Nonspecific early protective immunity in Francisella and Listeria infections can be dependent on lymphocytes. - Wikidata - awgldk - TriplyDB

Nonspecific early protective immunity in Francisella and Listeria infections can be dependent on lymphocytes.

Nonspecific early protective immunity in Francisella and Listeria infections can be dependent on lymphocytes.

http://www.wikidata.org/entity/Q33757039

about

Importance of B cells, but not specific antibodies, in primary and secondary protective immunity to the intracellular bacterium Francisella tularensis live vaccine strain Purified lipopolysaccharide from Francisella tularensis live vaccine strain (LVS) induces protective immunity against LVS infection that requires B cells and gamma interferon.Intranasal vaccination with a defined attenuated Francisella novicida strain induces gamma interferon-dependent antibody-mediated protection against tularemia.Impaired antibacterial host defense in mice lacking the N-formylpeptide receptor.YopP-expressing variant of Y. pestis activates a potent innate immune response affording cross-protection against yersiniosis and tularemia [corrected].B1a cells enhance susceptibility to infection with virulent Francisella tularensis via modulation of NK/NKT cell responses.Recombinant attenuated Listeria monocytogenes vaccine expressing Francisella tularensis IglC induces protection in mice against aerosolized Type A F. tularensis.

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P2860

Nonspecific early protective immunity in Francisella and Listeria infections can be dependent on lymphocytes.

http://www.wikidata.org/entity/Q33757039

description

1998 nî lūn-bûn

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1998 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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1998 թվականի հուլիսին հրատարակված գիտական հոդված

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1998年の論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年论文

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Infection and Immunity

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Nonspecific early protective i ...... n be dependent on lymphocytes.

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A T MacIntyre

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K L Elkins

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T R Rhinehart-Jones

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P2860

Studies with MHC-deficient knock-out mice reveal impact of both MHC I- and MHC II-dependent T cell responses on Listeria monocytogenes infection

Inability of the Francisella tularensis lipopolysaccharide to mimic or to antagonize the induction of cell activation by endotoxins

Minimal requirements for murine resistance to infection with Francisella tularensis LVS.

Innate immunity to a facultative intracellular bacterial pathogen.

Live vaccine strain of Francisella tularensis: infection and immunity in mice.

Immunity to Salmonella typhimurium infection in C3H/HeJ and C3H/HeNCrlBR mice: studies with an aromatic-dependent live S. typhimurium strain as a vaccine

Rapid generation of specific protective immunity to Francisella tularensis.

Early pathogenesis of infection in the liver with the facultative intracellular bacteria Listeria monocytogenes, Francisella tularensis, and Salmonella typhimurium involves lysis of infected hepatocytes by leukocytes.

Introduction of Francisella tularensis at skin sites induces resistance to infection and generation of protective immunity.

Loss of either CD4+ or CD8+ T cells does not affect the magnitude of protective immunity to an intracellular pathogen, Francisella tularensis strain LVS.

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108373

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