Discovery of recurrent t(6;7)(p25.3;q32.3) translocations in ALK-negative anaplastic large cell lymphomas by massively parallel genomic sequencing - Wikidata - awgldk - TriplyDB

Discovery of recurrent t(6;7)(p25.3;q32.3) translocations in ALK-negative anaplastic large cell lymphomas by massively parallel genomic sequencing

Discovery of recurrent t(6;7)(p25.3;q32.3) translocations in ALK-negative anaplastic large cell lymphomas by massively parallel genomic sequencing

http://www.wikidata.org/entity/Q33732485

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Mate-Pair Sequencing as a Powerful Clinical Tool for the Characterization of Cancers with a DNA Viral Etiology Peripheral T-cell and NK-cell lymphomas in the WHO classification: pearls and pitfalls ALK-positive anaplastic large cell lymphoma limited to the skin: clinical, histopathological and molecular analysis of 6 pediatric cases. A report from the ALCL99 study New molecular insights into peripheral T cell lymphomas Hypomethylation of dual specificity phosphatase 22 promoter correlates with duration of service in firefighters and is inducible by low-dose benzo[a]pyrene Multiple cutaneous lymphoproliferative disorders showing a retained tumor clone by T-cell receptor gene rearrangement analysis: a case series of four patients and review of the literature.Mate pair sequencing of whole-genome-amplified DNA following laser capture microdissection of prostate cancer.The heterogeneous landscape of ALK negative ALCL.GATA-3 expression identifies a high-risk subset of PTCL, NOS with distinct molecular and clinical features.Genomic organization and evolution of double minutes/homogeneously staining regions with MYC amplification in human cancer.What is a true ALCL?ALK-negative anaplastic large cell lymphoma is a genetically heterogeneous disease with widely disparate clinical outcomes.Identification of submicroscopic genetic changes and precise breakpoint mapping in myelofibrosis using high resolution mate-pair sequencing.Genome-wide analysis reveals recurrent structural abnormalities of TP63 and other p53-related genes in peripheral T-cell lymphomas.Global mutational profiling of formalin-fixed human colon cancers from a pathology archive BIMA V3: an aligner customized for mate pair library sequencing.Genomic rearrangements define lineage relationships between adjacent lepidic and invasive components in lung adenocarcinoma.Chromosomal rearrangements and copy number abnormalities of TP63 correlate with p63 protein expression in lung adenocarcinoma.Identification of independent primary tumors and intrapulmonary metastases using DNA rearrangements in non-small-cell lung cancer.Expression of the chemokine receptor gene, CCR8, is associated With DUSP22 rearrangements in anaplastic large cell lymphoma Robustness of Next Generation Sequencing on Older Formalin-Fixed Paraffin-Embedded Tissue.How I treat the peripheral T-cell lymphomas Whole-genome sequencing reveals oncogenic mutations in mycosis fungoides.Oncogenic role of miR-155 in anaplastic large cell lymphoma lacking the t(2;5) translocation.Secondary cutaneous involvement by systemic anaplastic lymphoma kinase-negative anaplastic large-cell lymphoma with 6p25.3 rearrangement.Genetics of anaplastic large cell lymphoma Morphologic Features of ALK-negative Anaplastic Large Cell Lymphomas With DUSP22 Rearrangements The 2016 revision of the World Health Organization classification of lymphoid neoplasms.Adult systemic anaplastic large-cell lymphoma: recommendations for diagnosis and management Pathobiology of T-cell and NK-cell lymphomas.MicroRNA expression profiling identifies molecular signatures associated with anaplastic large cell lymphoma Activation of TAK1 by MYD88 L265P drives malignant B-cell Growth in non-Hodgkin lymphoma.Molecular alterations and tumor suppressive function of the DUSP22 (Dual Specificity Phosphatase 22) gene in peripheral T-cell lymphoma subtypes.Update: peripheral T-cell lymphomas.Making a long story short: noncoding RNAs and chromosome change.Novel treatment avenues for peripheral T-cell lymphomas.Beyond NPM-anaplastic lymphoma kinase driven lymphomagenesis: alternative drivers in anaplastic large cell lymphoma.Peripheral T cell lymphoma in Asia.Molecular genetics of peripheral T-cell lymphomas.Harnessing massively parallel sequencing in personalized head and neck oncology.

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Discovery of recurrent t(6;7)(p25.3;q32.3) translocations in ALK-negative anaplastic large cell lymphomas by massively parallel genomic sequencing

http://www.wikidata.org/entity/Q33732485

description

2010 nî lūn-bûn

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2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

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2010年論文

@zh-tw

2010年论文

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name

Discovery of recurrent t(6;7)( ...... ly parallel genomic sequencing

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Discovery of recurrent t(6;7)( ...... ly parallel genomic sequencing

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type

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Discovery of recurrent t(6;7)( ...... ly parallel genomic sequencing

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Discovery of recurrent t(6;7)( ...... ly parallel genomic sequencing

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Discovery of recurrent t(6;7)( ...... ly parallel genomic sequencing

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Discovery of recurrent t(6;7)( ...... ly parallel genomic sequencing

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BLOOD-2010-08-303305

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Discovery of recurrent t(6;7)( ...... ly parallel genomic sequencing

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Ahmet Dogan

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Bruce W Eckloff

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David I Smith

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Eric D Wieben

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George Vasmatzis

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Julie C Porcher

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Mark E Law

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Nazan Ozsan

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Sarah H Johnson

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Stephen M Ansell

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P2860

Transcriptome sequencing to detect gene fusions in cancer

miR-29a suppresses tristetraprolin, which is a regulator of epithelial polarity and metastasis

Development of anaplastic lymphoma kinase (ALK) small-molecule inhibitors for cancer therapy.

microRNA-29a induces aberrant self-renewal capacity in hematopoietic progenitors, biased myeloid development, and acute myeloid leukemia.

Inhibition of T cell antigen receptor signaling by VHR-related MKPX (VHX), a new dual specificity phosphatase related to VH1 related (VHR).

Deletion of the protein tyrosine phosphatase gene PTPN2 in T-cell acute lymphoblastic leukemia.

Telomere loss as a mechanism for chromosome instability in human cancer

Prognostic significance of anaplastic lymphoma kinase (ALK) protein expression in adults with anaplastic large cell lymphoma.

Protein tyrosine phosphatase receptor-type O truncated (PTPROt) regulates SYK phosphorylation, proximal B-cell-receptor signaling, and cellular proliferation.

ALK- anaplastic large-cell lymphoma is clinically and immunophenotypically different from both ALK+ ALCL and peripheral T-cell lymphoma, not otherwise specified: report from the International Peripheral T-Cell Lymphoma Project.

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915-919

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10.1182/BLOOD-2010-08-303305

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Andrew L Feldman

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