Enhancement of lipopolysaccharide-induced neutrophil oxygen radical production by tumor necrosis factor alpha
about
Hemoglobin toxicity in experimental bacterial peritonitis is due to production of reactive oxygen species.Bacterial lipopolysaccharide and tumor necrosis factor alpha synergistically increase expression of human endothelial adhesion molecules through activation of NF-kappaB and p38 mitogen-activated protein kinase signaling pathways.Thrombin-derived host defence peptide modulates neutrophil rolling and migration in vitro and functional response in vivo.Zinc ascorbate has superoxide dismutase-like activity and in vitro antimicrobial activity against Staphylococcus aureus and Escherichia coli.The Response of HeLa Cells to Fluorescent NanoDiamond Uptake.
P2860
Enhancement of lipopolysaccharide-induced neutrophil oxygen radical production by tumor necrosis factor alpha
description
1998 nî lūn-bûn
@nan
1998 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Enhancement of lipopolysacchar ...... by tumor necrosis factor alpha
@ast
Enhancement of lipopolysacchar ...... by tumor necrosis factor alpha
@en
type
label
Enhancement of lipopolysacchar ...... by tumor necrosis factor alpha
@ast
Enhancement of lipopolysacchar ...... by tumor necrosis factor alpha
@en
prefLabel
Enhancement of lipopolysacchar ...... by tumor necrosis factor alpha
@ast
Enhancement of lipopolysacchar ...... by tumor necrosis factor alpha
@en
P2093
P2860
P1476
Enhancement of lipopolysacchar ...... by tumor necrosis factor alpha
@en
P2093
A Ferrante
D A Rathjen
H P Jersmann
P2860
P304
P407
P577
1998-04-01T00:00:00Z