Mice over-expressing the myocardial creatine transporter develop progressive heart failure and show decreased glycolytic capacity
about
Investigating cardiac energetics in heart failureDetermining Maximum Glycolytic Capacity Using Extracellular Flux Measurements(1)H-MR spectroscopy for analysis of cardiac lipid and creatine metabolism.Stoichiometry of STAT3 and mitochondrial proteins: Implications for the regulation of oxidative phosphorylation by protein-protein interactions.Heart failure induces significant changes in nuclear pore complex of human cardiomyocytes.In vivo proton T1 relaxation times of mouse myocardial metabolites at 9.4 T.Homogenous protein programming in the mammalian left and right ventricle free walls.Augmentation of Creatine in the Heart.Moderate elevation of intracellular creatine by targeting the creatine transporter protects mice from acute myocardial infarctionMagnetic resonance imaging and spectroscopy of the murine cardiovascular systemSystems Biology and Noninvasive Imaging of Atherosclerosis.Proteomic and metabolomic changes driven by elevating myocardial creatine suggest novel metabolic feedback mechanismsNormal cardiac function in mice with supraphysiological cardiac creatine levelsHomoarginine in the renal and cardiovascular systems.MRS: a noninvasive window into cardiac metabolism.Rearrangement of energetic and substrate utilization networks compensate for chronic myocardial creatine kinase deficiency.Metabolic Reprogramming via PPARα Signaling in Cardiac Hypertrophy and Failure From Metabolomics to Epigenetics.Muscle energy stores and stroke rates of emperor penguins: implications for muscle metabolism and dive performanceMetabolic homeostasis is maintained in myocardial hibernation by adaptive changes in the transcriptome and proteome.A role for thioredoxin-interacting protein (Txnip) in cellular creatine homeostasis.Dioxygen and Metabolism; Dangerous Liaisons in Cardiac Function and Disease.Atorvastatin Attenuates Metabolic Remodeling in Ischemic Myocardium through the Downregulation of UCP2 Expression.The creatine kinase system as a therapeutic target for myocardial ischaemia-reperfusion injury
P2860
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P2860
Mice over-expressing the myocardial creatine transporter develop progressive heart failure and show decreased glycolytic capacity
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Mice over-expressing the myoca ...... decreased glycolytic capacity
@ast
Mice over-expressing the myoca ...... decreased glycolytic capacity
@en
type
label
Mice over-expressing the myoca ...... decreased glycolytic capacity
@ast
Mice over-expressing the myoca ...... decreased glycolytic capacity
@en
prefLabel
Mice over-expressing the myoca ...... decreased glycolytic capacity
@ast
Mice over-expressing the myoca ...... decreased glycolytic capacity
@en
P2093
P2860
P50
P1476
Mice over-expressing the myoca ...... decreased glycolytic capacity
@en
P2093
Angel M Aponte
Colin O Wu
Julie Wallis
Jurgen E Schneider
Kieran Clarke
Liam Sebag-Montefiore
Michiel Ten Hove
Robert S Balaban
Stefan Neubauer
P2860
P304
P356
10.1016/J.YJMCC.2009.10.033
P577
2009-11-11T00:00:00Z