Effects of foot-and-mouth disease virus nonstructural proteins on the structure and function of the early secretory pathway: 2BC but not 3A blocks endoplasmic reticulum-to-Golgi transport.
about
ACBD3-mediated recruitment of PI4KB to picornavirus RNA replication sitesValosin-containing protein (VCP/p97) is required for poliovirus replication and is involved in cellular protein secretion pathway in poliovirus infectionBiological function of Foot-and-mouth disease virus non-structural proteins and non-coding elementsViroporin Activity of the Foot-and-Mouth Disease Virus Non-Structural 2B ProteinThe encephalomyocarditis virusFoot-and-mouth disease virus 2C is a hexameric AAA+ protein with a coordinated ATP hydrolysis mechanismA complex comprising phosphatidylinositol 4-kinase IIIβ, ACBD3, and Aichi virus proteins enhances phosphatidylinositol 4-phosphate synthesis and is critical for formation of the viral replication complex.Foot-and-mouth disease virus infection inhibits LGP2 protein expression to exaggerate inflammatory response and promote viral replication.Feline calicivirus p32, p39 and p30 proteins localize to the endoplasmic reticulum to initiate replication complex formationFoot-and-mouth disease virus replicates only transiently in well-differentiated porcine nasal epithelial cells.Human rhinovirus 16 causes Golgi apparatus fragmentation without blocking protein secretion.COPI is required for enterovirus 71 replicationFoot-and-mouth disease virus 3C protease induces fragmentation of the Golgi compartment and blocks intra-Golgi transport.Mitochondrial genomes are retained by selective constraints on protein targeting.Inhibition of protein trafficking by coxsackievirus b3: multiple viral proteins target a single organelleInduction of foot-and-mouth disease virus-specific cytotoxic T cell killing by vaccination.Foot-and-mouth disease virus, but not bovine enterovirus, targets the host cell cytoskeleton via the nonstructural protein 3Cpro.Bovine plasmacytoid dendritic cells are the major source of type I interferon in response to foot-and-mouth disease virus in vitro and in vivo.Effects of picornavirus 3A Proteins on Protein Transport and GBF1-dependent COP-I recruitment.Inhibition of the secretory pathway by foot-and-mouth disease virus 2BC protein is reproduced by coexpression of 2B with 2C, and the site of inhibition is determined by the subcellular location of 2C.Protein 2B of Coxsackievirus B3 Induces Autophagy Relying on Its Transmembrane Hydrophobic SequencesTransgenic expression of non-structural genes of Theiler's virus suppresses initial viral replication and pathogenesis of demyelination.A continuous bovine kidney cell line constitutively expressing bovine αvβ6 integrin has increased susceptibility to foot-and-mouth disease virusCharacterization of cytotoxic T lymphocyte function after foot-and-mouth disease virus infection and vaccination.A role for endoplasmic reticulum exit sites in foot-and-mouth disease virus infectionFoot-and-Mouth Disease Virus Viroporin 2B Antagonizes RIG-I-Mediated Antiviral Effects by Inhibition of Its Protein Expression.Evidence of activation and suppression during the early immune response to foot-and-mouth disease virus.The pathogenesis of foot-and-mouth disease II: viral pathways in swine, small ruminants, and wildlife; myotropism, chronic syndromes, and molecular virus-host interactions.Viral infection: Moving through complex and dynamic cell-membrane structures.Foot-and-mouth disease virus replicates independently of phosphatidylinositol 4-phosphate and type III phosphatidylinositol 4-kinases.Membrane topology and cellular dynamics of foot-and-mouth disease virus 3A protein.African swine fever virus causes microtubule-dependent dispersal of the trans-golgi network and slows delivery of membrane protein to the plasma membrane.Mutations that hamper dimerization of foot-and-mouth disease virus 3A protein are detrimental for infectivity.Evolution of poliovirus defective interfering particles expressing Gaussia luciferase.Bioinformatics and Molecular Analysis of the Evolutionary Relationship between Bovine Rhinitis A Viruses and Foot-And-Mouth Disease Virus.Functional analysis of picornavirus 2B proteins: effects on calcium homeostasis and intracellular protein trafficking.Guinea pig-adapted foot-and-mouth disease virus with altered receptor recognition can productively infect a natural host.The Amino Acid Substitution Q65H in the 2C Protein of Swine Vesicular Disease Virus Confers Resistance to Golgi Disrupting Drugs.Foot-and-mouth disease virus induces autophagosomes during cell entry via a class III phosphatidylinositol 3-kinase-independent pathwayNonstructural proteins 2C and 3D are involved in autophagy as induced by the encephalomyocarditis virus.
P2860
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P2860
Effects of foot-and-mouth disease virus nonstructural proteins on the structure and function of the early secretory pathway: 2BC but not 3A blocks endoplasmic reticulum-to-Golgi transport.
description
2005 nî lūn-bûn
@nan
2005 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Effects of foot-and-mouth dise ...... reticulum-to-Golgi transport.
@ast
Effects of foot-and-mouth dise ...... reticulum-to-Golgi transport.
@en
type
label
Effects of foot-and-mouth dise ...... reticulum-to-Golgi transport.
@ast
Effects of foot-and-mouth dise ...... reticulum-to-Golgi transport.
@en
prefLabel
Effects of foot-and-mouth dise ...... reticulum-to-Golgi transport.
@ast
Effects of foot-and-mouth dise ...... reticulum-to-Golgi transport.
@en
P2093
P2860
P1433
P1476
Effects of foot-and-mouth dise ...... reticulum-to-Golgi transport.
@en
P2093
Caroline Knox
Gareth Howell
Graham J Belsham
Katy Moffat
Martin D Ryan
Paul Monaghan
Thomas Wileman
P2860
P304
P356
10.1128/JVI.79.7.4382-4395.2005
P577
2005-04-01T00:00:00Z