The cellular robustness by genetic redundancy in budding yeast.
about
Growth of novel epistatic interactions by gene duplicationTopological properties of robust biological and computational networks.Insight into the Recent Genome Duplication of the Halophilic Yeast Hortaea werneckii: Combining an Improved Genome with Gene Expression and Chromatin StructureAll or nothing: protein complexes flip essentiality between distantly related eukaryotes.Duplication and retention biases of essential and non-essential genes revealed by systematic knockdown analyses.Systematic lipidomic analysis of yeast protein kinase and phosphatase mutants reveals novel insights into regulation of lipid homeostasis.Human monogenic disease genes have frequently functionally redundant paralogs.Transcriptomic identification of iron-regulated and iron-independent gene copies within the heavily duplicated Trichomonas vaginalis genome.The role of protein interactions in mediating essentiality and synthetic lethality.Transcriptome Analysis of Spermophilus lateralis and Spermophilus tridecemlineatus Liver Does Not Suggest the Presence of Spermophilus-Liver-Specific Reference Genes.Biomolecular self-defense and futility of high-specificity therapeutic targeting.Structural and Functional Characterization of a Caenorhabditis elegans Genetic Interaction Network within PathwaysFeature Identification of Compensatory Gene Pairs without Sequence Homology in Yeast.Growth condition dependency is the major cause of non-responsiveness upon genetic perturbation.Human gut microbes use multiple transporters to distinguish vitamin B₁₂ analogs and compete in the gut.Genetic robustness and functional evolution of gene duplicatesMolecular mechanisms of paralogous compensation and the robustness of cellular networks.Orderly progression through S-phase requires dynamic ubiquitylation and deubiquitylation of PCNA.CRISPR Technology Reveals RAD(51)-ical Mechanisms of Repair in Roundworms: An Educational Primer for Use with "Promotion of Homologous Recombination by SWS-1 in Complex with RAD-51 Paralogs in Caenorhabditis elegans".Analysis of indel variations in the human disease-associated genes CDKN2AIP, WDR66, USP20 and OR7C2 in a Korean population.Genetic redundancy is prevalent within the 6.7 Mb Sinorhizobium meliloti genome.Loss of αB-crystallin function in zebrafish reveals critical roles in the development of the lens and stress resistance of the heart.Control of primary metabolism by a virulence regulatory network promotes robustness in a plant pathogen.Quantification of oxidative stress phenotypes based on high-throughput growth profiling of protein kinase and phosphatase knockouts.
P2860
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P2860
The cellular robustness by genetic redundancy in budding yeast.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
The cellular robustness by genetic redundancy in budding yeast.
@ast
The cellular robustness by genetic redundancy in budding yeast.
@en
type
label
The cellular robustness by genetic redundancy in budding yeast.
@ast
The cellular robustness by genetic redundancy in budding yeast.
@en
prefLabel
The cellular robustness by genetic redundancy in budding yeast.
@ast
The cellular robustness by genetic redundancy in budding yeast.
@en
P2093
P2860
P1433
P1476
The cellular robustness by genetic redundancy in budding yeast.
@en
P2093
Jingjing Li
Zhaolei Zhang
Zineng Yuan
P2860
P304
P356
10.1371/JOURNAL.PGEN.1001187
P577
2010-11-04T00:00:00Z