HP1α mediates defective heterochromatin repair and accelerates senescence in Zmpste24-deficient cells.
about
Sustained accumulation of prelamin A and depletion of lamin A/C both cause oxidative stress and mitochondrial dysfunction but induce different cell fatesBarrier-to-autointegration factor (BAF) involvement in prelamin A-related chromatin organization changes.HistoneH3 demethylase JMJD2A promotes growth of liver cancer cells through up-regulating miR372.
P2860
HP1α mediates defective heterochromatin repair and accelerates senescence in Zmpste24-deficient cells.
description
2014 nî lūn-bûn
@nan
2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
HP1α mediates defective hetero ...... e in Zmpste24-deficient cells.
@ast
HP1α mediates defective hetero ...... e in Zmpste24-deficient cells.
@en
type
label
HP1α mediates defective hetero ...... e in Zmpste24-deficient cells.
@ast
HP1α mediates defective hetero ...... e in Zmpste24-deficient cells.
@en
prefLabel
HP1α mediates defective hetero ...... e in Zmpste24-deficient cells.
@ast
HP1α mediates defective hetero ...... e in Zmpste24-deficient cells.
@en
P2093
P2860
P356
P1433
P1476
HP1α mediates defective hetero ...... e in Zmpste24-deficient cells.
@en
P2093
Baohua Liu
Guangqian Zhou
Huiling Zheng
Liyun Gong
Vaidehi Krishnan
Xianhui Yin
P2860
P304
P356
10.4161/CC.28105
P577
2014-02-14T00:00:00Z