Complement activation in multiple sclerosis plaques: an immunohistochemical analysis.
about
miRNAs Participate in MS Pathological Processes and Its Therapeutic ResponseEmerging and Novel Functions of Complement Protein C1qNutrition facts in multiple sclerosisProteomic analysis of cerebrospinal fluid for relapsing-remitting multiple sclerosis and clinically isolated syndromeComplementing the inflammasome.Stepchild or Prodigy? Neuroprotection in Multiple Sclerosis (MS) Research.Adipsin Is Associated with Multiple Sclerosis: A Follow-Up Study of Adipokines.Protein-Based Classifier to Predict Conversion from Clinically Isolated Syndrome to Multiple SclerosisComplement is activated in progressive multiple sclerosis cortical grey matter lesions.Next generation transcriptomics and genomics elucidate biological complexity of microglia in health and disease.The role of complement in neurological and neuropsychiatric diseases.Axonal degeneration in multiple sclerosis: defining therapeutic targets by identifying the causes of pathology.Cell specificity dictates similarities in gene expression in multiple sclerosis, Parkinson's disease, and Alzheimer's disease.Complement regulation and kidney diseases: recent knowledge of the double-edged roles of complement activation in nephrology.Tissue microarray methodology identifies complement pathway activation and dysregulation in progressive multiple sclerosis.Calcineurin proteolysis in astrocytes: Implications for impaired synaptic functionComplement C1q-C3-associated synaptic changes in multiple sclerosis hippocampus.Complement activation and expression during chronic relapsing experimental autoimmune encephalomyelitis in the Biozzi ABH mouse.Increase in NF-κB-sensitive miRNA-146a and miRNA-155 in multiple sclerosis (MS) and pro-inflammatory neurodegeneration.Therapeutic potential of curcumin for multiple sclerosis.Complement in the pathogenesis of Alzheimer's disease.The complement system as a biomarker of disease activity and response to treatment in multiple sclerosis.Gut dysbiosis breaks immunological tolerance toward the central nervous system during young adulthood.Multiple sclerosis: Serum anti-CNS autoantibodies.Antineutrophil Cytoplasmic Antibody and Multiple Sclerosis.Systemic inhibition of the membrane attack complex impedes neuroinflammation in chronic relapsing experimental autoimmune encephalomyelitis.Identification of brain antigens recognized by autoantibodies in experimental autoimmune encephalomyelitis-induced animals treated with etomoxir or interferon-β.Investigation of ischemic and demyelinating lesions by cerebral vasoreactivity based on transcranial Doppler sonography: a comparative study
P2860
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P2860
Complement activation in multiple sclerosis plaques: an immunohistochemical analysis.
description
2014 nî lūn-bûn
@nan
2014 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Complement activation in multiple sclerosis plaques: an immunohistochemical analysis.
@ast
Complement activation in multiple sclerosis plaques: an immunohistochemical analysis.
@en
type
label
Complement activation in multiple sclerosis plaques: an immunohistochemical analysis.
@ast
Complement activation in multiple sclerosis plaques: an immunohistochemical analysis.
@en
prefLabel
Complement activation in multiple sclerosis plaques: an immunohistochemical analysis.
@ast
Complement activation in multiple sclerosis plaques: an immunohistochemical analysis.
@en
P2093
P2860
P356
P1476
Complement activation in multiple sclerosis plaques: an immunohistochemical analysis.
@en
P2093
Bethan Dancey
Claire L Harris
Gillian Ingram
James W Neal
Neil P Robertson
Sam Loveless
Svetlana Hakobyan
P2860
P2888
P356
10.1186/2051-5960-2-53
P577
2014-05-09T00:00:00Z
P5875
P6179
1038923504