Fibulin-1 is increased in asthma--a novel mediator of airway remodeling? - Wikidata - awgldk - TriplyDB

Fibulin-1 is increased in asthma--a novel mediator of airway remodeling?

Fibulin-1 is increased in asthma--a novel mediator of airway remodeling?

http://www.wikidata.org/entity/Q33725506

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Fibulin1C peptide induces cell attachment and extracellular matrix deposition in lung fibroblasts Point: alterations in airway smooth muscle phenotype do/do not cause airway hyperresponsiveness in asthma Airway smooth muscle in asthma: just a target for bronchodilation?The expression and activity of cathepsins D, H and K in asthmatic airways.Differential regulation of extracellular matrix and soluble fibulin-1 levels by TGF-β₁ in airway smooth muscle cells Fibulin-1 regulates the pathogenesis of tissue remodeling in respiratory diseases Innate-like functions of natural killer T cell subsets result from highly divergent gene programs Airway remodelling and inflammation in asthma are dependent on the extracellular matrix protein fibulin-1c.Mechanisms and treatments for severe, steroid-resistant allergic airway disease and asthma.SiMA: A simplified migration assay for analyzing neutrophil migration.The inhibitor of semicarbazide-sensitive amine oxidase, PXS-4728A, ameliorates key features of chronic obstructive pulmonary disease in a mouse model.Asthma Biomarkers: Do They Bring Precision Medicine Closer to the Clinic?Total and isoform-specific quantitative assessment of circulating fibulin-1 using selected reaction monitoring MS and time-resolved immunofluorometry.Phenotype and Functional Features of Human Telomerase Reverse Transcriptase Immortalized Human Airway Smooth Muscle Cells from Asthmatic and Non-Asthmatic Donors.Idiopathic Pulmonary Fibrosis: Molecular Endotypes of Fibrosis Stratifying Existing and Emerging Therapies.Fibroblast-to-myofibroblast transition in bronchial asthma

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Fibulin-1 is increased in asthma--a novel mediator of airway remodeling?

http://www.wikidata.org/entity/Q33725506

description

2010 nî lūn-bûn

@nan

2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Fibulin-1 is increased in asthma--a novel mediator of airway remodeling?

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Fibulin-1 is increased in asthma--a novel mediator of airway remodeling?

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type

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Fibulin-1 is increased in asthma--a novel mediator of airway remodeling?

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Fibulin-1 is increased in asthma--a novel mediator of airway remodeling?

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Fibulin-1 is increased in asthma--a novel mediator of airway remodeling?

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Fibulin-1 is increased in asthma--a novel mediator of airway remodeling?

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JOURNAL.PONE.0013360

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Fibulin-1 is increased in asthma--a novel mediator of airway remodeling?

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Carolyn Williams

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Janette K Burgess

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Judith L Black

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Justine Y Lau

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Lyn M Moir

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Melissa Baraket

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Nicole G Hansbro

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Paul S Foster

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Philip M Hansbro

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Steve D Wilton

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P2860

Fibulin-1 suppression of fibronectin-regulated cell adhesion and motility

The association of human fibulin-1 with elastic fibers: an immunohistological, ultrastructural, and RNA study

The self-association and fibronectin-binding sites of fibulin-1 map to calcium-binding epidermal growth factor-like domains

Functional classes of bronchial mucosa genes that are differentially expressed in asthma

The effect of IL-13 and IL-13R130Q, a naturally occurring IL-13 polymorphism, on the gene expression of human airway smooth muscle cells

Correlation between airway responsiveness and proteoglycan production by bronchial fibroblasts from normal and asthmatic subjects.

Immunological and pathobiological roles of fibulin-1 in breast cancer.

Hyperplasia of smooth muscle in mild to moderate asthma without changes in cell size or gene expression.

Role of human airway smooth muscle in altered extracellular matrix production in asthma.

Epithelial-mesenchymal interactions in the pathogenesis of asthma.

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e13360

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10.1371/JOURNAL.PONE.0013360

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Emma L. Beckett

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2010-10-13T00:00:00Z

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47520894

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