Staphylococcal peptidoglycan co-localizes with Nod2 and TLR2 and activates innate immune response via both receptors in primary murine keratinocytes
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Lipoproteins of Gram-Positive Bacteria: Key Players in the Immune Response and VirulenceNovel insights into the role of immune cells in skin and inducible skin-associated lymphoid tissue (iSALT)The Staphylococcus aureus peptidoglycan protects mice against the pathogen and eradicates experimentally induced infectionStimulation of platelet apoptosis by peptidoglycan from Staphylococcus aureus 113.CcpA Affects Infectivity of Staphylococcus aureus in a Hyperglycemic EnvironmentHost-induced bacterial cell wall decomposition mediates pattern-triggered immunity in Arabidopsis.Cell wall antibiotics provoke accumulation of anchored mCherry in the cross wall of Staphylococcus aureus.Convergence of innate immunity and insulin resistance as evidenced by increased nucleotide oligomerization domain (NOD) expression and signaling in monocytes from patients with type 2 diabetes.Pathogen-mediated proteolysis of the cell death regulator RIPK1 and the host defense modulator RIPK2 in human aortic endothelial cells.Functional characterizations of RIG-I to GCRV and viral/bacterial PAMPs in grass carp Ctenopharyngodon idella.Peptidoglycan Induces the Production of Interleukin-8 via Calcium Signaling in Human Gingival Epithelium.Contribution of toll-like receptor 2 to the innate response against Staphylococcus aureus infection in mice.NOD1 activators link innate immunity to insulin resistanceArabidopsis lysin-motif proteins LYM1 LYM3 CERK1 mediate bacterial peptidoglycan sensing and immunity to bacterial infectionThe νSaα Specific Lipoprotein Like Cluster (lpl) of S. aureus USA300 Contributes to Immune Stimulation and Invasion in Human Cells.The Glycogen Synthase Kinase 3α and β Isoforms Differentially Regulates Interleukin-12p40 Expression in Endothelial Cells Stimulated with Peptidoglycan from Staphylococcus aureusThe Role of TLR2 in Infection and ImmunityHost response signature to Staphylococcus aureus alpha-hemolysin implicates pulmonary Th17 responseEstradiol alters the immune-responsiveness of cervical epithelial cells stimulated with ligands of Toll-like receptors 2 and 4A novel DNA vaccine expressing the Ag85A-HA2 fusion protein provides protection against influenza A virus and Staphylococcus aureusBacteria activate sensory neurons that modulate pain and inflammation.Post-transcriptional regulation of cystic fibrosis transmembrane conductance regulator expression and function by microRNAs.Innate and adaptive immune responses against Staphylococcus aureus skin infections.Innate immune sensing 2.0 - from linear activation pathways to fine tuned and regulated innate immune networks.The function of TLR2 during staphylococcal diseases.Receptor-like kinases in plant innate immunity.Collaborative action of Toll-like and NOD-like receptors as modulators of the inflammatory response to pathogenic bacteria.Neisseria gonorrhoeae Lytic Transglycosylases LtgA and LtgD Reduce Host Innate Immune Signaling through TLR2 and NOD2.Lipoteichoic acids as a major virulence factor causing inflammatory responses via Toll-like receptor 2.Suppression of osteoblast Toll-like receptor 2 signaling by endothelin-1.Shared characteristics between Mycobacterium tuberculosis and fungi contribute to virulence.Signaling via pattern recognition receptors NOD2 and TLR2 contributes to immunomodulatory control of lethal pneumovirus infectionInflammatory responses to a Clostridium perfringens type A strain and α-toxin in primary intestinal epithelial cells of chicken embryos.Differential induction of inflammatory cytokines and reactive oxygen species in murine peritoneal macrophages and resident fresh bone marrow cells by acute staphylococcus aureus infection: contribution of toll-like receptor 2 (TLR2).NOD2 stimulation by Staphylococcus aureus-derived peptidoglycan is boosted by Toll-like receptor 2 costimulation with lipoproteins in dendritic cells.From cells to muropeptide structures in 24 h: peptidoglycan mapping by UPLC-MS.Toll-like receptor 2 has a prominent but nonessential role in innate immunity to Staphylococcus aureus pneumonia.Staphylococcus aureus modulation of innate immune responses through Toll-like (TLR), (NOD)-like (NLR) and C-type lectin (CLR) receptorsCommentary: Membrane-Derived Vesicles Promote Bacterial Virulence and Confer Protective Immunity in Murine Infection Models
P2860
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P2860
Staphylococcal peptidoglycan co-localizes with Nod2 and TLR2 and activates innate immune response via both receptors in primary murine keratinocytes
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Staphylococcal peptidoglycan c ...... n primary murine keratinocytes
@ast
Staphylococcal peptidoglycan c ...... n primary murine keratinocytes
@en
type
label
Staphylococcal peptidoglycan c ...... n primary murine keratinocytes
@ast
Staphylococcal peptidoglycan c ...... n primary murine keratinocytes
@en
prefLabel
Staphylococcal peptidoglycan c ...... n primary murine keratinocytes
@ast
Staphylococcal peptidoglycan c ...... n primary murine keratinocytes
@en
P2093
P2860
P1433
P1476
Staphylococcal peptidoglycan c ...... n primary murine keratinocytes
@en
P2093
Friedrich Götz
Maria Anna Müller-Anstett
Martin Schaller
Mulugeta Nega
Patrick Müller
Susanne Kaesler
Till Albrecht
P2860
P304
P356
10.1371/JOURNAL.PONE.0013153
P407
P577
2010-10-07T00:00:00Z