrin-like and metalloprotease (reprolysin type) with thrombospondin type 1 motifs: the ADAMTS family

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1494-1504

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scholarly article

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10.2353/AJPATH.2010.090655

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3

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176

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2010-01-21T00:00:00Z

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41111589

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20093484

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