Boron neutron capture therapy induces apoptosis of glioma cells through Bcl-2/Bax. - Wikidata - awgldk - TriplyDB

Boron neutron capture therapy induces apoptosis of glioma cells through Bcl-2/Bax.

Boron neutron capture therapy induces apoptosis of glioma cells through Bcl-2/Bax.

http://www.wikidata.org/entity/Q33759994

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Apoptosis through Bcl-2/Bax and cleaved caspase up-regulation in melanoma treated by boron neutron capture therapy Cyclic-RGDyC functionalized liposomes for dual-targeting of tumor vasculature and cancer cells in glioblastoma: An in vitro boron neutron capture therapy study Hydroxyapatite nanoparticles inhibit the growth of human glioma cells in vitro and in vivo.MiR-134 regulates the proliferation and invasion of glioblastoma cells by reducing Nanog expression.Boron neutron capture therapy induces cell cycle arrest and cell apoptosis of glioma stem/progenitor cells in vitro LRIG1 enhances chemosensitivity by modulating BCL-2 expression and receptor tyrosine kinase signaling in glioma cells.Overexpressed miRNA-137 inhibits human glioma cells growth by targeting Rac1.Folate receptor-mediated boron-10 containing carbon nanoparticles as potential delivery vehicles for boron neutron capture therapy of nonfunctional pituitary adenomas.Effect of boron neutron capture therapy for recurrent anaplastic meningioma: an autopsy case report.

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P2860

Boron neutron capture therapy induces apoptosis of glioma cells through Bcl-2/Bax.

http://www.wikidata.org/entity/Q33759994

description

2010 nî lūn-bûn

@nan

2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Boron neutron capture therapy induces apoptosis of glioma cells through Bcl-2/Bax.

@ast

Boron neutron capture therapy induces apoptosis of glioma cells through Bcl-2/Bax.

@en

type

label

Boron neutron capture therapy induces apoptosis of glioma cells through Bcl-2/Bax.

@ast

Boron neutron capture therapy induces apoptosis of glioma cells through Bcl-2/Bax.

@en

prefLabel

Boron neutron capture therapy induces apoptosis of glioma cells through Bcl-2/Bax.

@ast

Boron neutron capture therapy induces apoptosis of glioma cells through Bcl-2/Bax.

@en

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Boron neutron capture therapy induces apoptosis of glioma cells through Bcl-2/Bax.

@en

P2093

Geng Guo

http://www.w3.org/2001/XMLSchema#string

Haining Zhen

http://www.w3.org/2001/XMLSchema#string

Peng Wang

http://www.w3.org/2001/XMLSchema#string

Wei Zhang

http://www.w3.org/2001/XMLSchema#string

Xiang Zhang

http://www.w3.org/2001/XMLSchema#string

Xin Cheng

http://www.w3.org/2001/XMLSchema#string

Xinbiao Jiang

http://www.w3.org/2001/XMLSchema#string

Xinggang Mao

http://www.w3.org/2001/XMLSchema#string

P2860

Bcl-2 inhibitors: targeting mitochondrial apoptotic pathways in cancer therapy

Thymidine kinase 1 as a molecular target for boron neutron capture therapy of brain tumors

An inhibitor of Bcl-2 family proteins induces regression of solid tumours

Genotoxic effects in M5 cells and Chinese hamster V79 cells after exposure to 7Li-beam (LET=60 keV/microm) and correlation of their survival dynamics to nuclear damages and cell death.

Survival benefit of Boron neutron capture therapy for recurrent malignant gliomas.

Survival benefit from boron neutron capture therapy for the newly diagnosed glioblastoma patients.

Nitric oxide induces cell death by regulating anti-apoptotic BCL-2 family members.

Boron neutron capture therapy of brain tumors: an emerging therapeutic modality.

Convection enhanced delivery of boronated EGF as a molecular targeting agent for neutron capture therapy of brain tumors

Radiation-induced apoptosis.

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