Phenotypic plasticity of neoplastic ovarian epithelium: unique cadherin profiles in tumor progression - Wikidata - awgldk - TriplyDB

Phenotypic plasticity of neoplastic ovarian epithelium: unique cadherin profiles in tumor progression

Phenotypic plasticity of neoplastic ovarian epithelium: unique cadherin profiles in tumor progression

http://www.wikidata.org/entity/Q33719284

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CD164 regulates the tumorigenesis of ovarian surface epithelial cells through the SDF-1α/CXCR4 axis The Potential Role of the Proteases Cathepsin D and Cathepsin L in the Progression and Metastasis of Epithelial Ovarian Cancer Role of mesenchymal cells in the natural history of ovarian cancer: a review E-cadherin's dark side: possible role in tumor progression Mechanisms involved in breast cancer liver metastasis R-Ketorolac Targets Cdc42 and Rac1 and Alters Ovarian Cancer Cell Behaviors Critical for Invasion and Metastasis Identification of ovarian cancer metastatic miRNAs Regulation of the Epithelial-Mesenchymal Transition by Claudin-3 and Claudin-4 Targeting the EGF receptor for ovarian cancer therapy.Inhibition of the JAK2/STAT3 pathway in ovarian cancer results in the loss of cancer stem cell-like characteristics and a reduced tumor burden Hydrogen peroxide mediates EGF-induced down-regulation of E-cadherin expression via p38 MAPK and snail in human ovarian cancer cells.E-cadherin expression in ovarian cancer in the laying hen, Gallus domesticus, compared to human ovarian cancer.Interplay between neural-cadherin and vascular endothelial-cadherin in breast cancer progression.Epidermal growth factor receptor-mediated membrane type 1 matrix metalloproteinase endocytosis regulates the transition between invasive versus expansive growth of ovarian carcinoma cells in three-dimensional collagen Analysis of epithelial and mesenchymal markers in ovarian cancer reveals phenotypic heterogeneity and plasticity.Indometacin ameliorates high glucose-induced proliferation and invasion via modulation of e-cadherin in pancreatic cancer cells.Heterogeneous Cadherin Expression and Multicellular Aggregate Dynamics in Ovarian Cancer Dissemination Differential expressions of adhesive molecules and proteases define mechanisms of ovarian tumor cell matrix penetration/invasion.Breast carcinoma cells re-express E-cadherin during mesenchymal to epithelial reverting transition.Neuronal transcription factor Brn-3a(l) is over expressed in high-grade ovarian carcinomas and tumor cells from ascites of patients with advanced-stage ovarian cancer Ovarian cancer development and metastasis.Uncovering molecular events associated with the chemosuppressive effects of flaxseed: a microarray analysis of the laying hen model of ovarian cancer The cadherin switch in ovarian high-grade serous carcinoma is associated with disease progression.Assessment of ovarian cancer spheroid attachment and invasion of mesothelial cells in real time.Paclitaxel resistance and multicellular spheroid formation are induced by kallikrein-related peptidase 4 in serous ovarian cancer cells in an ascites mimicking microenvironment.Multivalent epigenetic marks confer microenvironment-responsive epigenetic plasticity to ovarian cancer cells.Fibroblast growth factor 2 induces E-cadherin down-regulation via PI3K/Akt/mTOR and MAPK/ERK signaling in ovarian cancer cells Emerging roles of Kruppel-like factor 6 and Kruppel-like factor 6 splice variant 1 in ovarian cancer progression and treatment.Cancer-associated fibroblasts and their putative role in potentiating the initiation and development of epithelial ovarian cancer.MUC16 mucin (CA125) regulates the formation of multicellular aggregates by altering β-catenin signaling Integrin regulation of beta-catenin signaling in ovarian carcinoma.Diverse mechanisms for activation of Wnt signalling in the ovarian tumour microenvironment.Immunohistological insight into the correlation between neuropilin-1 and epithelial-mesenchymal transition markers in epithelial ovarian cancer.Contributions of the Epidermal Growth Factor Receptor to Acquisition of Platinum Resistance in Ovarian Cancer Cells.Pygopus-2 promotes invasion and metastasis of hepatic carcinoma cell by decreasing E-cadherin expression.Evolution of adaptive phenotypic traits without positive Darwinian selection.The cell surface glycoprotein CUB domain-containing protein 1 (CDCP1) contributes to epidermal growth factor receptor-mediated cell migration Increased DNA methyltransferase activity and DNA methylation following Epidermal Growth Factor stimulation in ovarian cancer cells.Lysophosphatidic Acid disrupts junctional integrity and epithelial cohesion in ovarian cancer cells.Epithelial-mesenchymal transition in ovarian carcinoma

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Phenotypic plasticity of neoplastic ovarian epithelium: unique cadherin profiles in tumor progression

http://www.wikidata.org/entity/Q33719284

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2008 nî lūn-bûn

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2008 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2008 թվականի ապրիլին հրատարակված գիտական հոդված

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2008年の論文

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M Sharon Stack

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Reema Zeineldin

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P2860

Disaggregation and invasion of ovarian carcinoma ascites spheroids

Opinion: migrating cancer stem cells - an integrated concept of malignant tumour progression

Proteinase requirements of epidermal growth factor-induced ovarian cancer cell invasion

Possible involvement of TWIST in enhanced peritoneal metastasis of epithelial ovarian carcinoma

Molecular pathways regulating EGF-induced epithelio-mesenchymal transition in human ovarian surface epithelium

Cancer statistics, 2007

Snail, Zeb and bHLH factors in tumour progression: an alliance against the epithelial phenotype?

Drug penetration in solid tumours

Molecular requirements for epithelial-mesenchymal transition during tumor progression

The cell of origin of ovarian epithelial tumors and the ovarian surface epithelium dogma: does the emperor have no clothes?

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phenotypic plasticity

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